Frank J. Probst

3.7k total citations
27 papers, 1.4k citations indexed

About

Frank J. Probst is a scholar working on Molecular Biology, Genetics and Sensory Systems. According to data from OpenAlex, Frank J. Probst has authored 27 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 18 papers in Genetics and 7 papers in Sensory Systems. Recurrent topics in Frank J. Probst's work include Congenital heart defects research (8 papers), Genomic variations and chromosomal abnormalities (8 papers) and Hearing, Cochlea, Tinnitus, Genetics (7 papers). Frank J. Probst is often cited by papers focused on Congenital heart defects research (8 papers), Genomic variations and chromosomal abnormalities (8 papers) and Hearing, Cochlea, Tinnitus, Genetics (7 papers). Frank J. Probst collaborates with scholars based in United States, Poland and Canada. Frank J. Probst's co-authors include Sally A. Camper, Thomas B. Friedman, Aihui Wang, Robert A. Fridell, Jeffrey W. Touchman, Konrad Noben‐Trauth, Robert J. Morell, Yong Liang, Yehoash Raphael and Cynthia C. Morton and has published in prestigious journals such as Science, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Frank J. Probst

27 papers receiving 1.4k citations

Peers

Frank J. Probst

GENET

NEURO

Kimia Kahrizi Iran

Benjamin K. August United States

Isabelle Schrauwen United States

Hashem Shahin Palestinian Territory

Adam P. DeLuca United States

Karen P. Steel United Kingdom

Susan A. Cook United States

M. S. Deol United Kingdom

Salvatore Melchionda Italy

Karl B. Shpargel United States

Kimia Kahrizi Iran

Frank J. Probst

1.8k ×2.3

826 ×1.3

835 ×1.9

GENET

382 ×1.5

NEURO

162 ×0.9

Citations per year, relative to Frank J. Probst Frank J. Probst (= 1×) peers Kimia Kahrizi

Countries citing papers authored by Frank J. Probst

Since Specialization

Citations

This map shows the geographic impact of Frank J. Probst's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frank J. Probst with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frank J. Probst more than expected).

Fields of papers citing papers by Frank J. Probst

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frank J. Probst. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frank J. Probst. The network helps show where Frank J. Probst may publish in the future.

Co-authorship network of co-authors of Frank J. Probst

This figure shows the co-authorship network connecting the top 25 collaborators of Frank J. Probst. A scholar is included among the top collaborators of Frank J. Probst based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frank J. Probst. Frank J. Probst is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Mullegama, Sureni V., Steven D. Klein, Stephen R. Williams, et al.. (2021). Transcriptome analysis of MBD5-associated neurodevelopmental disorder (MAND) neural progenitor cells reveals dysregulation of autism-associated genes. Scientific Reports. 11(1). 11295–11295. 6 indexed citations

Probst, Frank J., Regis A. James, Lindsay C. Burrage, et al.. (2015). De novo deletions and duplications of 17q25.3 cause susceptibility to cardiovascular malformations. Orphanet Journal of Rare Diseases. 10(1). 75–75. 21 indexed citations

Dharmadhikari, Avinash V., Tomasz Gambin, Przemysław Szafrański, et al.. (2014). Molecular and clinical analyses of 16q24.1 duplications involving FOXF1 identify an evolutionarily unstable large minisatellite. BMC Medical Genetics. 15(1). 128–128. 21 indexed citations

Raznahan, Armin, Yanhe Lue, Frank J. Probst, et al.. (2014). Triangulating the sexually dimorphic brain through high-resolution neuroimaging of murine sex chromosome aneuploidies. Brain Structure and Function. 220(6). 3581–3593. 18 indexed citations

Raznahan, Armin, Frank J. Probst, Mark R. Palmert, Jay N. Giedd, & Jason P. Lerch. (2013). High resolution whole brain imaging of anatomical variation in XO, XX, and XY mice. NeuroImage. 83. 962–968. 33 indexed citations

