Matthew T. Gillespie

17.6k total citations · 2 hit papers
156 papers, 14.3k citations indexed

About

Matthew T. Gillespie is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Matthew T. Gillespie has authored 156 papers receiving a total of 14.3k indexed citations (citations by other indexed papers that have themselves been cited), including 121 papers in Molecular Biology, 84 papers in Oncology and 20 papers in Immunology. Recurrent topics in Matthew T. Gillespie's work include Bone health and treatments (73 papers), Bone Metabolism and Diseases (61 papers) and Antimicrobial Resistance in Staphylococcus (19 papers). Matthew T. Gillespie is often cited by papers focused on Bone health and treatments (73 papers), Bone Metabolism and Diseases (61 papers) and Antimicrobial Resistance in Staphylococcus (19 papers). Matthew T. Gillespie collaborates with scholars based in Australia, United States and Japan. Matthew T. Gillespie's co-authors include T. John Martin, T. John Martin, Nobuyuki Udagawa, J Quinn, Nicole J. Horwood, Jan Elliott, Naoyuki Takahashi, Eijiro Jimi, Tatsuo Suda and Kanami Itoh and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Matthew T. Gillespie

150 papers receiving 14.0k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Modulation of Osteoclast Differentiation and Function by the New Members of the Tumor Necrosis Factor Receptor and Ligand Families

1999 1.8k citations Nobuyuki Udagawa, Eijiro Jimi et al. profile →
IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis

1999 1.4k citations Nobuyuki Udagawa, Kanami Itoh et al. Journal of Clinical Investigation profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
Matthew T. Gillespie	9.6k	6.6k	2.3k	2.2k	2.0k	156	14.3k
F. Patrick Ross	10.8k 1.1×	6.0k 0.9×	2.5k 1.1×	2.4k 1.1×	2.3k 1.2×	196	16.8k
Hiroshi Takayanagi	11.8k 1.2×	6.9k 1.0×	5.5k 2.4×	3.1k 1.4×	2.3k 1.1×	163	19.1k
M. Neale Weitzmann	6.6k 0.7×	3.3k 0.5×	1.7k 0.7×	962 0.4×	2.7k 1.4×	135	10.6k
Hisataka Yasuda	11.1k 1.2×	7.7k 1.2×	1.7k 0.7×	2.1k 0.9×	2.8k 1.4×	118	14.7k
Tomoki Nakashima	7.0k 0.7×	3.9k 0.6×	2.4k 1.0×	1.7k 0.8×	1.5k 0.7×	133	11.4k
G. David Roodman	7.1k 0.7×	7.2k 1.1×	1.4k 0.6×	1.6k 0.7×	1.9k 0.9×	198	13.0k
Sunao Takeshita	5.5k 0.6×	2.8k 0.4×	1.1k 0.5×	991 0.4×	1.3k 0.7×	59	8.2k
Lianping Xing	8.6k 0.9×	5.3k 0.8×	1.8k 0.8×	2.2k 1.0×	2.1k 1.1×	177	14.2k
Hiroshi Takayanagi	7.8k 0.8×	4.4k 0.7×	2.4k 1.0×	1.6k 0.7×	1.5k 0.8×	58	10.9k
Dirk Anderson	7.0k 0.7×	5.2k 0.8×	8.6k 3.7×	1.0k 0.5×	852 0.4×	67	16.4k

Countries citing papers authored by Matthew T. Gillespie

Since Specialization

Citations

This map shows the geographic impact of Matthew T. Gillespie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew T. Gillespie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew T. Gillespie more than expected).

Fields of papers citing papers by Matthew T. Gillespie

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew T. Gillespie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew T. Gillespie. The network helps show where Matthew T. Gillespie may publish in the future.

Co-authorship network of co-authors of Matthew T. Gillespie

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew T. Gillespie. A scholar is included among the top collaborators of Matthew T. Gillespie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew T. Gillespie. Matthew T. Gillespie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

McKinstry, William J., Galina Polekhina, Koh Sato, et al.. (2009). Crystallization of the receptor-binding domain of parathyroid hormone-related protein in complex with a neutralizing monoclonal antibody Fab fragment. Acta Crystallographica Section F Structural Biology and Crystallization Communications. 65(4). 336–338. 4 indexed citations

2.

Kartsogiannis, Vicky, Natalie A. Sims, J Quinn, et al.. (2008). Osteoclast Inhibitory Lectin, an Immune Cell Product That Is Required for Normal Bone Physiology in Vivo. Journal of Biological Chemistry. 283(45). 30850–30860. 27 indexed citations

3.

