Dagmar Wachten

3.1k total citations
60 papers, 1.9k citations indexed

About

Dagmar Wachten is a scholar working on Molecular Biology, Genetics and Reproductive Medicine. According to data from OpenAlex, Dagmar Wachten has authored 60 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 21 papers in Genetics and 16 papers in Reproductive Medicine. Recurrent topics in Dagmar Wachten's work include Genetic and Kidney Cyst Diseases (17 papers), Sperm and Testicular Function (16 papers) and Reproductive Biology and Fertility (14 papers). Dagmar Wachten is often cited by papers focused on Genetic and Kidney Cyst Diseases (17 papers), Sperm and Testicular Function (16 papers) and Reproductive Biology and Fertility (14 papers). Dagmar Wachten collaborates with scholars based in Germany, United States and Norway. Dagmar Wachten's co-authors include Jan N. Hansen, U. Benjamin Kaupp, Melanie Balbach, Timo Strünker, Jan F. Jikeli, Nathalie Jurisch‐Yaksi, Luis Álvarez, David U. Mick, Christoph Brenker and Christa Ringers and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Dagmar Wachten

54 papers receiving 1.8k citations

Peers

Dagmar Wachten

GENET

PHEOH

CMN

Johannes Wilbertz Sweden

Masayuki Tsuda Japan

C. Marc Luetjens United States

Renaud Legouis France

Mark S. Roberson United States

Yoshitaka Fujihara Japan

Anthony W.S. Chan United States

Tetsuya Sato Japan

Anne Guiochon‐Mantel France

F. Kent Hamra United States

Johannes Wilbertz Sweden

Dagmar Wachten

1.3k ×1.4

511 ×1.1

GENET

310 ×0.7

357 ×1.0

PHEOH

405 ×1.4

CMN

Citations per year, relative to Dagmar Wachten Dagmar Wachten (= 1×) peers Johannes Wilbertz

Countries citing papers authored by Dagmar Wachten

Since Specialization

Citations

This map shows the geographic impact of Dagmar Wachten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dagmar Wachten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dagmar Wachten more than expected).

Fields of papers citing papers by Dagmar Wachten

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dagmar Wachten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dagmar Wachten. The network helps show where Dagmar Wachten may publish in the future.

Co-authorship network of co-authors of Dagmar Wachten

This figure shows the co-authorship network connecting the top 25 collaborators of Dagmar Wachten. A scholar is included among the top collaborators of Dagmar Wachten based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dagmar Wachten. Dagmar Wachten is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wachten, Dagmar & Søren T. Christensen. (2025). Primary cilia signalling at a glance. Journal of Cell Science. 138(20).

Stahl, Rainer, et al.. (2025). Potent optogenetic regulation of gene expression in mammalian cells for bioproduction and basic research. Nucleic Acids Research. 53(12). 1 indexed citations

Wachten, Dagmar, et al.. (2025). Macrophages in metaflammation – fueling chronic inflammation in metabolic disease. Pflügers Archiv - European Journal of Physiology. 478(1). 10–10.

Ashraf, Azhaar, et al.. (2025). Renal tissue-resident macrophages promote cystogenesis in early polycystic kidney disease. Journal of Cell Science. 138(20).

Müller, Franziska E., Stefan Passlick, Dagmar Wachten, et al.. (2023). Induced Remodelling of Astrocytes In Vitro and In Vivo by Manipulation of Astrocytic RhoA Activity. Cells. 12(2). 331–331. 2 indexed citations

Schneider, Simon, Lena Arévalo, Jan N. Hansen, et al.. (2023). Cylicins are a structural component of the sperm calyx being indispensable for male fertility in mice and human. eLife. 12. 11 indexed citations

Wachten, Dagmar & Pleasantine Mill. (2023). The cilia mechanosensation debate gets (bio)physical. Nature Reviews Nephrology. 19(5). 279–280. 5 indexed citations

Feistel, Kerstin, Benjamin M. Friedrich, Anne Grapin‐Botton, et al.. (2023). Emerging principles of primary cilia dynamics in controlling tissue organization and function. The EMBO Journal. 42(21). e113891–e113891. 25 indexed citations

Latz, Eicke, et al.. (2022). Primary cilia and their effects on immune cell functions and metabolism: a model. Trends in Immunology. 43(5). 366–378. 12 indexed citations

10.

Balbach, Melanie, Navpreet Kaur, Carla Ritagliati, et al.. (2021). Soluble adenylyl cyclase inhibition prevents human sperm functions essential for fertilization. Molecular Human Reproduction. 27(9). 44 indexed citations

11.

Hansen, Jan N., et al.. (2021). CiliaQ: a simple, open-source software for automated quantification of ciliary morphology and fluorescence in 2D, 3D, and 4D images. The European Physical Journal E. 44(2). 18–18. 44 indexed citations

12.

Bonaguro, Lorenzo, Nadine Spielmann, Patricia da Silva‐Buttkus, et al.. (2021). Creld1 regulates myocardial development and function. Journal of Molecular and Cellular Cardiology. 156. 45–56. 11 indexed citations

13.

Oura, Seiya, Kaori Nozawa, Julio M Castaneda, et al.. (2020). Cfap97d1 is important for flagellar axoneme maintenance and male mouse fertility. PLoS Genetics. 16(8). e1008954–e1008954. 14 indexed citations

14.

Schiffer, Christian, Christoph Brenker, Hussein Hamzeh, et al.. (2020). Rotational motion and rheotaxis of human sperm do not require functional CatSper channels and transmembrane Ca ²⁺ signaling. The EMBO Journal. 39(4). e102363–e102363. 43 indexed citations

15.

Balbach, Melanie, Hussein Hamzeh, Jan F. Jikeli, et al.. (2020). Molecular Mechanism Underlying the Action of Zona-pellucida Glycoproteins on Mouse Sperm. Frontiers in Cell and Developmental Biology. 8. 572735–572735. 20 indexed citations

16.

Guo, Jiami, James M. Otis, Lei Xing, et al.. (2019). Primary Cilia Signaling Promotes Axonal Tract Development and Is Disrupted in Joubert Syndrome-Related Disorders Models. Developmental Cell. 51(6). 759–774.e5. 65 indexed citations

17.

Körschen, Heinz G., Anke Penno, Andreas Rennhack, et al.. (2017). Identification of a feedback loop involving β-glucosidase 2 and its product sphingosine sheds light on the molecular mechanisms in Gaucher disease. Journal of Biological Chemistry. 292(15). 6177–6189. 18 indexed citations

18.

Mass, Elvira, Dagmar Wachten, Anna C. Aschenbrenner, André Voelzmann, & Michael Hoch. (2014). Murine Creld1 Controls Cardiac Development through Activation of Calcineurin/NFATc1 Signaling. Developmental Cell. 28(6). 711–726. 24 indexed citations

19.

Rabionet, Mariona, Christian Marsching, Richard Jennemann, et al.. (2013). 1-O-acylceramides are natural components of human and mouse epidermis. Journal of Lipid Research. 54(12). 3312–3321. 64 indexed citations

20.

Drawnel, Faye, Dagmar Wachten, Jeffery D. Molkentin, et al.. (2012). IP3-induced calcium release engages a pro-hypertrophic autoamplifying loop that relieves miR-133a mediated suppression of IP3RII calcium channels. Cardiovascular Research. 93. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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