Karine Crozat
- Immunology top 0.2%
- Molecular Biology top 5%
- Epidemiology top 2%
- Oncology top 5%
- Cancer Research top 5%
- Co-authors
- Bruce BeutlerPhilippe GeorgelKasper HoebeXin DuSosathya SovathSuzanne MuddMarc DalodSophie Rutschmann
- Topics
- Immune Cell Function and Interaction (25 papers)T-cell and B-cell Immunology (20 papers)Immune Response and Inflammation (17 papers)
- Partner nations
- United StatesFranceJapan
In The Last Decade
Karine Crozat
48 papers receiving 6.7k citations
Hit Papers
Peers
Comparison fields: 5 of 115
- Immunology 5.4k
- Molecular Biology 1.5k
- Epidemiology 1.1k
- Oncology 757
- Cancer Research 543
Countries citing papers authored by Karine Crozat
This map shows the geographic impact of Karine Crozat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Karine Crozat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Karine Crozat more than expected).
Fields of papers citing papers by Karine Crozat
This network shows the impact of papers produced by Karine Crozat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Karine Crozat. The network helps show where Karine Crozat may publish in the future.
Co-authorship network of co-authors of Karine Crozat
This figure shows the co-authorship network connecting the top 25 collaborators of Karine Crozat. A scholar is included among the top collaborators of Karine Crozat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Karine Crozat. Karine Crozat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 77 | |
| 2 | 141 | |
| 3 | 31 | |
| 4 | 28 | |
| 5 | 14 | |
| 6 | 79 | |
| 7 | 48 | |
| 8 | 51 | |
| 9 | 89 | |
| 10 | 35 | |
| 11 | 12 | |
| 12 | 59 | |
| 13 | 138 | |
| 14 | 48 | |
| 15 | The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9breakdown → | 553 |
| 16 | CD36 is a sensor of diacylglyceridesbreakdown → | 693 |
| 17 | 12 | |
| 18 | 62 | |
| 19 | 41 | |
| 20 | Identification of Lps2 as a key transducer of MyD88-independent TIR signallingbreakdown → | 1009 |
About Karine Crozat
Karine Crozat is a scholar working on Immunology, Microbiology and Epidemiology, having authored 48 papers that have together received 6.8k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (25 papers), T-cell and B-cell Immunology (20 papers) and Immune Response and Inflammation (17 papers). The work is most often cited by research in Immunology (5.4k citations), Microbiology (401 citations) and Cancer Research (543 citations). Karine Crozat has collaborated with scholars based in United States, France and Japan. Frequent co-authors include Bruce Beutler, Philippe Georgel, Kasper Hoebe, Xin Du, Sosathya Sovath, Suzanne Mudd, Marc Dalod, Sophie Rutschmann, Koichi Tabeta and Edith M. Janssen. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Nature Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.