Probst, Frank J., Daniela del Gaudio, Andrew P. Salinger, et al.. (2013). A Point Mutation in the Gene for Asparagine-Linked Glycosylation 10B (Alg10b) Causes Nonsyndromic Hearing Impairment in Mice (Mus musculus). PLoS ONE. 8(11). e80408–e80408. 7 indexed citations

Wiszniewska, Joanna, Weimin Bi, Chad A. Shaw, et al.. (2013). Combined array CGH plus SNP genome analyses in a single assay for optimized clinical testing. European Journal of Human Genetics. 22(1). 79–87. 77 indexed citations

Han, Hyo Jin, Christie M. Buchovecky, Michael J. Yetman, et al.. (2012). Strain Background Influences Neurotoxicity and Behavioral Abnormalities in Mice Expressing the Tetracycline Transactivator. Journal of Neuroscience. 32(31). 10574–10586. 66 indexed citations

McGuire, Amy L., et al.. (2012). Currents in Contemporary Bioethics. The Journal of Law Medicine & Ethics. 40(4). 1040–1046. 5 indexed citations

10.

Nagamani, Sandesh C. Sreenath, Ayelet Erez, Frank J. Probst, et al.. (2012). Small genomic rearrangements involving FMR1 support the importance of its gene dosage for normal neurocognitive function. Neurogenetics. 13(4). 333–339. 20 indexed citations

11.

Breman, Amy M., Frank J. Probst, Maria Blazo, et al.. (2011). Identification of complex chromosome 18 rearrangements by FISH and array CGH in two patients with apparent isochromosome 18q. American Journal of Medical Genetics Part A. 155(6). 1465–1468. 1 indexed citations

12.

Probst, Frank J. & Monica J. Justice. (2010). Mouse Mutagenesis with the Chemical Supermutagen ENU. Methods in enzymology on CD-ROM/Methods in enzymology. 477. 297–312. 26 indexed citations

13.

Szafrański, Przemysław, Christian P. Schaaf, Richard Person, et al.. (2010). Structures and molecular mechanisms for common 15q13.3 microduplications involving CHRNA7: benign or pathological?. Human Mutation. 31(7). 840–850. 94 indexed citations

14.

Probst, Frank J., M. Lance Cooper, Sau Wai Cheung, & Monica J. Justice. (2008). Genotype, Phenotype, and Karyotype Correlation in the XO Mouse Model of Turner Syndrome. Journal of Heredity. 99(5). 512–517. 30 indexed citations

15.

Károlyi, I, Kärin Halsey, Lisa A. Beyer, et al.. (2007). Dietary thyroid hormone replacement ameliorates hearing deficits in hypothyroid mice. Mammalian Genome. 18(8). 596–608. 31 indexed citations

16.

Probst, Frank J., Elizabeth Roeder, Zhishuo Ou, et al.. (2007). Chromosomal microarray analysis (CMA) detects a large X chromosome deletion including FMR1, FMR2, and IDS in a female patient with mental retardation. American Journal of Medical Genetics Part A. 143A(12). 1358–1365. 44 indexed citations

17.

Kanzaki, Sho, Lisa A. Beyer, I Károlyi, et al.. (2006). Transgene correction maintains normal cochlear structure and function in 6-month-old Myo15a mutant mice. Hearing Research. 214(1-2). 37–44. 13 indexed citations

18.

Probst, Frank J. & Sally A. Camper. (1999). The role of mouse mutants in the identification of human hereditary hearing loss genes. Hearing Research. 130(1-2). 1–6. 26 indexed citations

19.

Liang, Yong, Aihui Wang, Inna A. Belyantseva, et al.. (1999). Characterization of the Human and Mouse Unconventional Myosin XV Genes Responsible for Hereditary Deafness DFNB3 and Shaker 2. Genomics. 61(3). 243–258. 128 indexed citations

20.

Probst, Frank J., Qi Zhao, Aihui Wang, et al.. (1999). A Physical Map of the Mouseshaker-2Region Contains Many of the Genes Commonly Deleted in Smith–Magenis Syndrome (del17p11.2p11.2). Genomics. 55(3). 348–352. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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