Isabel Lorenzo

2.9k total citations
19 papers, 2.1k citations indexed

About

Isabel Lorenzo is a scholar working on Molecular Biology, Immunology and Immunology and Allergy. According to data from OpenAlex, Isabel Lorenzo has authored 19 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 6 papers in Immunology and 5 papers in Immunology and Allergy. Recurrent topics in Isabel Lorenzo's work include Cell Adhesion Molecules Research (5 papers), Reproductive Biology and Fertility (3 papers) and Epigenetics and DNA Methylation (2 papers). Isabel Lorenzo is often cited by papers focused on Cell Adhesion Molecules Research (5 papers), Reproductive Biology and Fertility (3 papers) and Epigenetics and DNA Methylation (2 papers). Isabel Lorenzo collaborates with scholars based in United States, Ukraine and Japan. Isabel Lorenzo's co-authors include Arthur L. Beaudet, Daniel C. Bullard, N A Doyle, Claire M. Doerschuk, Klaus Ley, Avi Orr‐Urtreger, Leah Goldberg, John A. Dani, James W. Patrick and Mariella De Biasi and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Experimental Medicine and Journal of Neuroscience.

In The Last Decade

Isabel Lorenzo

18 papers receiving 2.1k citations

Peers

Isabel Lorenzo
Marcela V. Karpuj United States
Paul R. Borghesani United States
Roberto Adachi United States
Thomas Amatruda United States
Karin Franz-Bacon United States
Rainer Maier Switzerland
Isabel Lorenzo
Citations per year, relative to Isabel Lorenzo Isabel Lorenzo (= 1×) peers Hidetoshi Hasuwa

Countries citing papers authored by Isabel Lorenzo

Since Specialization
Citations

This map shows the geographic impact of Isabel Lorenzo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Isabel Lorenzo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Isabel Lorenzo more than expected).

Fields of papers citing papers by Isabel Lorenzo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Isabel Lorenzo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Isabel Lorenzo. The network helps show where Isabel Lorenzo may publish in the future.

