Kanami Itoh

3.9k total citations · 1 hit paper
13 papers, 3.2k citations indexed

About

Kanami Itoh is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Kanami Itoh has authored 13 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Oncology and 3 papers in Cancer Research. Recurrent topics in Kanami Itoh's work include Bone Metabolism and Diseases (12 papers), Bone health and treatments (11 papers) and NF-κB Signaling Pathways (3 papers). Kanami Itoh is often cited by papers focused on Bone Metabolism and Diseases (12 papers), Bone health and treatments (11 papers) and NF-κB Signaling Pathways (3 papers). Kanami Itoh collaborates with scholars based in Japan, Australia and United States. Kanami Itoh's co-authors include Nobuyuki Udagawa, Matthew T. Gillespie, T. John Martin, Naoyuki Takahashi, Kenichiro MATSUZAKI, Seiji Saito, Shigeru Ishiyama, Naoyuki Kamatani, Shigeru Kotake and Kazuhiko Inoue and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Immunology and Endocrinology.

In The Last Decade

Kanami Itoh

13 papers receiving 3.2k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis

1999 1.4k citations Shigeru Kotake, Nobuyuki Udagawa et al. Journal of Clinical Investigation profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
Kanami Itoh	1.9k	1.3k	950	740	462	13	3.2k
Ayako Suematsu	1.6k 0.8×	981 0.7×	892 0.9×	599 0.8×	259 0.6×	12	2.6k
Akiko Kukita	2.0k 1.1×	1.1k 0.8×	536 0.6×	546 0.7×	362 0.8×	83	3.1k
Yuho Kadono	2.7k 1.4×	1.7k 1.2×	1.2k 1.3×	967 1.3×	548 1.2×	75	4.4k
Leonard Rifas	1.7k 0.9×	941 0.7×	423 0.4×	630 0.9×	562 1.2×	49	3.5k
Mark Tometsko	3.0k 1.6×	2.6k 1.9×	1.1k 1.1×	470 0.6×	560 1.2×	18	4.3k
Emma Timms	3.0k 1.6×	2.3k 1.7×	2.9k 3.0×	563 0.8×	507 1.1×	31	6.1k
Shigeru Ishiyama	1.0k 0.5×	626 0.5×	918 1.0×	658 0.9×	181 0.4×	22	2.4k
G R Mundy	2.1k 1.1×	2.2k 1.6×	335 0.4×	379 0.5×	512 1.1×	51	3.8k
Amy J. Koh	1.4k 0.7×	1.2k 0.9×	806 0.8×	262 0.4×	350 0.8×	53	2.8k

Countries citing papers authored by Kanami Itoh

Since Specialization

Citations

This map shows the geographic impact of Kanami Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kanami Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kanami Itoh more than expected).

Fields of papers citing papers by Kanami Itoh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kanami Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kanami Itoh. The network helps show where Kanami Itoh may publish in the future.

Co-authorship network of co-authors of Kanami Itoh

This figure shows the co-authorship network connecting the top 25 collaborators of Kanami Itoh. A scholar is included among the top collaborators of Kanami Itoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kanami Itoh. Kanami Itoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Suda, Koji, Nobuyuki Udagawa, Nobuaki Sato, et al.. (2004). Suppression of Osteoprotegerin Expression by Prostaglandin E2 Is Crucially Involved in Lipopolysaccharide-Induced Osteoclast Formation. The Journal of Immunology. 172(4). 2504–2510. 142 indexed citations

Itoh, Kanami, Nobuyuki Udagawa, Koji Suda, et al.. (2003). Lipopolysaccharide Promotes the Survival of Osteoclasts Via Toll-Like Receptor 4, but Cytokine Production of Osteoclasts in Response to Lipopolysaccharide Is Different from That of Macrophages. The Journal of Immunology. 170(7). 3688–3695. 163 indexed citations

Itoh, Kanami, et al.. (2003). Specific biological functions of vacuolar‐type H⁺‐ATPase and lysosomal cysteine proteinase, cathepsin K, in osteoclasts. The Anatomical Record Part A Discoveries in Molecular Cellular and Evolutionary Biology. 270A(2). 152–161. 16 indexed citations

Takami, Masamichi, K. Suda, Kanami Itoh, et al.. (2003). Involvement of vacuolar H+-ATPase in incorporation of risedronate into osteoclasts. Bone. 32(4). 341–349. 26 indexed citations

Takami, Masamichi, et al.. (2002). Regulation of osteoclast differentiation and function by receptor activator of NFkB ligand and osteoprotegerin. The Anatomical Record. 268(2). 137–146. 52 indexed citations

Li, Xiaotong, Nobuyuki Udagawa, Kanami Itoh, et al.. (2002). p38 MAPK-Mediated Signals Are Required for Inducing Osteoclast Differentiation But Not for Osteoclast Function. Endocrinology. 143(8). 3105–3113. 246 indexed citations

Murase, Yoshiyuki, Nobuo Okahashi, Takeyoshi Koseki, et al.. (2001). Possible involvement of protein kinases and Smad2 signaling pathways on osteoclast differentiation enhanced by activin A. Journal of Cellular Physiology. 188(2). 236–242. 24 indexed citations

Itoh, Kanami, Nobuyuki Udagawa, Takenobu Katagiri, et al.. (2001). Bone Morphogenetic Protein 2 Stimulates Osteoclast Differentiation and Survival Supported by Receptor Activator of Nuclear Factor-κB Ligand. Endocrinology. 142(8). 3656–3662. 211 indexed citations

Quinn, J, Kanami Itoh, Nobuyuki Udagawa, et al.. (2001). Transforming Growth Factor β Affects Osteoclast Differentiation via Direct and Indirect Actions. Journal of Bone and Mineral Research. 16(10). 1787–1794. 217 indexed citations

10.

Itoh, Kanami, Nobuyuki Udagawa, Kenichiro MATSUZAKI, et al.. (2000). Importance of Membrane- or Matrix-Associated Forms of M-CSF and RANKL/ODF in Osteoclastogenesis Supported by SaOS-4/3 Cells Expressing Recombinant PTH/PTHrP Receptors. Journal of Bone and Mineral Research. 15(9). 1766–1775. 74 indexed citations

11.

Udagawa, Nobuyuki, Naoyuki Takahashi, Hisataka Yasuda, et al.. (2000). Osteoprotegerin Produced by Osteoblasts Is an Important Regulator in Osteoclast Development and Function*. Endocrinology. 141(9). 3478–3484. 329 indexed citations

12.

Udagawa, Nobuyuki, N. Takahashi, Eijiro Jimi, et al.. (1999). Osteoblasts/stromal cells stimulate osteoclast activation through expression of osteoclast differentiation factor/RANKL but not macrophage colony-stimulating factor. Bone. 25(5). 517–523. 358 indexed citations

13.

Kotake, Shigeru, Nobuyuki Udagawa, Kenichiro MATSUZAKI, et al.. (1999). IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis. Journal of Clinical Investigation. 103(9). 1345–1352. 1386 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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