Ayako Suematsu

3.2k total citations · 2 hit papers
12 papers, 2.6k citations indexed

About

Ayako Suematsu is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Ayako Suematsu has authored 12 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Oncology and 5 papers in Cancer Research. Recurrent topics in Ayako Suematsu's work include Bone Metabolism and Diseases (10 papers), NF-κB Signaling Pathways (5 papers) and Bone health and treatments (4 papers). Ayako Suematsu is often cited by papers focused on Bone Metabolism and Diseases (10 papers), NF-κB Signaling Pathways (5 papers) and Bone health and treatments (4 papers). Ayako Suematsu collaborates with scholars based in Japan, United States and France. Ayako Suematsu's co-authors include Tatsuhiko Kodama, Kojiro Sato, Yasuyuki Morishita, Hiroshi Takayanagi, Kazuo Okamoto, Akira Yamaguchi, Sakae Tanaka, Shizuo Akira, Yuho Kadono and J. Daniel and has published in prestigious journals such as Nature, Nature Medicine and Nature Communications.

In The Last Decade

Ayako Suematsu

12 papers receiving 2.5k citations

Hit Papers

Th17 functions as an osteoclastogenic helper T cell subse... 2004 2026 2011 2018 2006 2004 400 800 1.2k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Th17 functions as an osteoclastogenic helper T cell subset that links T cell activation and bone destruction
    2006 1.2k citations Kojiro Sato, Ayako Suematsu et al. The Journal of Experimental Medicine profile →
  2. Costimulatory signals mediated by the ITAM motif cooperate with RANKL for bone homeostasis
    2004 673 citations Takako Koga, Masanori Inui et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ayako Suematsu Japan 11 1.6k 981 892 599 367 12 2.6k
Jan Elliott Australia 15 1.8k 1.1× 1.4k 1.4× 462 0.5× 391 0.7× 323 0.9× 18 2.4k
Antonio Oliveira-dos-Santos Canada 10 2.6k 1.6× 1.9k 1.9× 1.1k 1.3× 479 0.8× 726 2.0× 12 3.8k
Paul R. Odgren United States 27 2.0k 1.3× 870 0.9× 307 0.3× 383 0.6× 310 0.8× 56 2.6k
T Tamura Japan 7 1.1k 0.7× 941 1.0× 376 0.4× 294 0.5× 236 0.6× 9 1.7k
Jean Chappel United States 24 1.4k 0.9× 742 0.8× 546 0.6× 406 0.7× 237 0.6× 35 2.3k
Tetsuro Ikebe Japan 28 1.2k 0.8× 1.0k 1.0× 251 0.3× 257 0.4× 680 1.9× 81 2.5k
Midori Nakamura Japan 21 1.2k 0.8× 740 0.8× 329 0.4× 271 0.5× 211 0.6× 57 1.9k
Hidefumi Fukushima Japan 30 2.2k 1.4× 952 1.0× 273 0.3× 242 0.4× 608 1.7× 54 2.9k
Sakamuri V. Reddy United States 24 1.7k 1.1× 1.1k 1.2× 265 0.3× 294 0.5× 504 1.4× 43 2.3k
Helen Weedon Australia 23 621 0.4× 424 0.4× 901 1.0× 726 1.2× 138 0.4× 39 2.1k

Countries citing papers authored by Ayako Suematsu

Since Specialization
Citations

This map shows the geographic impact of Ayako Suematsu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ayako Suematsu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ayako Suematsu more than expected).

Fields of papers citing papers by Ayako Suematsu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ayako Suematsu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ayako Suematsu. The network helps show where Ayako Suematsu may publish in the future.

Co-authorship network of co-authors of Ayako Suematsu

This figure shows the co-authorship network connecting the top 25 collaborators of Ayako Suematsu. A scholar is included among the top collaborators of Ayako Suematsu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ayako Suematsu. Ayako Suematsu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Okamoto, Kazuo, Yuta Nakai, Ryunosuke Muro, et al.. (2019). Soluble RANKL is physiologically dispensable but accelerates tumour metastasis to bone. Nature Metabolism. 1(9). 868–875. 58 indexed citations
2.
Negishi‐Koga, Takako, Noriko Komatsu, Kazuo Okamoto, et al.. (2015). Immune complexes regulate bone metabolism through FcRγ signalling. Nature Communications. 6(1). 6637–6637. 111 indexed citations
3.
Negishi‐Koga, Takako, Yusuke Nagai, Ayako Suematsu, et al.. (2015). Phosphoproteomic analysis of kinase-deficient mice reveals multiple TAK1 targets in osteoclast differentiation. Biochemical and Biophysical Research Communications. 463(4). 1284–1290. 11 indexed citations
4.
Suematsu, Ayako, Yasuhito Tajiri, Tomoki Nakashima, et al.. (2007). Scientific basis for the efficacy of combined use of antirheumatic drugs against bone destruction in rheumatoid arthritis. Modern Rheumatology. 17(1). 17–23. 33 indexed citations
5.
Suematsu, Ayako & Hiroshi Takayanagi. (2007). Interplay between the immune and skeletal cells in the regulation of inflammatory bone destruction. Japanese Journal of Clinical Immunology. 30(1). 22–28. 3 indexed citations
6.
Suematsu, Ayako, Yasuhito Tajiri, Tomoki Nakashima, et al.. (2007). Scientific basis for the efficacy of combined use of antirheumatic drugs against bone destruction in rheumatoid arthritis. Modern Rheumatology. 17(1). 17–23. 23 indexed citations
7.
Sato, Kojiro, Ayako Suematsu, Kazuo Okamoto, et al.. (2006). Th17 functions as an osteoclastogenic helper T cell subset that links T cell activation and bone destruction. The Journal of Experimental Medicine. 203(12). 2673–2682. 1232 indexed citations breakdown →
8.
Sato, Kojiro, Ayako Suematsu, Tomoki Nakashima, et al.. (2006). Regulation of osteoclast differentiation and function by the CaMK-CREB pathway. Nature Medicine. 12(12). 1410–1416. 287 indexed citations
9.
Sato, Kojiro, Ayako Suematsu, Kazuo Okamoto, et al.. (2006). Th17 functions as an osteoclastogenic helper T cell subset that links T cell activation and bone destruction. The Journal of Cell Biology. 175(4). i8–i8. 86 indexed citations
10.
Koga, Takako, Masanori Inui, Kazuya Inoue, et al.. (2004). Costimulatory signals mediated by the ITAM motif cooperate with RANKL for bone homeostasis. Nature. 428(6984). 758–763. 673 indexed citations breakdown →
11.
Koshihara, Yasuko, et al.. (2002). Osteoclastogenic potential of bone marrow cells increases with age in elderly women with fracture. Mechanisms of Ageing and Development. 123(10). 1321–1331. 25 indexed citations
12.
Yoshida, Tsuyoshi, Hiroshi Sakamoto, Toshiyuki Horiuchi, et al.. (2001). Involvement of Prostaglandin E2in Interleukin-1α-Induced Parathyroid Hormone-Related Peptide Production in Synovial Fibroblasts of Patients with Rheumatoid Arthritis. The Journal of Clinical Endocrinology & Metabolism. 86(7). 3272–3278. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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