Srikumar Chellappan

9.6k total citations · 2 hit papers
108 papers, 7.9k citations indexed

About

Srikumar Chellappan is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Srikumar Chellappan has authored 108 papers receiving a total of 7.9k indexed citations (citations by other indexed papers that have themselves been cited), including 85 papers in Molecular Biology, 56 papers in Oncology and 20 papers in Cell Biology. Recurrent topics in Srikumar Chellappan's work include Cancer-related Molecular Pathways (42 papers), Ubiquitin and proteasome pathways (18 papers) and Microtubule and mitosis dynamics (15 papers). Srikumar Chellappan is often cited by papers focused on Cancer-related Molecular Pathways (42 papers), Ubiquitin and proteasome pathways (18 papers) and Microtubule and mitosis dynamics (15 papers). Srikumar Chellappan collaborates with scholars based in United States, Canada and India. Srikumar Chellappan's co-authors include Joseph R. Nevins, Scott W. Hiebert, Maria Mudryj, Jonathan M. Horowitz, Courtney Schaal, Smitha Pillai, Piyali Dasgupta, Eric B. Haura, Sandeep Singh and Domenico Coppola and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Srikumar Chellappan

105 papers receiving 7.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The E2F transcription factor is a cellular target for the RB protein

1991 1.2k citations Srikumar Chellappan et al. profile →
The interaction of RB with E2F coincides with an inhibition of the transcriptional activity of E2F.

1992 516 citations Srikumar Chellappan et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Srikumar Chellappan United States	46	5.5k	3.4k	1.1k	952	705	108	7.9k
Grazia Ambrosini United States	27	4.8k 0.9×	2.2k 0.7×	743 0.7×	386 0.4×	692 1.0×	48	6.8k
Chuan‐Yuan Li United States	40	3.6k 0.7×	1.6k 0.5×	1.4k 1.3×	450 0.5×	476 0.7×	115	6.0k
Jason D. Weber United States	39	6.2k 1.1×	3.9k 1.1×	1.4k 1.3×	566 0.6×	688 1.0×	78	8.9k
Qiang Yu Singapore	55	6.0k 1.1×	1.9k 0.5×	1.9k 1.7×	552 0.6×	651 0.9×	139	8.4k
David Pei‐Cheng Lin Taiwan	20	6.7k 1.2×	6.2k 1.8×	1.6k 1.4×	679 0.7×	739 1.0×	59	9.5k
Masatoshi Kitagawa Japan	46	5.9k 1.1×	2.7k 0.8×	1.8k 1.7×	575 0.6×	1.0k 1.4×	144	7.8k
Alfonso Bellacosa United States	52	10.8k 2.0×	4.0k 1.2×	2.3k 2.1×	1.1k 1.2×	1.1k 1.5×	118	14.4k
Hallgeir Rui United States	50	3.1k 0.6×	3.5k 1.0×	1.3k 1.2×	894 0.9×	405 0.6×	204	6.8k
W. Douglas Cress United States	38	4.8k 0.9×	2.8k 0.8×	1.2k 1.1×	775 0.8×	389 0.6×	86	6.8k
Anthony P. Albino United States	48	3.4k 0.6×	2.5k 0.8×	1.2k 1.1×	527 0.6×	689 1.0×	115	6.6k

Countries citing papers authored by Srikumar Chellappan

Since Specialization

Citations

This map shows the geographic impact of Srikumar Chellappan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Srikumar Chellappan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Srikumar Chellappan more than expected).

Fields of papers citing papers by Srikumar Chellappan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Srikumar Chellappan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Srikumar Chellappan. The network helps show where Srikumar Chellappan may publish in the future.

Co-authorship network of co-authors of Srikumar Chellappan

This figure shows the co-authorship network connecting the top 25 collaborators of Srikumar Chellappan. A scholar is included among the top collaborators of Srikumar Chellappan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Srikumar Chellappan. Srikumar Chellappan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Padmanabhan, Jaya, Patrick T. Grogan, Derek R. Duckett, et al.. (2025). STEM-16. COMBINATION THERAPIES WITH CDK9 INHIBITORS AS A PROMISING STRATEGY TO OVERCOME THERAPY RESISTANCE IN GLIOBLASTOMAS. Neuro-Oncology. 27(Supplement_5). v81–v81.