Co-authorship network of co-authors of Isabel Lorenzo

This figure shows the co-authorship network connecting the top 25 collaborators of Isabel Lorenzo. A scholar is included among the top collaborators of Isabel Lorenzo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Isabel Lorenzo. Isabel Lorenzo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Song, I-Wen, Jason Hsu, Kempaiah Rayavara, et al.. (2024). Generation of a humanized mAce2 and a conditional hACE2 mouse models permissive to SARS-COV-2 infection. Mammalian Genome. 35(2). 113–121. 3 indexed citations
2.
Lanza, Denise G., Jianqiang Mao, Isabel Lorenzo, et al.. (2024). An oocyte‐specific Cas9‐expressing mouse for germline CRISPR/Cas9‐mediated genome editing. genesis. 62(2). e23589–e23589.
3.
Yang, Liubin, et al.. (2024). Machine learning in time-lapse imaging to differentiate embryos from young vs old mice. Biology of Reproduction. 110(6). 1115–1124. 3 indexed citations
4.
Stroup, Bridget M., Xiaohui Li, Yuqing Chen, et al.. (2023). Delayed skeletal development and IGF-1 deficiency in a mouse model of lysinuric protein intolerance. Disease Models & Mechanisms. 16(8). 1 indexed citations
5.
Arian, Sara E., Imen Chakchouk, Sangeetha Mahadevan, et al.. (2020). Reproductive Outcomes from Maternal Loss of Nlrp2 Are Not Improved by IVF or Embryo Transfer Consistent with Oocyte-Specific Defect. Reproductive Sciences. 28(7). 1850–1865. 5 indexed citations
6.
Pan, Yanzhen, Danielle Bousquet‐Moore, Portia A. McCoy, et al.. (2020). Synaptic dysfunction and abnormal behaviors in mice lacking major isoforms of Shank3. UNC Libraries. 1 indexed citations
7.
Lanza, Denise G., Isabel Lorenzo, Lan Liao, et al.. (2018). Comparative analysis of single-stranded DNA donors to generate conditional null mouse alleles. BMC Biology. 16(1). 69–69. 50 indexed citations
8.
Mahadevan, Sangeetha, et al.. (2017). Maternally expressed NLRP2 links the subcortical maternal complex (SCMC) to fertility, embryogenesis and epigenetic reprogramming. Scientific Reports. 7(1). 44667–44667. 72 indexed citations
9.
Probst, Frank J., Daniela del Gaudio, Andrew P. Salinger, et al.. (2013). A Point Mutation in the Gene for Asparagine-Linked Glycosylation 10B (Alg10b) Causes Nonsyndromic Hearing Impairment in Mice (Mus musculus). PLoS ONE. 8(11). e80408–e80408. 7 indexed citations
10.
Wang, Xiaoming, Portia A. McCoy, Ramona M. Rodriguiz, et al.. (2011). Synaptic dysfunction and abnormal behaviors in mice lacking major isoforms of Shank3. Human Molecular Genetics. 20(15). 3093–3108. 430 indexed citations
11.
Jiang, Yong‐hui, Yanzhen Pan, Li Zhu, et al.. (2010). Altered Ultrasonic Vocalization and Impaired Learning and Memory in Angelman Syndrome Mouse Model with a Large Maternal Deletion from Ube3a to Gabrb3. PLoS ONE. 5(8). e12278–e12278. 132 indexed citations
12.
González-Buitrago, J.M., Laura Ferreira, & Isabel Lorenzo. (2006). Urinary proteomics. Clinica Chimica Acta. 375(1-2). 49–56. 66 indexed citations
13.
Scharffetter‐­Kochanek, Karin, Huifang Lu, Keith E. Norman, et al.. (1998). Spontaneous Skin Ulceration and Defective T Cell Function in CD18 Null Mice. The Journal of Experimental Medicine. 188(1). 119–131. 288 indexed citations
14.
Orr‐Urtreger, Avi, Mayuko Saeki, Isabel Lorenzo, et al.. (1997). Mice Deficient in the α7 Neuronal Nicotinic Acetylcholine Receptor Lack α-Bungarotoxin Binding Sites and Hippocampal Fast Nicotinic Currents. Journal of Neuroscience. 17(23). 9165–9171. 428 indexed citations
15.
Bullard, Daniel C., Eric J. Kunkel, Hiroshi Kubo, et al.. (1996). Infectious susceptibility and severe deficiency of leukocyte rolling and recruitment in E-selectin and P-selectin double mutant mice.. The Journal of Experimental Medicine. 183(5). 2329–2336. 320 indexed citations
16.
Bullard, Daniel C., et al.. (1996). Reduced susceptibility to collagen-induced arthritis in mice deficient in intercellular adhesion molecule-1. The Journal of Immunology. 157(7). 3153–3158. 50 indexed citations
17.
Bullard, Daniel C., Li Qin, Isabel Lorenzo, et al.. (1995). P-selectin/ICAM-1 double mutant mice: acute emigration of neutrophils into the peritoneum is completely absent but is normal into pulmonary alveoli.. Journal of Clinical Investigation. 95(4). 1782–1788. 248 indexed citations
18.
Wilson, Raymond W., Tohru Yorifuji, Isabel Lorenzo, et al.. (1993). Expression of Human CD18 in Murine Granulocytes and Improved Efficiency for Infection of Deficient Human Lymphoblasts. Human Gene Therapy. 4(1). 25–34. 10 indexed citations
19.
Demarquoy, Jean, Gail E. Herman, Isabel Lorenzo, et al.. (1992). Long-Term Expression of Human Argininosuccinate Synthetase in Mice Following Bone Marrow Transplantation with Retrovirus-Transduced Hematopoietic Stem Cells. Human Gene Therapy. 3(1). 3–10. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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