Kate J. Heesom
- Molecular Biology top 2%
- Cell Biology top 0.5%
- Physiology top 5%
- Infectious Diseases top 5%
- Epidemiology top 5%
- Co-authors
- Peter J. CullenFlorian SteinbergRichard M. DentonDavid A. MatthewsIngeborg HersS K MouleDaniel C. BerwickJeremy M. Tavaré
- Topics
- Cellular transport and secretion (22 papers)Antibiotic Resistance in Bacteria (15 papers)Erythrocyte Function and Pathophysiology (13 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryNature Communications
- Partner nations
- United KingdomUnited StatesAustralia
In The Last Decade
Kate J. Heesom
152 papers receiving 5.3k citations
Hit Papers
Peers
Comparison fields: 5 of 141
- Molecular Biology 2.9k
- Cell Biology 1.2k
- Physiology 681
- Infectious Diseases 514
- Epidemiology 492
Countries citing papers authored by Kate J. Heesom
This map shows the geographic impact of Kate J. Heesom's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate J. Heesom with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate J. Heesom more than expected).
Fields of papers citing papers by Kate J. Heesom
This network shows the impact of papers produced by Kate J. Heesom. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate J. Heesom. The network helps show where Kate J. Heesom may publish in the future.
Co-authorship network of co-authors of Kate J. Heesom
This figure shows the co-authorship network connecting the top 25 collaborators of Kate J. Heesom. A scholar is included among the top collaborators of Kate J. Heesom based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kate J. Heesom. Kate J. Heesom is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 20 | |
| 4 | 3 | |
| 5 | 18 | |
| 6 | 12 | |
| 7 | 5 | |
| 8 | 18 | |
| 9 | 9 | |
| 10 | 9 | |
| 11 | 11 | |
| 12 | 22 | |
| 13 | 16 | |
| 14 | 14 | |
| 15 | 16 | |
| 16 | 7 | |
| 17 | 18 | |
| 18 | 15 | |
| 19 | 157 | |
| 20 | 120 |
About Kate J. Heesom
Kate J. Heesom is a scholar working on Molecular Medicine, Cell Biology and Endocrinology, having authored 156 papers that have together received 5.4k indexed citations. Recurring topics across this work include Cellular transport and secretion (22 papers), Antibiotic Resistance in Bacteria (15 papers) and Erythrocyte Function and Pathophysiology (13 papers). The work is most often cited by research in Cell Biology (1.2k citations), Molecular Biology (2.9k citations) and Molecular Medicine (195 citations). Kate J. Heesom has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include Peter J. Cullen, Florian Steinberg, Richard M. Denton, David A. Matthews, Ingeborg Hers, S K Moule, Daniel C. Berwick, Jeremy M. Tavaré, Matthew Gallon and Elaine Thomas. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.