Shigeru Ishiyama

2.9k total citations · 1 hit paper
22 papers, 2.4k citations indexed

About

Shigeru Ishiyama is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Immunology. According to data from OpenAlex, Shigeru Ishiyama has authored 22 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cardiology and Cardiovascular Medicine, 8 papers in Molecular Biology and 7 papers in Immunology. Recurrent topics in Shigeru Ishiyama's work include Viral Infections and Immunology Research (7 papers), Cardiovascular Effects of Exercise (5 papers) and Cardiac Fibrosis and Remodeling (4 papers). Shigeru Ishiyama is often cited by papers focused on Viral Infections and Immunology Research (7 papers), Cardiovascular Effects of Exercise (5 papers) and Cardiac Fibrosis and Remodeling (4 papers). Shigeru Ishiyama collaborates with scholars based in Japan, Australia and China. Shigeru Ishiyama's co-authors include Nobuyuki Udagawa, Naoyuki Kamatani, Seiji Saito, Shigeru Kotake, Kazuhiko Inoue, Kanami Itoh, Matthew T. Gillespie, T. John Martin, Kenichiro MATSUZAKI and Toshio Nishikawa and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and The Journal of Immunology.

In The Last Decade

Shigeru Ishiyama

21 papers receiving 2.3k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis

1999 1.4k citations Shigeru Kotake, Nobuyuki Udagawa et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Shigeru Ishiyama Japan	15	1.0k	918	658	626	366	22	2.4k
Fabia Brentano Switzerland	21	1.1k 1.1×	1.2k 1.3×	924 1.4×	530 0.8×	77 0.2×	23	2.9k
Hans P. Kiener Austria	29	677 0.7×	700 0.8×	803 1.2×	336 0.5×	67 0.2×	51	2.2k
Yasuo Koishihara Japan	26	1.2k 1.2×	1.1k 1.2×	384 0.6×	1.3k 2.2×	53 0.1×	39	3.1k
Lisa G. M. van Baarsen Netherlands	31	572 0.6×	1.4k 1.5×	1.5k 2.3×	615 1.0×	127 0.3×	76	3.1k
Birgitte Oppers‐Walgreen Netherlands	15	654 0.7×	1.5k 1.6×	910 1.4×	647 1.0×	35 0.1×	17	2.5k
Abdelilah Wakkach France	19	789 0.8×	1.2k 1.3×	174 0.3×	473 0.8×	66 0.2×	27	2.3k
Vincent Praloran France	31	1.0k 1.0×	790 0.9×	383 0.6×	546 0.9×	105 0.3×	127	3.3k
Klaus M. Hummel Switzerland	21	623 0.6×	350 0.4×	695 1.1×	361 0.6×	50 0.1×	26	1.6k
Martine Chabaud France	10	482 0.5×	1.6k 1.8×	989 1.5×	664 1.1×	39 0.1×	12	2.6k
Birgit Niederreiter Austria	24	762 0.8×	662 0.7×	680 1.0×	374 0.6×	38 0.1×	58	1.8k

Countries citing papers authored by Shigeru Ishiyama

Since Specialization

Citations

This map shows the geographic impact of Shigeru Ishiyama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shigeru Ishiyama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shigeru Ishiyama more than expected).

Fields of papers citing papers by Shigeru Ishiyama

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shigeru Ishiyama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shigeru Ishiyama. The network helps show where Shigeru Ishiyama may publish in the future.

Co-authorship network of co-authors of Shigeru Ishiyama

This figure shows the co-authorship network connecting the top 25 collaborators of Shigeru Ishiyama. A scholar is included among the top collaborators of Shigeru Ishiyama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shigeru Ishiyama. Shigeru Ishiyama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

1.

Seko, Yoshinori, Shigeru Ishiyama, Takeshi Kasajima, et al.. (2002). Expression of tumor necrosis factor ligand superfamily costimulatory molecules CD27L, CD30L, OX40L and 4-1BBL in the heart of patients with acute myocarditis and dilated cardiomyopathy. Cardiovascular Pathology. 11(3). 166–170. 17 indexed citations

2.

Toyozaki, Tetsuya, Toshihiro Saito, Hirokazu Shiraishi, et al.. (2001). Macrophage Inflammatory Protein-1α Relates to the Recruitment of Inflammatory Cells in Myosin-Induced Autoimmune Myocarditis in Rats. Laboratory Investigation. 81(7). 929–936. 11 indexed citations

3.

Imanaka‐Yoshida, Kyoko, Michiaki Hiroe, Toshio Nishikawa, et al.. (2001). Tenascin-C Modulates Adhesion of Cardiomyocytes to Extracellular Matrix during Tissue Remodeling after Myocardial Infarction. Laboratory Investigation. 81(7). 1015–1024. 145 indexed citations

4.

