Thomas M. Magin

11.2k total citations · 2 hit papers
135 papers, 8.8k citations indexed

About

Thomas M. Magin is a scholar working on Cell Biology, Molecular Biology and Urology. According to data from OpenAlex, Thomas M. Magin has authored 135 papers receiving a total of 8.8k indexed citations (citations by other indexed papers that have themselves been cited), including 109 papers in Cell Biology, 66 papers in Molecular Biology and 33 papers in Urology. Recurrent topics in Thomas M. Magin's work include Skin and Cellular Biology Research (105 papers), Cellular Mechanics and Interactions (38 papers) and Hair Growth and Disorders (33 papers). Thomas M. Magin is often cited by papers focused on Skin and Cellular Biology Research (105 papers), Cellular Mechanics and Interactions (38 papers) and Hair Growth and Disorders (33 papers). Thomas M. Magin collaborates with scholars based in Germany, United States and United Kingdom. Thomas M. Magin's co-authors include Rudolf E. Leube, Michael Hesse, Werner W. Franke, Julia Reichelt, Meçhthild Hatzfeld, David W. Melton, Klaus Weber, Reinhard Windoffer, Preethi Vijayaraj and Kristin Seltmann and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Thomas M. Magin

132 papers receiving 8.6k citations

Hit Papers

New consensus nomenclature for mammalian keratins 2006 2026 2012 2019 2006 2020 100 200 300 400 500
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. New consensus nomenclature for mammalian keratins
    2006 544 citations E. Birgitte Lane, Thomas M. Magin et al. profile →
  2. Epidermolysis bullosa
    2020 210 citations Cristina Has, Thomas M. Magin et al. profile →

Peers

Thomas M. Magin
Michele De Luca Italy
John Couchman United States
Lutz Langbein Germany
Akemi Ishida‐Yamamoto Japan
Giovanna Zambruno Italy
Daniel Hohl Switzerland
Yann Barrandon Switzerland
Maksim V. Plikus United States
David T. Woodley United States
R. Moll Germany
Michele De Luca Italy
Thomas M. Magin
Citations per year, relative to Thomas M. Magin Thomas M. Magin (= 1×) peers Michele De Luca

Countries citing papers authored by Thomas M. Magin

Since Specialization
Citations

This map shows the geographic impact of Thomas M. Magin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas M. Magin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas M. Magin more than expected).

Fields of papers citing papers by Thomas M. Magin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas M. Magin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas M. Magin. The network helps show where Thomas M. Magin may publish in the future.

Co-authorship network of co-authors of Thomas M. Magin

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas M. Magin. A scholar is included among the top collaborators of Thomas M. Magin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas M. Magin. Thomas M. Magin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Russo, Jacopo Di, Thomas M. Magin, & Rudolf E. Leube. (2023). A keratin code defines the textile nature of epithelial tissue architecture. Current Opinion in Cell Biology. 85. 102236–102236. 3 indexed citations
3.
Nahaboo, Wallis, Marjorie Vermeersch, Marie Versaevel, et al.. (2022). Keratin filaments mediate the expansion of extra‐embryonic membranes in the post‐gastrulation mouse embryo. The EMBO Journal. 41(7). e108747–e108747. 12 indexed citations
4.
Jahnke, Heinz‐Georg, Matthias Rübsam, Eric W. Lin, et al.. (2022). Kinase Inhibition by PKC412 Prevents Epithelial Sheet Damage in Autosomal Dominant Epidermolysis Bullosa Simplex through Keratin and Cell Contact Stabilization. Journal of Investigative Dermatology. 142(12). 3282–3293. 7 indexed citations
5.
Fujiwara, Sachiko, Shinji Deguchi, & Thomas M. Magin. (2020). Disease-associated keratin mutations reduce traction forces and compromise adhesion and collective migration. Journal of Cell Science. 133(14). 19 indexed citations
6.
Bouameur, Jamal-Eddine & Thomas M. Magin. (2017). Lessons from Animal Models of Cytoplasmic Intermediate Filament Proteins. Sub-cellular biochemistry. 82. 171–230. 17 indexed citations
7.
Hering, Lars, et al.. (2016). Novel origin of lamin-derived cytoplasmic intermediate filaments in tardigrades. eLife. 5. e11117–e11117. 20 indexed citations
8.
Schwarz, Nicole, Georg Dreissen, Rudolf E. Leube, et al.. (2015). Distinct Impact of Two Keratin Mutations Causing Epidermolysis Bullosa Simplex on Keratinocyte Adhesion and Stiffness. Journal of Investigative Dermatology. 135(10). 2437–2445. 41 indexed citations
9.
Magin, Thomas M., et al.. (2015). Keratin Isotypes Control Desmosome Stability and Dynamics through PKCα. Journal of Investigative Dermatology. 136(1). 202–213. 42 indexed citations
10.
Käs, Josef A., Anatol W. Fritsch, Kristin Seltmann, & Thomas M. Magin. (2014). Keratins Significantly Contribute to Cell Stiffness and Impact Invasive Behavior. Biophysical Journal. 106(2). 574a–575a. 5 indexed citations
11.
Fabris, Gloria, Reinhard Windoffer, Nicole Schwarz, et al.. (2013). Keratins as the main component for the mechanical integrity of keratinocytes. Proceedings of the National Academy of Sciences. 110(46). 18513–18518. 176 indexed citations
12.
Seltmann, Kristin, Anatol W. Fritsch, Josef A. Käs, & Thomas M. Magin. (2013). Keratins significantly contribute to cell stiffness and impact invasive behavior. Proceedings of the National Academy of Sciences. 110(46). 18507–18512. 200 indexed citations
13.
Roth, Wera, et al.. (2013). Skin Fragility and Impaired Desmosomal Adhesion in Mice Lacking All Keratins. Journal of Investigative Dermatology. 134(4). 1012–1022. 36 indexed citations
14.
Duan, Yuanyuan, Ying Sun, Fan Zhang, et al.. (2012). Keratin K18 Increases Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Surface Expression by Binding to Its C-terminal Hydrophobic Patch. Journal of Biological Chemistry. 287(48). 40547–40559. 21 indexed citations
15.
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Hesse, Michael, et al.. (2005). Rescue of keratin 18/19 doubly deficient mice using aggregation with tetraploid embryos. European Journal of Cell Biology. 84(2-3). 355–361. 8 indexed citations
17.
Windoffer, Reinhard, et al.. (2003). Epidermolysis Bullosa Simplex-Type Mutations Alter the Dynamics of the Keratin Cytoskeleton and Reveal a Contribution of Actin to the Transport of Keratin Subunits. Molecular Biology of the Cell. 15(3). 990–1002. 84 indexed citations
18.
Porter, Rebecca M., Declan P. Lunny, Neil Wilson, et al.. (2002). Defolliculated (Dfl): A Dominant Mouse Mutation Leading to Poor Sebaceous Gland Differentiation and Total Elimination of Pelage Follicles. Journal of Investigative Dermatology. 119(1). 32–37. 45 indexed citations
19.
Theis, Martin, Thomas M. Magin, Achim Plum, & Klaus Willecke. (2000). General or Cell Type-Specific Deletion and Replacement of Connexin-Coding DNA in the Mouse. Methods. 20(2). 205–218. 53 indexed citations
20.
Plum, Achim, Thomas M. Magin, Frank Dombrowski, et al.. (2000). Unique and shared functions of different connexins in mice. Current Biology. 10(18). 1083–1091. 222 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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