Kate M. Vignali

6.5k total citations · 1 hit paper
32 papers, 4.2k citations indexed

About

Kate M. Vignali is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Kate M. Vignali has authored 32 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Immunology, 11 papers in Oncology and 8 papers in Molecular Biology. Recurrent topics in Kate M. Vignali's work include Immune Cell Function and Interaction (23 papers), T-cell and B-cell Immunology (18 papers) and Immunotherapy and Immune Responses (11 papers). Kate M. Vignali is often cited by papers focused on Immune Cell Function and Interaction (23 papers), T-cell and B-cell Immunology (18 papers) and Immunotherapy and Immune Responses (11 papers). Kate M. Vignali collaborates with scholars based in United States, Austria and Japan. Kate M. Vignali's co-authors include Dario A.A. Vignali, Creg J. Workman, Lauren W. Collison, Timothy Kuo, Richard S. Blumberg, Yao Wang, Richard Cross, Kelli L. Boyd, David Sehy and Andrea L. Szymczak-Workman and has published in prestigious journals such as Nature, Journal of Biological Chemistry and The EMBO Journal.

In The Last Decade

Kate M. Vignali

31 papers receiving 4.1k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The inhibitory cytokine IL-35 contributes to regulatory T-cell function

2007 1.6k citations Lauren W. Collison, Creg J. Workman et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kate M. Vignali United States	22	3.0k	1.2k	847	340	319	32	4.2k
Byoung S. Kwon South Korea	42	3.6k 1.2×	1.7k 1.4×	892 1.1×	335 1.0×	330 1.0×	100	4.9k
Philip J. Lucas United States	23	4.5k 1.5×	1.1k 0.9×	713 0.8×	353 1.0×	397 1.2×	32	5.6k
Luca Piali Switzerland	23	2.0k 0.7×	1.3k 1.0×	876 1.0×	316 0.9×	310 1.0×	32	3.5k
Clemens Scheinecker Austria	37	3.2k 1.1×	1.0k 0.8×	1.6k 1.8×	296 0.9×	261 0.8×	64	5.6k
Dhavalkumar D. Patel United States	30	2.3k 0.8×	1.7k 1.4×	793 0.9×	252 0.7×	328 1.0×	53	4.0k
Segundo González Spain	44	3.5k 1.2×	1.6k 1.3×	845 1.0×	330 1.0×	447 1.4×	109	5.2k
Satoru Kumaki Japan	25	3.5k 1.2×	1.1k 0.9×	606 0.7×	300 0.9×	566 1.8×	74	4.3k
Hironobu Asao Japan	35	2.6k 0.9×	1.2k 1.0×	1.4k 1.6×	388 1.1×	314 1.0×	85	4.5k
Terry I. Guinter United States	19	3.8k 1.3×	953 0.8×	525 0.6×	290 0.9×	249 0.8×	24	4.5k
Francisco Borrego United States	40	4.3k 1.4×	1.3k 1.0×	732 0.9×	169 0.5×	473 1.5×	97	5.3k

Countries citing papers authored by Kate M. Vignali

Since Specialization

Citations

This map shows the geographic impact of Kate M. Vignali's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate M. Vignali with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate M. Vignali more than expected).

Fields of papers citing papers by Kate M. Vignali

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate M. Vignali. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate M. Vignali. The network helps show where Kate M. Vignali may publish in the future.

Co-authorship network of co-authors of Kate M. Vignali

This figure shows the co-authorship network connecting the top 25 collaborators of Kate M. Vignali. A scholar is included among the top collaborators of Kate M. Vignali based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kate M. Vignali. Kate M. Vignali is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cui, Jian, Andrea L. Szymczak-Workman, Kate M. Vignali, et al.. (2023). IFNγ-induction of TH1-like regulatory T cells controls antiviral responses. Nature Immunology. 24(5). 841–854. 37 indexed citations

Guy, Clifford S., Diana M. Mitrea, Po-Chien Chou, et al.. (2022). LAG3 associates with TCR–CD3 complexes and suppresses signaling by driving co-receptor–Lck dissociation. Nature Immunology. 23(5). 757–767. 127 indexed citations

Andrews, Lawrence P., Kate M. Vignali, Andrea L. Szymczak-Workman, et al.. (2021). A Cre-driven allele-conditioning line to interrogate CD4+ conventional T cells. Immunity. 54(10). 2209–2217.e6. 9 indexed citations

