Amy J. Koh

3.5k total citations
53 papers, 2.8k citations indexed

About

Amy J. Koh is a scholar working on Oncology, Molecular Biology and Immunology. According to data from OpenAlex, Amy J. Koh has authored 53 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Oncology, 24 papers in Molecular Biology and 15 papers in Immunology. Recurrent topics in Amy J. Koh's work include Bone health and treatments (30 papers), Bone Metabolism and Diseases (16 papers) and Phagocytosis and Immune Regulation (11 papers). Amy J. Koh is often cited by papers focused on Bone health and treatments (30 papers), Bone Metabolism and Diseases (16 papers) and Phagocytosis and Immune Regulation (11 papers). Amy J. Koh collaborates with scholars based in United States, Türkiye and France. Amy J. Koh's co-authors include Laurie K. McCauley, Thomas J. Rosol, Evan T. Keller, Hernan Roca, Kenneth J. Pienta, Fabiana N. Soki, Martha J. Somerman, Burak Demiralp, Gary E. Landreth and Charles R. Maliszewski and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Amy J. Koh

52 papers receiving 2.8k citations

Peers

Amy J. Koh

ONCOL

IMMUN

OSM

RHEUM

Masanori Koide Japan

Akiko Kukita Japan

Patricia Collin‐Osdoby United States

Qingxia Wei Canada

Kihyuk Shin South Korea

Leonard Rifas United States

Eisei Kondo Japan

Mo Kang United States

Midori Nakamura Japan

Hidefumi Fukushima Japan

Masanori Koide Japan

Amy J. Koh

1.5k ×1.1

951 ×0.8

ONCOL

385 ×0.5

IMMUN

340 ×1.0

OSM

339 ×1.3

RHEUM

Citations per year, relative to Amy J. Koh Amy J. Koh (= 1×) peers Masanori Koide

Countries citing papers authored by Amy J. Koh

Since Specialization

Citations

This map shows the geographic impact of Amy J. Koh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amy J. Koh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amy J. Koh more than expected).

Fields of papers citing papers by Amy J. Koh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amy J. Koh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amy J. Koh. The network helps show where Amy J. Koh may publish in the future.

Co-authorship network of co-authors of Amy J. Koh

This figure shows the co-authorship network connecting the top 25 collaborators of Amy J. Koh. A scholar is included among the top collaborators of Amy J. Koh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amy J. Koh. Amy J. Koh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Koh, Amy J., Robert Kent, Kenneth M. Kozloff, et al.. (2024). CCL2/CCR2 Signalling in Mesenchymal Stem/Progenitor Cell Recruitment and Fracture Healing in Mice. Journal of Cellular and Molecular Medicine. 28(24). e70300–e70300.

Batoon, Lena, Amy J. Koh, Susan Millard, et al.. (2024). Induction of osteoblast apoptosis stimulates macrophage efferocytosis and paradoxical bone formation. Bone Research. 12(1). 43–43. 19 indexed citations

Batoon, Lena, et al.. (2023). Caspase-9 driven murine model of selective cell apoptosis and efferocytosis. Cell Death and Disease. 14(1). 58–58. 16 indexed citations

Sinder, Benjamin P., Amy J. Koh, Lena Batoon, et al.. (2023). Parathyroid hormone and trabectedin have differing effects on macrophages and stress fracture repair. Bone. 179. 116983–116983. 1 indexed citations

Koh, Amy J., et al.. (2022). Anabolic actions of parathyroid hormone in a hypophosphatasia mouse model. Osteoporosis International. 33(11). 2423–2433. 1 indexed citations

Koh, Amy J., et al.. (2020). Anabolic actions of PTH in murine models: two decades of insights. Journal of Bone and Mineral Research. 36(10). 1979–1998. 15 indexed citations

Roca, Hernan, Jacqueline Jones, Amy J. Koh, et al.. (2017). Apoptosis-induced CXCL5 accelerates inflammation and growth of prostate tumor metastases in bone. Journal of Clinical Investigation. 128(1). 248–266. 103 indexed citations

Michalski, Megan N., et al.. (2016). Modulation of Osteoblastic Cell Efferocytosis by Bone Marrow Macrophages. Journal of Cellular Biochemistry. 117(12). 2697–2706. 52 indexed citations

Al‐Dujaili, Saja, Amy J. Koh, Ming Dang, et al.. (2015). Calcium Sensing Receptor Function Supports Osteoblast Survival and Acts as a Co‐Factor in PTH Anabolic Actions in Bone. Journal of Cellular Biochemistry. 117(7). 1556–1567. 30 indexed citations

10.

Soki, Fabiana N., Amy J. Koh, Jacqueline Jones, et al.. (2014). Polarization of Prostate Cancer-associated Macrophages Is Induced by Milk Fat Globule-EGF Factor 8 (MFG-E8)-mediated Efferocytosis. Journal of Biological Chemistry. 289(35). 24560–24572. 140 indexed citations

11.

Park, Serk In, Changki Lee, W. David Sadler, et al.. (2013). Parathyroid Hormone–Related Protein Drives a CD11b+Gr1+ Cell–Mediated Positive Feedback Loop to Support Prostate Cancer Growth. Cancer Research. 73(22). 6574–6583. 46 indexed citations

12.

Cho, Sun Wook, Flávia Q. Pirih, Amy J. Koh, et al.. (2013). The Soluble Interleukin-6 Receptor Is a Mediator of Hematopoietic and Skeletal Actions of Parathyroid Hormone. Journal of Biological Chemistry. 288(10). 6814–6825. 35 indexed citations

13.

Park, Serk In, Jinhui Liao, Janice E. Berry, et al.. (2012). Cyclophosphamide Creates a Receptive Microenvironment for Prostate Cancer Skeletal Metastasis. Cancer Research. 72(10). 2522–2532. 58 indexed citations

14.

Novince, Chad M., Payam Entezami, Christopher G. Wilson, et al.. (2012). Impact of proteoglycan‐4 and parathyroid hormone on articular cartilage. Journal of Orthopaedic Research®. 31(2). 183–190. 11 indexed citations

15.

Soki, Fabiana N., Xin Li, Janice E. Berry, et al.. (2012). The effects of zoledronic acid in the bone and vasculature support of hematopoietic stem cell niches. Journal of Cellular Biochemistry. 114(1). 67–78. 28 indexed citations

16.

Li, Xin, Jinhui Liao, Serk In Park, et al.. (2011). Drugs which inhibit osteoclast function suppress tumor growth through calcium reduction in bone. Bone. 48(6). 1354–1361. 14 indexed citations

17.

Liao, Jinhui, Xin Li, Amy J. Koh, et al.. (2008). Tumor expressed PTHrP facilitates prostate cancer‐induced osteoblastic lesions. International Journal of Cancer. 123(10). 2267–2278. 80 indexed citations

18.

Pettway, Glenda J., Jeffrey A. Meganck, Amy J. Koh, et al.. (2008). Parathyroid hormone mediates bone growth through the regulation of osteoblast proliferation and differentiation. Bone. 42(4). 806–818. 95 indexed citations

19.

Berry, Janice E., et al.. (2004). The Role of Parathyroid Hormone‐Related Protein in the Regulation of Osteoclastogenesis by Cementoblasts. Journal of Periodontology. 75(9). 1247–1254. 71 indexed citations

20.

McCauley, Laurie K., et al.. (1997). Proto-Oncogene c-fosIs Transcriptionally Regulated by Parathyroid Hormone (PTH) and PTH-Related Protein in a Cyclic Adenosine Monophosphate-Dependent Manner in Osteoblastic Cells¹. Endocrinology. 138(12). 5427–5433. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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