James E. Faber

8.4k total citations · 1 hit paper
146 papers, 5.9k citations indexed

About

James E. Faber is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, James E. Faber has authored 146 papers receiving a total of 5.9k indexed citations (citations by other indexed papers that have themselves been cited), including 66 papers in Molecular Biology, 53 papers in Physiology and 31 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in James E. Faber's work include Nitric Oxide and Endothelin Effects (33 papers), Receptor Mechanisms and Signaling (27 papers) and Angiogenesis and VEGF in Cancer (21 papers). James E. Faber is often cited by papers focused on Nitric Oxide and Endothelin Effects (33 papers), Receptor Mechanisms and Signaling (27 papers) and Angiogenesis and VEGF in Cancer (21 papers). James E. Faber collaborates with scholars based in United States, Italy and Japan. James E. Faber's co-authors include Dan Chalothorn, Nengyu Yang, Xiaohua Xin, Hua Zhang, Robert Sealock, Hua Zhang, Michael J. Brody, Pranay Prabhakar, Andrea D. Eckhart and Xuming Dai and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

James E. Faber

136 papers receiving 5.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Vascular contributions to cognitive impairment and dementia including Alzheimer's disease

2014 451 citations James E. Faber et al. profile →

Peers

Countries citing papers authored by James E. Faber

Since Specialization

Citations

This map shows the geographic impact of James E. Faber's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Faber with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Faber more than expected).

Fields of papers citing papers by James E. Faber

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James E. Faber. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Faber. The network helps show where James E. Faber may publish in the future.

Co-authorship network of co-authors of James E. Faber

This figure shows the co-authorship network connecting the top 25 collaborators of James E. Faber. A scholar is included among the top collaborators of James E. Faber based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James E. Faber. James E. Faber is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Collateral blood vessels in stroke and ischemic disease: Formation, physiology, rarefaction, remodeling 2025 ·Journal of Cerebral Blood Flow & Metabolism ·James E. Faber	3
2	Sex and Genetic Background Effects on the Outcome of Experimental Intracranial Aneurysms 2020 ·Stroke ·Takeshi Yanagisawa, Hua Zhang, Tomoaki Suzuki, Yoshinobu Kamio, Tsubasa Takizawa, Andreia Morais, David Y. Chung, Tao Qin, Yuichi Murayama, James E. Faber, Aman B. Patel, Cenk Ayata	9
3	Transient versus Permanent MCA Occlusion in Mice Genetically Modified to Have Good versus Poor Collaterals 2019 ·PubMed ·Hua Zhang, James E. Faber	13
4	Cerebral Collateral Circulation: A Review in the Context of Ischemic Stroke and Mechanical Thrombectomy 2018 ·World Neurosurgery ·(unknown), Clemens M. Schirmer, Oded Goren, Hua Zhang, Amir Aghajanian, James E. Faber, Christoph J. Griessenauer	43
5	Protective and detrimental effects of neuroectodermal cell–derived tissue factor in mouse models of stroke 2016 ·JCI Insight ·Shaobin Wang, Brandi Reeves, Erica Sparkenbaugh, Janice Russell, Zbigniew Sołtys, Hua Zhang, James E. Faber, Nigel S. Key, Daniel Kirchhofer, D. Neil Granger, Nigel Mackman, Rafał Pawliński	5
6	Cardiovascular risk factors cause premature rarefaction of the collateral circulation and greater ischemic tissue injury 2015 ·Angiogenesis ·Scott M. Moore, (unknown), (unknown), Claire M. Doerschuk, James E. Faber	56
7	Potential Compensation among Group I PAK Members in Hindlimb Ischemia and Wound Healing 2014 ·PLoS ONE ·Laila Elsherif, Mehmet Özler, Mohamed A. Zayed, (unknown), Jonathan Chernoff, James E. Faber, Leslie V. Parise	6
8	Congenic Fine-Mapping Identifies a Major Causal Locus for Variation in the Native Collateral Circulation and Ischemic Injury in Brain and Lower Extremity 2013 ·Circulation Research ·Robert Sealock, Hua Zhang, Jennifer L. Lucitti, Scott M. Moore, James E. Faber	57
9	Hypertension and Other Cardiovascular Risk Factors Lead to Premature Rarefaction of the Native Collateral Circulation 2013 ·Journal of Vascular Surgery ·Scott M. Moore, (unknown), James E. Faber	3
10	Formation of the Collateral Circulation Is Regulated by Vascular Endothelial Growth Factor-A and A Disintegrin and Metalloprotease Family Members 10 and 17 2012 ·Circulation Research ·Jennifer L. Lucitti, (unknown), (unknown), Jody J. Haigh, Ralf H. Adams, James E. Faber	99
11	eNOS Deficiency Acts through Endothelin to Aggravate sFlt-1–Induced Pre-Eclampsia–Like Phenotype 2012 ·Journal of the American Society of Nephrology ·Feng Li, John R. Hagaman, Hyung‐Suk Kim, Nobuyo Maeda, J. Charles Jennette, James E. Faber, S. Ananth Karumanchi, Oliver Smithies, Nobuyuki Takahashi	92
12	Genetic Architecture Underlying Variation in Extent and Remodeling of the Collateral Circulation 2010 ·Circulation Research ·Shiliang Wang, Hua Zhang, Xuming Dai, Robert Sealock, James E. Faber	73
13	Formation and maturation of the native cerebral collateral circulation 2010 ·Journal of Molecular and Cellular Cardiology ·Dan Chalothorn, James E. Faber	82
14	Enhanced α₁-adrenergic trophic activity in pulmonary artery of hypoxic pulmonary hypertensive rats 2006 ·American Journal of Physiology-Heart and Circulatory Physiology ·James E. Faber, (unknown), Sundeep Salvi, Hua Zhang	21
15	Catecholamines augment collateral vessel growth and angiogenesis in hindlimb ischemia 2005 ·American Journal of Physiology-Heart and Circulatory Physiology ·Dan Chalothorn, Hua Zhang, Jason A. Clayton, Steven Thomas, James E. Faber	83
16	Norepinephrine-induced calcium signaling and expression of adrenoceptors in avian tendon cells 2004 ·American Journal of Physiology-Cell Physiology ·Michelle E. Wall, James E. Faber, Xi Yang, Mari Tsuzaki, Albert J. Banes	20
17	ATP modulates load‐inducible IL‐1β, COX 2, and MMP‐3 gene expression in human tendon cells 2003 ·Journal of Cellular Biochemistry ·Mari Tsuzaki, Donald K. Bynum, L C Almekinders, Xi Yang, James E. Faber, A.J. Banes	118
18	Platelet-Derived Growth Factor Inhibits α1D-Adrenergic Receptor Expression in Vascular Smooth Muscle Cells In Vitro and Ex Vivo 1999 ·Molecular Pharmacology ·Xiaohua Xin, Nengyu Yang, James E. Faber	10
19	Regulation of Vascular Smooth Muscle Growth by α1-Adrenoreceptor Subtypes in Vitro and in Situ 1995 ·Journal of Biological Chemistry ·Liqian Chen, Xiaohua Xin, Andrea D. Eckhart, Nengyu Yang, James E. Faber	117
20	In vitro Analysis of Alpha-Adrenoceptor Interactions with the Myogenic Response in Resistance Vessels 1992 ·Journal of Vascular Research ·(unknown), Kazuhiko Nishigaki, (unknown), James E. Faber	15

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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