K. Mikoshiba
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- Neuroscience and Neuropharmacology Research 10
- Sensory Systems top 2%
- Physiology top 2%
- Erythrocyte Function and Pathophysiology 2
- Developmental Neuroscience top 5%
- Neurogenesis and neuroplasticity mechanisms 3
- Cell Biology top 5%
- Cellular transport and secretion 6
- Endoplasmic Reticulum Stress and Disease 2
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- Ion channel regulation and function 11
- Protein Kinase Regulation and GTPase Signaling 3
- Receptor Mechanisms and Signaling 3
- Co-authors
- Teiichi FuruichiJun ArugaMichisuke YuzakiShinji NakadeHideyuki OkanoHiroo OguraKatsunori NakataTakeharu Nagai
- Partner nations
- JapanUnited StatesPoland
In The Last Decade
K. Mikoshiba
31 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 96
- Cellular and Molecular Neuroscience 754
- Sensory Systems 195
- Physiology 144
- Developmental Neuroscience 100
- Cell Biology 359
Countries citing papers authored by K. Mikoshiba
This map shows the geographic impact of K. Mikoshiba's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K. Mikoshiba with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K. Mikoshiba more than expected).
Fields of papers citing papers by K. Mikoshiba
This network shows the impact of papers produced by K. Mikoshiba. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K. Mikoshiba. The network helps show where K. Mikoshiba may publish in the future.
Co-authorship network
The 25 scholars most cited alongside K. Mikoshiba, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | IP3-induced calcium release engages a pro-hypertrophic autoamplifying loop that relieves miR-133a mediated suppression of IP3RII calcium channels | 2012 | 1 |
| 2 | 2003 | 26 | |
| 3 | 2001 | 58 | |
| 4 | 2001 | 3 | |
| 5 | 1999 | 148 | |
| 6 | 1999 | 13 | |
| 7 | 1999 | 16 | |
| 8 | 1998 | 147 | |
| 9 | 1997 | 58 | |
| 10 | 1996 | 47 | |
| 11 | 1996 | 49 | |
| 12 | 1996 | 30 | |
| 13 | 1994 | 14 | |
| 14 | 1994 | 64 | |
| 15 | 1994 | 20 | |
| 16 | 1993 | 197 | |
| 17 | 1993 | 11 | |
| 18 | 1992 | 32 | |
| 19 | 1992 | 121 | |
| 20 | 1989 | 6 |
About K. Mikoshiba
K. Mikoshiba is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Aging, having authored 31 papers that have together received 1.8k indexed citations. Recurring topics across this work include Ion channel regulation and function (11 papers), Neuroscience and Neuropharmacology Research (10 papers), Cellular transport and secretion (6 papers), Protein Kinase Regulation and GTPase Signaling (3 papers), Receptor Mechanisms and Signaling (3 papers), Neurogenesis and neuroplasticity mechanisms (3 papers), Erythrocyte Function and Pathophysiology (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (754 citations), Sensory Systems (195 citations) and Physiology (144 citations). K. Mikoshiba has collaborated with scholars based in Japan, United States and Poland. Frequent co-authors include Teiichi Furuichi, Jun Aruga, Michisuke Yuzaki, Shinji Nakade, Hideyuki Okano, Hiroo Ogura, Katsunori Nakata, Takeharu Nagai, Hiroki Hamanaka and Sang Ki Rhee. Their work appears in journals such as Cell, Journal of Biological Chemistry and Neuron.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.