Liu, Chang, Ashwin Somasundaram, Sasikanth Manne, et al.. (2020). Neuropilin-1 is a T cell memory checkpoint limiting long-term antitumor immunity. Nature Immunology. 21(9). 1010–1021. 101 indexed citations

Liu, Chang, Maria Chikina, Rahul Deshpande, et al.. (2019). Treg Cells Promote the SREBP1-Dependent Metabolic Fitness of Tumor-Promoting Macrophages via Repression of CD8+ T Cell-Derived Interferon-γ. Immunity. 51(2). 381–397.e6. 266 indexed citations

Zhang, Qianxia, Maria Chikina, Andrea L. Szymczak-Workman, et al.. (2017). LAG3 limits regulatory T cell proliferation and function in autoimmune diabetes. Science Immunology. 2(9). 120 indexed citations

Bettini, Matthew L., et al.. (2017). Cutting Edge: CD3 ITAM Diversity Is Required for Optimal TCR Signaling and Thymocyte Development. The Journal of Immunology. 199(5). 1555–1560. 35 indexed citations

He, Yanan, Sneha Rangarajan, Melissa C. Kerzic, et al.. (2015). Identification of the Docking Site for CD3 on the T Cell Receptor β Chain by Solution NMR. Journal of Biological Chemistry. 290(32). 19796–19805. 34 indexed citations

Guy, Clifford S., Kate M. Vignali, Jamshid Temirov, et al.. (2013). Distinct TCR signaling pathways drive proliferation and cytokine production in T cells. Nature Immunology. 14(3). 262–270. 181 indexed citations

10.

Collison, Lauren W., Greg M. Delgoffe, Clifford S. Guy, et al.. (2012). The composition and signaling of the IL-35 receptor are unconventional. Nature Immunology. 13(3). 290–299. 357 indexed citations

11.

Li, Nianyu, Yao Wang, Karen Forbes, et al.. (2007). Metalloproteases regulate T‐cell proliferation and effector function via LAG‐3. The EMBO Journal. 26(2). 494–504. 212 indexed citations

12.

Collison, Lauren W., Creg J. Workman, Timothy Kuo, et al.. (2007). The inhibitory cytokine IL-35 contributes to regulatory T-cell function. Nature. 450(7169). 566–569. 1553 indexed citations breakdown →

13.

Holst, Jeff, Kate M. Vignali, Amanda R. Burton, & Dario A.A. Vignali. (2006). Rapid analysis of T-cell selection in vivo using T cell–receptor retrogenic mice. Nature Methods. 3(3). 191–197. 127 indexed citations

14.

Holst, Jeff, Andrea L. Szymczak-Workman, Kate M. Vignali, et al.. (2006). Generation of T-cell receptor retrogenic mice. Nature Protocols. 1(1). 406–417. 216 indexed citations

15.

Szymczak, Andrea L, Creg J. Workman, Diana Gil, et al.. (2005). The CD3ε Proline-Rich Sequence, and Its Interaction with Nck, Is Not Required for T Cell Development and Function. The Journal of Immunology. 175(1). 270–275. 60 indexed citations

16.

Szymczak, Andrea L, Creg J. Workman, Yao Wang, et al.. (2004). Addendum: Correction of multi-gene deficiency in vivo using a single 'self-cleaving' 2A peptide–based retroviral vector. Nature Biotechnology. 22(6). 760–760. 2 indexed citations

17.

Arnold, Paula Y., Nicole L. La Gruta, Timothy J. Miller, et al.. (2002). The Majority of Immunogenic Epitopes Generate CD4+ T Cells That Are Dependent on MHC Class II-Bound Peptide-Flanking Residues. The Journal of Immunology. 169(8). 4674–4674. 5 indexed citations

18.

Arnold, Paula Y., Kate M. Vignali, Nicole L. La Gruta, et al.. (2002). Reliable generation and use of MHC class II:γ2aFc multimers for the identification of antigen-specific CD4+ T cells. Journal of Immunological Methods. 271(1-2). 137–151. 26 indexed citations

19.

Carson, Richard T., Drashti Desai, Kate M. Vignali, & Dario A.A. Vignali. (1999). Immunoregulation of Th cells by naturally processed peptide antagonists.. PubMed. 162(1). 1–4. 23 indexed citations

20.

Carson, Richard T., Kate M. Vignali, David L. Woodland, & Dario A.A. Vignali. (1997). T Cell Receptor Recognition of MHC Class II–Bound Peptide Flanking Residues Enhances Immunogenicity and Results in Altered TCR V Region Usage. Immunity. 7(3). 387–399. 147 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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