Jeff Blackinton
- Neurology top 1%
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 5%
- Physiology top 5%
- Neurology top 5%
- Co-authors
- Mark CooksonKelly Jean Thomas CraigRili AhmadAlexandra BeilinaDavid W. MillerElisa GreggioMarcel P. van der BrugJack D. Keene
- Topics
- Parkinson's Disease Mechanisms and Treatments (7 papers)Nuclear Receptors and Signaling (4 papers)RNA regulation and disease (3 papers)
- Journals
- Proceedings of the National Academy of SciencesNucleic Acids ResearchJournal of Biological Chemistry
- Partner nations
- United StatesSwedenUnited Kingdom
In The Last Decade
Jeff Blackinton
11 papers receiving 1.7k citations
Hit Papers
Peers
Comparison fields: 5 of 87
- Neurology 1.1k
- Molecular Biology 818
- Cellular and Molecular Neuroscience 421
- Physiology 385
- Neurology 282
Countries citing papers authored by Jeff Blackinton
This map shows the geographic impact of Jeff Blackinton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jeff Blackinton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jeff Blackinton more than expected).
Fields of papers citing papers by Jeff Blackinton
This network shows the impact of papers produced by Jeff Blackinton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jeff Blackinton. The network helps show where Jeff Blackinton may publish in the future.
Co-authorship network of co-authors of Jeff Blackinton
This figure shows the co-authorship network connecting the top 25 collaborators of Jeff Blackinton. A scholar is included among the top collaborators of Jeff Blackinton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jeff Blackinton. Jeff Blackinton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 22 | |
| 2 | 20 | |
| 3 | 83 | |
| 4 | 371 | |
| 5 | 67 | |
| 6 | 243 | |
| 7 | 173 | |
| 8 | 45 | |
| 9 | Kinase activity is required for the toxic effects of mutant LRRK2/dardarinbreakdown → | 591 |
| 10 | 66 | |
| 11 | 3 |
About Jeff Blackinton
Jeff Blackinton is a scholar working on Neurology, Cellular and Molecular Neuroscience and Virology, having authored 11 papers that have together received 1.7k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (7 papers), Nuclear Receptors and Signaling (4 papers) and RNA regulation and disease (3 papers). The work is most often cited by research in Neurology (1.1k citations), Neurology (282 citations) and Cellular and Molecular Neuroscience (421 citations). Jeff Blackinton has collaborated with scholars based in United States, Sweden and United Kingdom. Frequent co-authors include Mark Cookson, Kelly Jean Thomas Craig, Rili Ahmad, Alexandra Beilina, David W. Miller, Elisa Greggio, Marcel P. van der Brug, Jack D. Keene, Rina Bandopadhyay and Anna Sandebring. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.