Pompolo, S., et al.. (2008). Calcitonin Attenuates the Anabolic Effect of PTH in vivo and Rapidly Upregulates Sclerostin Expression. Journal of Bone and Mineral Research. 23. 1 indexed citations

4.

Quinn, J, Natalie A. Sims, Keith Thompson, et al.. (2008). IL-23 Inhibits Osteoclastogenesis Indirectly through Lymphocytes and Is Required for the Maintenance of Bone Mass in Mice. The Journal of Immunology. 181(8). 5720–5729. 77 indexed citations

5.

Gillespie, Matthew T., et al.. (2007). Active osteoclasts are required for PTH anabolic effects in young female rats. Bone. 40(6).

6.

Gillespie, Matthew T., et al.. (2006). Calcitonin attenuates the anabolic effect of PTH in young rats.. Journal of Bone and Mineral Research. 21. 2 indexed citations

7.

Romas, Evange & Matthew T. Gillespie. (2006). Inflammation-Induced Bone Loss: Can it Be Prevented?. Rheumatic Disease Clinics of North America. 32(4). 759–773. 66 indexed citations

8.

Price, John T., J Quinn, Natalie A. Sims, et al.. (2005). The HSP90 pharmacological inhibitor, 17-AAG, enhances breast-bone metastasis in a nude mouse model and stimulates in vitro and in vivo osteoclast formation. Cancer Treatment Reviews. 31. 1 indexed citations

9.

Sims, Natalie A., Brendan J. Jenkins, J Quinn, et al.. (2004). Glycoprotein 130 regulates bone turnover and bone size by distinct downstream signaling pathways. Journal of Clinical Investigation. 113(3). 379–389. 151 indexed citations

10.

Cingolani, Gino, Janna Bednenko, Matthew T. Gillespie, & Larry Gerace. (2002). Molecular Basis for the Recognition of a Nonclassical Nuclear Localization Signal by Importin β. Molecular Cell. 10(6). 1345–1353. 163 indexed citations

11.

Horwood, Nicole J., Jan Elliott, T. John Martin, & Matthew T. Gillespie. (2001). IL-12 Alone and in Synergy with IL-18 Inhibits Osteoclast Formation In Vitro. The Journal of Immunology. 166(8). 4915–4921. 201 indexed citations

12.

Udagawa, Nobuyuki, N. Takahashi, Eijiro Jimi, et al.. (1999). Osteoblasts/stromal cells stimulate osteoclast activation through expression of osteoclast differentiation factor/RANKL but not macrophage colony-stimulating factor. Bone. 25(5). 517–523. 358 indexed citations

13.

Kartsogiannis, Vicky, Hong Zhou, Nicole J. Horwood, et al.. (1999). Localization of RANKL (receptor activator of NFκB ligand) mRNA and protein in skeletal and extraskeletal tissues. Bone. 25(5). 525–534. 263 indexed citations

14.

Quinn, J, Maria Morfis, Mark H.C. Lam, et al.. (1999). Calcitonin receptor antibodies in the identification of osteoclasts. Bone. 25(1). 1–8. 76 indexed citations

15.

Horwood, Nicole J., Jan Elliott, T. John Martin, & Matthew T. Gillespie. (1998). Osteotropic Agents Regulate the Expression of Osteoclast Differentiation Factor and Osteoprotegerin in Osteoblastic Stromal Cells. Endocrinology. 139(11). 4743–4743. 357 indexed citations

16.

Gillespie, Matthew T., et al.. (1997). Calcitonin receptors, bone sialoprotein and osteopontin are expressed in primary breast cancers. International Journal of Cancer. 73(6). 812–815. 86 indexed citations

17.

Gillespie, Matthew T., et al.. (1997). Calcitonin receptors, bone sialoprotein and osteopontin are expressed in primary breast cancers. International Journal of Cancer. 73(6). 812–815. 7 indexed citations

18.

Moseley, Jane M., Matthew T. Gillespie, & Mark A. Thiede. (1995). Parathyroid Hormone-Related Protein. Critical Reviews in Clinical Laboratory Sciences. 32(3). 299–343. 151 indexed citations

19.

Gillespie, Matthew T., et al.. (1987). Homologous direct repeat sequences associated with mercury, methicillin, tetracycline and trimethoprim resistance determinants inStaphylococcus aureus. FEMS Microbiology Letters. 43(2). 165–171. 53 indexed citations

20.

Lyon, B. R., Matthew T. Gillespie, & Ronald A. Skurray. (1987). Detection and Characterization of IS256, an Insertion Sequence in Staphylococcus aureus. Microbiology. 133(11). 3031–3038. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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