John G. Lewis

6.3k total citations
171 papers, 4.7k citations indexed

About

John G. Lewis is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Behavioral Neuroscience. According to data from OpenAlex, John G. Lewis has authored 171 papers receiving a total of 4.7k indexed citations (citations by other indexed papers that have themselves been cited), including 68 papers in Endocrinology, Diabetes and Metabolism, 36 papers in Molecular Biology and 31 papers in Behavioral Neuroscience. Recurrent topics in John G. Lewis's work include Adrenal Hormones and Disorders (41 papers), Stress Responses and Cortisol (31 papers) and Hormonal and reproductive studies (31 papers). John G. Lewis is often cited by papers focused on Adrenal Hormones and Disorders (41 papers), Stress Responses and Cortisol (31 papers) and Hormonal and reproductive studies (31 papers). John G. Lewis collaborates with scholars based in New Zealand, United Kingdom and Australia. John G. Lewis's co-authors include Peter A. Elder, David J. Torpy, P.A. Elder, Ruth Feldman, Orna Zagoory‐Sharon, Aron Weller, Ari Levine, Jui T. Ho, R. Ghanadian and Warrick J. Inder and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Nucleic Acids Research.

In The Last Decade

John G. Lewis

169 papers receiving 4.5k citations

Peers

John G. Lewis
Theodore C. Friedman United States
Robert T. Chatterton United States
Giuseppe Delitala Italy
J. Landon United Kingdom
Beverley E. Pearson Murphy Canada
John W. Honour United Kingdom
Egil Haug Norway
Andrew N. Margioris Greece
Evangelia Charmandari Greece
David J. Torpy Australia
Theodore C. Friedman United States
John G. Lewis
Citations per year, relative to John G. Lewis John G. Lewis (= 1×) peers Theodore C. Friedman

Countries citing papers authored by John G. Lewis

Since Specialization
Citations

This map shows the geographic impact of John G. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John G. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John G. Lewis more than expected).

Fields of papers citing papers by John G. Lewis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John G. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John G. Lewis. The network helps show where John G. Lewis may publish in the future.

Co-authorship network of co-authors of John G. Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of John G. Lewis. A scholar is included among the top collaborators of John G. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John G. Lewis. John G. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Slow, Sandy, John Pearson, Christopher M Florkowski, et al.. (2020). Effect of genetic factors on the response to vitamin D3 supplementation in the VIDARIS randomized controlled trial. Nutrition. 75-76. 110761–110761. 7 indexed citations
2.
Nenke, Marni A., Emily J. Meyer, John G. Lewis, et al.. (2017). Differential Effects of Estrogen on Corticosteroid-Binding Globulin Forms Suggests Reduced Cleavage in Pregnancy. Journal of the Endocrine Society. 1(3). 202–210. 23 indexed citations
3.
Lewis, John G. & Peter A. Elder. (2017). Monoclonal antibodies to the reactive centre loop (RCL) of human corticosteroid-binding globulin (CBG) can protect against proteolytic cleavage. The Journal of Steroid Biochemistry and Molecular Biology. 171. 247–253. 1 indexed citations
4.
Lindsay, Angus, John G. Lewis, Nicholas Gill, Steven P. Gieseg, & Nick Draper. (2015). Immunity, inflammatory and psychophysiological stress response during a competition of professional rugby union. SHILAP Revista de lepidopterología. 26(4). 153–160. 6 indexed citations
5.
Nenke, Marni A., Mark Holmes, Wayne Rankin, John G. Lewis, & David J. Torpy. (2015). Corticosteroid-binding globulin cleavage is paradoxically reduced in alpha-1 antitrypsin deficiency: Implications for cortisol homeostasis. Clinica Chimica Acta. 452. 27–31. 17 indexed citations
6.
Rankin, Wayne, Marianne J. Chapman, Natalie E. Stevens, et al.. (2014). Depletion of high‐affinity corticosteroid‐binding globulin corresponds to illness severity in sepsis and septic shock; clinical implications. Clinical Endocrinology. 82(6). 801–807. 45 indexed citations
7.
Simard, Marc, Lesley A. Hill, John G. Lewis, & Geoffrey L. Hammond. (2014). Naturally Occurring Mutations of Human Corticosteroid-Binding Globulin. The Journal of Clinical Endocrinology & Metabolism. 100(1). E129–E139. 26 indexed citations
8.
Ho, J. T., Jennifer Keogh, SR Bornstein, et al.. (2007). Moderate Weight Loss Reduces Renin and Aldosterone but does not Influence Basal or Stimulated Pituitary-adrenal Axis Function. Hormone and Metabolic Research. 39(9). 694–699. 46 indexed citations
9.
Ho, J. T., M. John Chapman, T. Quach, et al.. (2006). Septic Shock and Sepsis: A Comparison of Total and Free Plasma Cortisol Levels. The Journal of Clinical Endocrinology & Metabolism. 91(1). 105–114. 234 indexed citations
10.
Lewis, John G., Christopher J. Bagley, Peter A. Elder, Anthony W. Bachmann, & David J. Torpy. (2005). Plasma free cortisol fraction reflects levels of functioning corticosteroid-binding globulin. Clinica Chimica Acta. 359(1-2). 189–194. 165 indexed citations
11.
Lewis, John G., Brett Shand, P.A. Elder, & Russell Scott. (2004). Plasma sex hormone‐binding globulin rather than corticosteroid‐binding globulin is a marker of insulin resistance in obese adult males. Diabetes Obesity and Metabolism. 6(4). 259–263. 22 indexed citations
12.
13.
Lewis, John G., et al.. (2000). Lack of Effect of Dietary Chromium Supplementation on Glucose Tolerance, Plasma Insulin and Lipoprotein Levels in Patients with Type 2 Diabetes. International Journal for Vitamin and Nutrition Research. 70(1). 14–18. 37 indexed citations
14.
Lewis, John G., Peter M. George, & Peter A. Elder. (1997). Plasma androsterone/epiandrosterone sulfates as markers of 5α-reductase activity: Effect of finasteride in normal men. Steroids. 62(8-9). 632–635. 13 indexed citations
15.
16.
Elder, Peter A., et al.. (1990). Use of a monoclonal antibody to estrone-3-glucuronide in an enzyme-linked immunosorbent assay (ELISA). Journal of Steroid Biochemistry. 36(5). 439–443. 13 indexed citations
17.
Cairns, S R, et al.. (1985). Persistent nephrogenic diabetes insipidus, hyperparathyroidism, and hypothyroidism after lithium treatment.. BMJ. 290(6467). 516–517. 20 indexed citations
18.
Ghanadian, R., et al.. (1977). Androgen concentrations in prostate and serum of the male and female Praomys (Mastomys) natalensis.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 15(3). 212–4. 5 indexed citations
19.
Lewis, John G.. (1962). Gout, Steatorrhoea, and Megaloblastic Anaemia. Annals of the Rheumatic Diseases. 21(3). 284–286. 5 indexed citations
20.
Lewis, John G. & C Symons. (1958). VASCULAR DISEASE IN A DIABETIC CLINIC. The Lancet. 272(7054). 985–988. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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