Kotake, Shigeru, Nobuyuki Udagawa, Masayuki Hakoda, et al.. (2001). Activated human T cells directly induce osteoclastogenesis from human monocytes: Possible role of T cells in bone destruction in rheumatoid arthritis patients. Arthritis & Rheumatism. 44(5). 1003–1012. 352 indexed citations

5.

Kotake, Shigeru, Nobuyuki Udagawa, Masayuki Hakoda, et al.. (2001). Activated human T cells directly induce osteoclastogenesis from human monocytes: Possible role of T cells in bone destruction in rheumatoid arthritis patients. Arthritis & Rheumatism. 44(5). 1003–1012. 14 indexed citations

6.

Nagata, Makoto, Michiaki Hiroe, Shigeru Ishiyama, et al.. (2000). Apoptotic Cell Death in Arrhythmogenic Right Ventricular Cardiomyopathy. A Comparative Study with Idiopathic Sustained Ventricular Tachycardia.. Japanese Heart Journal. 41(6). 733–741. 19 indexed citations

7.

Shimojo, Takashi, Michiaki Hiroe, Shigeru Ishiyama, et al.. (1999). Nitric Oxide Induces Apoptotic Death of Cardiomyocytes via a Cyclic-GMP-Dependent Pathway. Experimental Cell Research. 247(1). 38–47. 82 indexed citations

8.

Nishikawa, Toshio, Shigeru Ishiyama, Yasunari Sakomura, et al.. (1999). Ultrastructural features of the myocardium of children with dilated cardiomyopathy. Heart and Vessels. 14(1). 52–56. 3 indexed citations

9.

Ishiyama, Shigeru, et al.. (1999). Programmed Cell Death in the Myocardium of Arrhythmogenic Right Ventricular Cardiomyopathy in Children and Adults. Cardiovascular Pathology. 8(4). 185–189. 20 indexed citations

10.

Nishikawa, Toshio, Shigeru Ishiyama, Yasunari Sakomura, et al.. (1999). Histopathologic aspects of endomyocardial biopsy in pediatric patients with idiopathic ventricular tachycardia. Pediatrics International. 41(5). 534–537. 6 indexed citations

11.

Ito, Hiroshi, Takashi Shimojo, Hiroyuki Fujisaki, et al.. (1999). Thermal preconditioning protects rat cardiac muscle cells from doxorubicin-induced apoptosis. Life Sciences. 64(9). 755–761. 49 indexed citations

12.

Kotake, Shigeru, Nobuyuki Udagawa, Kenichiro MATSUZAKI, et al.. (1999). IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis. Journal of Clinical Investigation. 103(9). 1345–1352. 1386 indexed citations breakdown →

13.

Shimojo, Takashi, Toshio Nishikawa, Shigeru Ishiyama, et al.. (1998). Participation of Nitric Oxide and Peroxynitrite in the Development of Myocardial Tissue Damage in Acute Myocardial Infarction. Cardiovascular Pathology. 7(1). 25–30. 8 indexed citations

14.

Toyozaki, Tetsuya, Michiaki Hiroe, Toshihiro Saito, et al.. (1998). Levels of Soluble Fas in Patients With Myocarditis, Heart Failure of Unknown Origin, and in Healthy Volunteers. The American Journal of Cardiology. 81(6). 798–800. 40 indexed citations

15.

Ishiyama, Shigeru, Michiaki Hiroe, Toshio Nishikawa, et al.. (1998). The Fas/Fas Ligand System Is Involved in the Pathogenesis of Autoimmune Myocarditis in Rats. The Journal of Immunology. 161(9). 4695–4701. 57 indexed citations

16.

Amano, Jun, Shigeru Ishiyama, Hiroyuki Tanaka, et al.. (1997). Proliferation of smooth muscle cells in acute allograft vascular rejection. Journal of Thoracic and Cardiovascular Surgery. 113(1). 19–25. 15 indexed citations

17.

Nishikawa, Toshio, et al.. (1996). Hypertrophic cardiomyopathy in Noonan syndrome. Pediatrics International. 38(1). 91–98. 31 indexed citations

18.

Ishiyama, Shigeru, et al.. (1995). The effect of forskolin on the teratogenicity of methylxanthines in the chick embryo heart. Reproductive Toxicology. 9(2). 165–168. 2 indexed citations

19.

Hiroe, Michiaki, Shigeru Ishiyama, Toshio Nishikawa, et al.. (1995). 726-1 Expression of Inducible Nitric Oxide Synthase in the Myocardium of Acute Myocarditis – A Serial Cardiac Biopsy Study. Journal of the American College of Cardiology. 25(2). 131A–131A. 2 indexed citations

20.

Hiroe, Michiaki, Masato Tanaka, Toshio Nishikawa, et al.. (1994). Distribution of desmin filaments in the myocardium of the failing heart. 27(3-4). 246–248. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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