Kouichi Inukai

7.2k total citations
99 papers, 5.6k citations indexed

About

Kouichi Inukai is a scholar working on Molecular Biology, Surgery and Physiology. According to data from OpenAlex, Kouichi Inukai has authored 99 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 71 papers in Molecular Biology, 29 papers in Surgery and 24 papers in Physiology. Recurrent topics in Kouichi Inukai's work include Metabolism, Diabetes, and Cancer (44 papers), Pancreatic function and diabetes (27 papers) and Adipose Tissue and Metabolism (17 papers). Kouichi Inukai is often cited by papers focused on Metabolism, Diabetes, and Cancer (44 papers), Pancreatic function and diabetes (27 papers) and Adipose Tissue and Metabolism (17 papers). Kouichi Inukai collaborates with scholars based in Japan, United States and Kenya. Kouichi Inukai's co-authors include Tomoichiro Asano, Hideki Katagiri, Motonobu Anai, Yoshitomo Oka, Shigehiro Katayama, Takehide Ogihara, Hisamitsu Ishihara, Masatoshi Kikuchi, Yasushi Fukushima and Takuya Awata and has published in prestigious journals such as Science, Journal of Biological Chemistry and SHILAP Revista de lepidopterología.

In The Last Decade

Kouichi Inukai

97 papers receiving 5.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kouichi Inukai Japan 41 3.1k 1.2k 1.2k 995 974 99 5.6k
Hiroyuki Tamemoto Japan 33 3.2k 1.0× 1.1k 0.9× 1.2k 1.0× 680 0.7× 931 1.0× 69 5.3k
Emmanuel Van Obberghen France 54 6.1k 2.0× 2.0k 1.6× 2.0k 1.7× 964 1.0× 1.4k 1.4× 150 9.7k
Pascale de Lonlay France 49 5.2k 1.7× 1.2k 1.0× 914 0.8× 436 0.4× 1.8k 1.8× 224 8.7k
Edward P. Feener United States 43 2.8k 0.9× 697 0.6× 1.3k 1.2× 659 0.7× 1.1k 1.1× 94 6.7k
J K Boyles United States 31 2.7k 0.9× 1.6k 1.3× 1.9k 1.7× 1.1k 1.1× 605 0.6× 42 6.6k
Luc Bertrand Belgium 46 5.3k 1.7× 2.1k 1.7× 1.5k 1.3× 986 1.0× 1.1k 1.2× 145 7.9k
Kenneth Siddle United Kingdom 50 5.9k 1.9× 2.0k 1.6× 1.4k 1.2× 672 0.7× 2.5k 2.6× 165 8.6k
Sotirios K. Karathanasis United States 42 3.1k 1.0× 2.1k 1.7× 831 0.7× 639 0.6× 1.5k 1.5× 85 6.2k
Hadi Al‐Hasani Germany 38 2.2k 0.7× 959 0.8× 1.2k 1.0× 551 0.6× 551 0.6× 138 4.2k
Richard M. Mortensen United States 43 4.9k 1.6× 1.1k 0.9× 1.6k 1.4× 968 1.0× 843 0.9× 104 7.7k

Countries citing papers authored by Kouichi Inukai

Since Specialization
Citations

This map shows the geographic impact of Kouichi Inukai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kouichi Inukai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kouichi Inukai more than expected).

Fields of papers citing papers by Kouichi Inukai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kouichi Inukai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kouichi Inukai. The network helps show where Kouichi Inukai may publish in the future.

