Alyse Frisbee

500 total citations
11 papers, 392 citations indexed

About

Alyse Frisbee is a scholar working on Immunology, Infectious Diseases and Surgery. According to data from OpenAlex, Alyse Frisbee has authored 11 papers receiving a total of 392 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 5 papers in Infectious Diseases and 3 papers in Surgery. Recurrent topics in Alyse Frisbee's work include Clostridium difficile and Clostridium perfringens research (5 papers), Immune Cell Function and Interaction (4 papers) and HIV Research and Treatment (3 papers). Alyse Frisbee is often cited by papers focused on Clostridium difficile and Clostridium perfringens research (5 papers), Immune Cell Function and Interaction (4 papers) and HIV Research and Treatment (3 papers). Alyse Frisbee collaborates with scholars based in United States, South Africa and Canada. Alyse Frisbee's co-authors include William A. Petri, Mayuresh M. Abhyankar, Jhansi L. Leslie, Z. Jennie, Morgan Simpson, Mahmoud Saleh, Carrie A. Cowardin, Erica L. Buonomo, Kenneth W. Scully and Patcharin Pramoonjago and has published in prestigious journals such as Nature Communications, Journal of Virology and Scientific Reports.

In The Last Decade

Alyse Frisbee

11 papers receiving 388 citations

Peers

Alyse Frisbee
Shay Leary Australia
Stella G. Hoft United States
Fiona Eyers Australia
Shay Leary Australia
Alyse Frisbee
Citations per year, relative to Alyse Frisbee Alyse Frisbee (= 1×) peers Shay Leary

Countries citing papers authored by Alyse Frisbee

Since Specialization
Citations

This map shows the geographic impact of Alyse Frisbee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alyse Frisbee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alyse Frisbee more than expected).

Fields of papers citing papers by Alyse Frisbee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alyse Frisbee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alyse Frisbee. The network helps show where Alyse Frisbee may publish in the future.

Co-authorship network of co-authors of Alyse Frisbee

This figure shows the co-authorship network connecting the top 25 collaborators of Alyse Frisbee. A scholar is included among the top collaborators of Alyse Frisbee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alyse Frisbee. Alyse Frisbee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Uddin, Md. Jashim, Jhansi L. Leslie, Stacey L. Burgess, et al.. (2021). The IL-33-ILC2 pathway protects from amebic colitis. Mucosal Immunology. 15(1). 165–175. 17 indexed citations
2.
Frisbee, Alyse & William A. Petri. (2020). Considering the Immune System during Fecal Microbiota Transplantation for Clostridioides difficile Infection. Trends in Molecular Medicine. 26(5). 496–507. 25 indexed citations
3.
Simpson, Morgan, Alyse Frisbee, Pankaj Kumar, et al.. (2020). Clostridioides difficile Binary Toxin Is Recognized by the Toll-Like Receptor 2/6 Heterodimer to Induce a Nuclear Factor-κB Response. The Journal of Infectious Diseases. 225(7). 1296–1300. 13 indexed citations
4.
Abhyankar, Mayuresh M., Z. Jennie, Kenneth W. Scully, et al.. (2020). Immune Profiling To Predict Outcome of Clostridioides difficile Infection. mBio. 11(3). 24 indexed citations
5.
Saleh, Mahmoud, Alyse Frisbee, Jhansi L. Leslie, et al.. (2019). Colitis-Induced Th17 Cells Increase the Risk for Severe Subsequent Clostridium difficile Infection. Cell Host & Microbe. 25(5). 756–765.e5. 76 indexed citations
6.
Frisbee, Alyse, Mahmoud Saleh, Mary K. Young, et al.. (2019). IL-33 drives group 2 innate lymphoid cell-mediated protection during Clostridium difficile infection. Nature Communications. 10(1). 2712–2712. 109 indexed citations
7.
Frisbee, Alyse, Todd Bradley, William J. Faison, et al.. (2019). HLA class II-Restricted CD8+ T cells in HIV-1 Virus Controllers. Scientific Reports. 9(1). 10165–10165. 9 indexed citations
8.
Hancock, Gemma, Hongbing Yang, Emma Wainwright, et al.. (2015). Identification of Effective Subdominant Anti-HIV-1 CD8+ T Cells Within Entire Post-infection and Post-vaccination Immune Responses. PLoS Pathogens. 11(2). e1004658–e1004658. 37 indexed citations
9.
Esrick, Erica B., Marie McConkey, Katherine I. Lin, Alyse Frisbee, & Benjamin L. Ebert. (2015). Inactivation of HDAC1 or HDAC2 induces gamma globin expression without altering cell cycle or proliferation. American Journal of Hematology. 90(7). 624–628. 23 indexed citations
10.
Blackinton, Jeff, Alyse Frisbee, Joy Pickeral, et al.. (2014). Transcriptional and Posttranscriptional Regulation of Cytokine Gene Expression in HIV-1 Antigen-Specific CD8+T Cells That Mediate Virus Inhibition. Journal of Virology. 88(17). 9514–9528. 20 indexed citations
11.
Kefas, Benjamin, Desiree H. Floyd, Laurey Comeau, et al.. (2013). A miR-297/hypoxia/DGK-α axis regulating glioblastoma survival. Neuro-Oncology. 15(12). 1652–1663. 39 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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