Bora-Singhal, Namrata, et al.. (2022). A Novel PHD2/VHL-mediated Regulation of YAP1 Contributes to VEGF Expression and Angiogenesis. Cancer Research Communications. 2(7). 624–638. 2 indexed citations

Jaiswal, Neha, Jiannong Li, Dung‐Tsa Chen, et al.. (2022). Ethnic and racial-specific differences in levels of centrosome-associated mitotic kinases, proliferative and epithelial-to-mesenchymal markers in breast cancers. Cell Division. 17(1). 6–6. 4 indexed citations

Faião‐Flores, Fernanda, Michael F. Emmons, Michael Durante, et al.. (2019). HDAC Inhibition Enhances the In Vivo Efficacy of MEK Inhibitor Therapy in Uveal Melanoma. Clinical Cancer Research. 25(18). 5686–5701. 69 indexed citations

Lin, Shengchen, Chongbiao Huang, Venugopal Gunda, et al.. (2019). Fascin Controls Metastatic Colonization and Mitochondrial Oxidative Phosphorylation by Remodeling Mitochondrial Actin Filaments. Cell Reports. 28(11). 2824–2836.e8. 55 indexed citations

Pillai, Smitha, José G. Treviño, Bhupendra Rawal, et al.. (2015). β-Arrestin-1 Mediates Nicotine-Induced Metastasis through E2F1 Target Genes That Modulate Epithelial–Mesenchymal Transition. Cancer Research. 75(6). 1009–1020. 73 indexed citations

Treviño, José G., Monika Verma, Sandeep Singh, et al.. (2013). Selective Disruption of Rb–Raf-1 Kinase Interaction Inhibits Pancreatic Adenocarcinoma Growth Irrespective of Gemcitabine Sensitivity. Molecular Cancer Therapeutics. 12(12). 2722–2734. 6 indexed citations

Davis, Rebecca K., Smitha Pillai, Nicholas J. Lawrence, & Srikumar Chellappan. (2012). TNF-α-mediated proliferation of vascular smooth muscle cells involves Raf-1-mediated inactivation of Rb and transcription of E2F1-regulated genes. Cell Cycle. 11(1). 109–118. 37 indexed citations

Rastogi, S., Wasia Rizwani, Bharat Joshi, Sateesh Kunigal, & Srikumar Chellappan. (2011). TNF-α response of vascular endothelial and vascular smooth muscle cells involve differential utilization of ASK1 kinase and p73. Cell Death and Differentiation. 19(2). 274–283. 62 indexed citations

10.

Pillai, Smitha, Michelle Kovacs, & Srikumar Chellappan. (2010). Regulation of Vascular Endothelial Growth Factor Receptors by Rb and E2F1: Role of Acetylation. Cancer Research. 70(12). 4931–4940. 56 indexed citations

11.

Kinkade, Rebecca, Piyali Dasgupta, Adam Carie, et al.. (2008). A Small Molecule Disruptor of Rb/Raf-1 Interaction Inhibits Cell Proliferation, Angiogenesis, and Growth of Human Tumor Xenografts in Nude Mice. Cancer Research. 68(10). 3810–3818. 45 indexed citations

12.

Pillai, Smitha & Srikumar Chellappan. (2007). Prohibitin 2 physically interacts with E2F1 and represses transcription. Cancer Research. 67. 1241–1241. 1 indexed citations

13.

Dasgupta, Piyali, et al.. (2006). Nicotine inhibits apoptosis induced by chemotherapeutic drugs by up-regulating XIAP and survivin. Proceedings of the National Academy of Sciences. 103(16). 6332–6337. 248 indexed citations

14.

Rastogi, S., Bharat Joshi, Piyali Dasgupta, et al.. (2006). Prohibitin Facilitates Cellular Senescence by Recruiting Specific Corepressors To Inhibit E2F Target Genes. Molecular and Cellular Biology. 26(11). 4161–4171. 75 indexed citations

15.

Clerck, Yves A. De, Bernard E. Weissman, Dihua Yu, et al.. (2006). Tumor progression and metastasis from genetic to microenvironmental determinants. Cancer Biology & Therapy. 5(12). 1588–1599. 1 indexed citations

16.

Adnane, Jalila, Bharat Joshi, Da Wang, et al.. (2006). Farnesyltransferase and geranylgeranyltransferase I inhibitors upregulate RhoB expression by HDAC1 dissociation, HAT association and histone acetylation of the RhoB promoter. Oncogene. 26(5). 633–640. 54 indexed citations

17.

Fusaro, Gina, Sheng Wang, & Srikumar Chellappan. (2002). Differential regulation of Rb family proteins and prohibitin during camptothecin-induced apoptosis. Oncogene. 21(29). 4539–4548. 91 indexed citations

18.

Wang, Sheng, Niharika Nath, Gina Fusaro, & Srikumar Chellappan. (1999). Rb and Prohibitin Target Distinct Regions of E2F1 for Repression and Respond to Different Upstream Signals. Molecular and Cellular Biology. 19(11). 7447–7460. 139 indexed citations

19.

Wang, Sheng, Richik N. Ghosh, & Srikumar Chellappan. (1998). Raf-1 Physically Interacts with Rb and Regulates Its Function: a Link between Mitogenic Signaling and Cell Cycle Regulation. Molecular and Cellular Biology. 18(12). 7487–7498. 106 indexed citations

20.

Wong, Kwok‐Kin, Xiaoming Zou, Kevin T. Merrell, et al.. (1995). v-Abl Activates c- myc Transcription through the E2F Site. Molecular and Cellular Biology. 15(12). 6535–6544. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact