Avi Orr‐Urtreger

16.1k total citations
194 papers, 8.6k citations indexed

About

Avi Orr‐Urtreger is a scholar working on Molecular Biology, Neurology and Physiology. According to data from OpenAlex, Avi Orr‐Urtreger has authored 194 papers receiving a total of 8.6k indexed citations (citations by other indexed papers that have themselves been cited), including 92 papers in Molecular Biology, 65 papers in Neurology and 36 papers in Physiology. Recurrent topics in Avi Orr‐Urtreger's work include Parkinson's Disease Mechanisms and Treatments (59 papers), Nicotinic Acetylcholine Receptors Study (40 papers) and Lysosomal Storage Disorders Research (32 papers). Avi Orr‐Urtreger is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (59 papers), Nicotinic Acetylcholine Receptors Study (40 papers) and Lysosomal Storage Disorders Research (32 papers). Avi Orr‐Urtreger collaborates with scholars based in Israel, United States and Austria. Avi Orr‐Urtreger's co-authors include Peter Lonai, Nir Giladi, Arthur L. Beaudet, Anat Bar‐Shira, David Givol, Tanya Gurevich, Anat Mirelman, James W. Patrick, Yuval Yaron and Mariella De Biasi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, JAMA and Journal of Biological Chemistry.

In The Last Decade

Avi Orr‐Urtreger

191 papers receiving 8.5k citations

Peers

Avi Orr‐Urtreger
Giorgio Casari Italy
Tatsushi Toda Japan
Kun Xia China
Masayuki Sasaki Japan
Dirk Troost Netherlands
Enza Maria Valente Italy
Safa Al‐Sarraj United Kingdom
Filippo M. Santorelli Italy
Stirling Carpenter Canada
Mark Armanini United States
Giorgio Casari Italy
Avi Orr‐Urtreger
Citations per year, relative to Avi Orr‐Urtreger Avi Orr‐Urtreger (= 1×) peers Giorgio Casari

Countries citing papers authored by Avi Orr‐Urtreger

Since Specialization
Citations

This map shows the geographic impact of Avi Orr‐Urtreger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Avi Orr‐Urtreger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Avi Orr‐Urtreger more than expected).

Fields of papers citing papers by Avi Orr‐Urtreger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Avi Orr‐Urtreger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Avi Orr‐Urtreger. The network helps show where Avi Orr‐Urtreger may publish in the future.

Co-authorship network of co-authors of Avi Orr‐Urtreger

This figure shows the co-authorship network connecting the top 25 collaborators of Avi Orr‐Urtreger. A scholar is included among the top collaborators of Avi Orr‐Urtreger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Avi Orr‐Urtreger. Avi Orr‐Urtreger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Shiner, Tamara, Gitit Kavé, Anat Mirelman, et al.. (2024). Effect of GBA1 Mutations and APOE Polymorphisms on Survival and Progression Among Ashkenazi Jews with Dementia with Lewy Bodies. Movement Disorders. 39(12). 2280–2285. 1 indexed citations
2.
Goldstein, Orly, Mali Gana‐Weisz, Sandro Banfi, et al.. (2023). Novel variants in genes related to vesicle-mediated-transport modify Parkinson's disease risk. Molecular Genetics and Metabolism. 139(2). 107608–107608. 5 indexed citations
3.
Thaler, Avner, Nir Giladi, Tanya Gurevich, et al.. (2021). Mutations in GBA and LRRK2 Are Not Associated with Increased Inflammatory Markers. Journal of Parkinson s Disease. 11(3). 1285–1296. 20 indexed citations
4.
Mirelman, Anat, Inbar Hillel, Lynn Rochester, et al.. (2020). Tossing and Turning in Bed: Nocturnal Movements in Parkinson's Disease. Movement Disorders. 35(6). 959–968. 40 indexed citations
5.
Thaler, Avner, Noa Bregman, Tanya Gurevich, et al.. (2018). Parkinson's disease phenotype is influenced by the severity of the mutations in the GBA gene. Parkinsonism & Related Disorders. 55. 45–49. 88 indexed citations
6.
Saunders‐Pullman, Rachel, Anat Mirelman, Roy N. Alcalay, et al.. (2018). Progression in the LRRK2-Associated Parkinson Disease Population. JAMA Neurology. 75(3). 312–312. 94 indexed citations
7.
Rozenkrantz, Liron, Ziv Gan‐Or, Mali Gana‐Weisz, et al.. (2016). SEPT14 Is Associated with a Reduced Risk for Parkinson’s Disease and Expressed in Human Brain. Journal of Molecular Neuroscience. 59(3). 343–350. 12 indexed citations
8.
Kobo, Hila, Anat Bar‐Shira, Dvir Dahary, et al.. (2015). Down-regulation of B cell-related genes in peripheral blood leukocytes of Parkinson's disease patients with and without GBA mutations. Molecular Genetics and Metabolism. 117(2). 179–185. 19 indexed citations
9.
Xanthos, Dimitris N., et al.. (2015). Role of α5-containing nicotinic receptors in neuropathic pain and response to nicotine. Neuropharmacology. 95. 37–49. 9 indexed citations
10.
Schreibmayer, Wolfgang, et al.. (2013). Single‐channel properties of α3β4, α3β4α5 and α3β4β2 nicotinic acetylcholine receptors in mice lacking specific nicotinic acetylcholine receptor subunits. The Journal of Physiology. 591(13). 3271–3288. 12 indexed citations
11.
Scholze, Petra, et al.. (2012). Subunit composition of α5‐containing nicotinic receptors in the rodent habenula. Journal of Neurochemistry. 121(4). 551–560. 20 indexed citations
12.
13.
Gil, Ziv, Dan M. Fliss, Leonor Leider–Trejo, et al.. (2004). Cytogenetic analysis of three variants of clival chordoma. Cancer Genetics and Cytogenetics. 154(2). 124–130. 16 indexed citations
14.
Mashiach, Roy, Avi Orr‐Urtreger, & Yuval Yaron. (2004). A Comparison between Maternal Serum Free β-Human Chorionic Gonadotrophin and Pregnancy-Associated Plasma Protein A Levels in First-Trimester Twin and Singleton Pregnancies. Fetal Diagnosis and Therapy. 19(2). 174–177. 11 indexed citations
15.
Lehavi, Ofer, Orna Aizenstein, Dani Bercovich, et al.. (2003). Screening for Familial Dysautonomia in Israel: Evidence for Higher Carrier Rate among Polish Ashkenazi Jews. Genetic Testing. 7(2). 139–142. 35 indexed citations
16.
Wang, Ningshan, Avi Orr‐Urtreger, & Amos D. Korczyn. (2002). The role of neuronal nicotinic acetylcholine receptor subunits in autonomic ganglia: lessons from knockout mice. Progress in Neurobiology. 68(5). 341–360. 67 indexed citations
17.
Rennert, Hanna, Dani Bercovich, Ayala Hubert, et al.. (2002). A Novel Founder Mutation in the RNASEL Gene, 471delAAAG, Is Associated with Prostate Cancer in Ashkenazi Jews. The American Journal of Human Genetics. 71(4). 981–984. 96 indexed citations
18.
Rozen, Paul, Ruth Shomrat, Hana Strul, et al.. (1999). Prevalence of the I1307K APC gene variant in Israeli Jews of differing ethnic origin and risk for colorectal cancer. Gastroenterology. 116(1). 54–57. 60 indexed citations
19.
Shomrat, Ruth, et al.. (1998). High Frequency of a Common Bloom Syndrome Ashkenazi Mutation Among Jews of Polish Origin. Genetic Testing. 2(4). 293–296. 41 indexed citations
20.
Orr‐Urtreger, Avi, R Ben-Levy, D Wen, et al.. (1993). Neural expression and chromosomal mapping of Neu differentiation factor to 8p12-p21.. Proceedings of the National Academy of Sciences. 90(5). 1867–1871. 100 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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