James C. Errey
- Molecular Biology top 2%
- Cellular and Molecular Neuroscience top 1%
- Organic Chemistry top 2%
- Radiology, Nuclear Medicine and Imaging top 2%
- Physiology top 0.5%
- Co-authors
- Fiona H. MarshallBenjamin G. DavisA.S. DoreMalcolm WeirRoger CookeMiles CongreveN.J. RobertsonGonçalo J. L. Bernardes
- Topics
- Receptor Mechanisms and Signaling (20 papers)Neuropeptides and Animal Physiology (14 papers)Glycosylation and Glycoproteins Research (11 papers)
- Partner nations
- United KingdomUnited StatesSwitzerland
In The Last Decade
James C. Errey
45 papers receiving 3.7k citations
Peers
Comparison fields: 5 of 114
- Molecular Biology 3.3k
- Cellular and Molecular Neuroscience 1.2k
- Organic Chemistry 977
- Radiology, Nuclear Medicine and Imaging 621
- Physiology 509
Countries citing papers authored by James C. Errey
This map shows the geographic impact of James C. Errey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James C. Errey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James C. Errey more than expected).
Fields of papers citing papers by James C. Errey
This network shows the impact of papers produced by James C. Errey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James C. Errey. The network helps show where James C. Errey may publish in the future.
Co-authorship network of co-authors of James C. Errey
This figure shows the co-authorship network connecting the top 25 collaborators of James C. Errey. A scholar is included among the top collaborators of James C. Errey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James C. Errey. James C. Errey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 22 | |
| 2 | 69 | |
| 3 | 33 | |
| 4 | 136 | |
| 5 | 196 | |
| 6 | 13 | |
| 7 | 324 | |
| 8 | 123 | |
| 9 | 453 | |
| 10 | 138 | |
| 11 | 126 | |
| 12 | 50 | |
| 13 | 78 | |
| 14 | 40 | |
| 15 | 19 | |
| 16 | 24 | |
| 17 | 33 | |
| 18 | 20 | |
| 19 | 39 | |
| 20 | 24 |
About James C. Errey
James C. Errey is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology, having authored 45 papers that have together received 3.9k indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (20 papers), Neuropeptides and Animal Physiology (14 papers) and Glycosylation and Glycoproteins Research (11 papers). The work is most often cited by research in Physiology (509 citations), Cellular and Molecular Neuroscience (1.2k citations) and Molecular Biology (3.3k citations). James C. Errey has collaborated with scholars based in United Kingdom, United States and Switzerland. Frequent co-authors include Fiona H. Marshall, Benjamin G. Davis, A.S. Dore, Malcolm Weir, Roger Cooke, Miles Congreve, N.J. Robertson, Gonçalo J. L. Bernardes, Justin M. Chalker and K.A. Bennett. Their work appears in journals such as Nature, Journal of the American Chemical Society and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.