Co-authorship network of co-authors of Kouichi Inukai

This figure shows the co-authorship network connecting the top 25 collaborators of Kouichi Inukai. A scholar is included among the top collaborators of Kouichi Inukai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kouichi Inukai. Kouichi Inukai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Ito, Daisuke, Kazuyuki Inoue, M. Yanagisawa, et al.. (2016). Effects of Ipragliflozin on Diabetic Nephropathy and Blood Pressure in Patients With Type 2 Diabetes: An Open-Label Study. Journal of Clinical Medicine Research. 9(2). 154–162. 10 indexed citations
3.
Ito, Daisuke, Kazuyuki Inoue, M. Yanagisawa, et al.. (2015). The Efficacy of Vildagliptin Concomitant With Insulin Therapy in Type 2 Diabetic Subjects. Journal of Clinical Medicine Research. 7(5). 303–307. 5 indexed citations
4.
Hosaka, Toshio, et al.. (2015). Influence of Treatment with Extracts of <i>Hypsyzigus marmoreus</i> Mushroom on Body Composition during Obesity Development in KK-A<sup>y</sup> Mice. Journal of Nutritional Science and Vitaminology. 61(1). 96–100. 4 indexed citations
5.
Ito, Daisuke, et al.. (2015). Efficacy and Clinical Characteristics of Liraglutide in Japanese Patients With Type 2 Diabetes. Journal of Clinical Medicine Research. 7(9). 694–699. 8 indexed citations
6.
Inukai, Kouichi, Akihiko Kudo, Fumiyuki Nakagawa, et al.. (2014). Long-Term Low Carbohydrate Diet Leads to Deleterious Metabolic Manifestations in Diabetic Mice. PLoS ONE. 9(8). e104948–e104948. 8 indexed citations
7.
Motoshima, Hiroyuki, Motoyuki Igata, Takeshi Matsumura, et al.. (2008). Rottlerin activates AMPK possibly through LKB1 in vascular cells and tissues. Biochemical and Biophysical Research Communications. 376(2). 434–438. 8 indexed citations
8.
Imai, Kenta, et al.. (2006). LKB1, an upstream AMPK kinase, regulates glucose and lipid metabolism in cultured liver and muscle cells. Biochemical and Biophysical Research Communications. 351(3). 595–601. 46 indexed citations
9.
Awata, Takuya, Susumu Kurihara, Nobuki Takata, et al.. (2005). Functional VEGF C-634G polymorphism is associated with development of diabetic macular edema and correlated with macular retinal thickness in type 2 diabetes. Biochemical and Biophysical Research Communications. 333(3). 679–685. 114 indexed citations
10.
Takata, Nobuki, Takuya Awata, Kouichi Inukai, et al.. (2004). Pro12Ala substitution in peroxisome proliferator-activated receptorγ2 is associated with low adiponectin concentrations in young Japanese men. Metabolism. 53(12). 1548–1551. 25 indexed citations
11.
Song, Haiyan, Nobuhiro Shojima, Hideyuki Sakoda, et al.. (2002). Resistin is regulated by C/EBPs, PPARs, and signal-transducing molecules. Biochemical and Biophysical Research Communications. 299(2). 291–298. 60 indexed citations
12.
Inukai, Kouichi, Makoto Funaki, Motonobu Anai, et al.. (2001). Five isoforms of the phosphatidylinositol 3‐kinase regulatory subunit exhibit different associations with receptor tyrosine kinases and their tyrosine phosphorylations. FEBS Letters. 490(1-2). 32–38. 41 indexed citations
13.
Asano, Tomoichiro, Akira Kanda, Hideki Katagiri, et al.. (2000). p110β Is Up-regulated during Differentiation of 3T3-L1 Cells and Contributes to the Highly Insulin-responsive Glucose Transport Activity. Journal of Biological Chemistry. 275(23). 17671–17676. 49 indexed citations
14.
Inukai, Kouichi, Kuniaki Takata, Tomoichiro Asano, et al.. (1997). Targeting of GLUT1-GLUT5 Chimeric Proteins in the Polarized Cell Line Caco-2. Molecular Endocrinology. 11(4). 442–449. 14 indexed citations
15.
Inukai, Kouichi, Makoto Funaki, Takehide Ogihara, et al.. (1997). p85α Gene Generates Three Isoforms of Regulatory Subunit for Phosphatidylinositol 3-Kinase (PI 3-Kinase), p50α, p55α, and p85α, with Different PI 3-Kinase Activity Elevating Responses to Insulin. Journal of Biological Chemistry. 272(12). 7873–7882. 132 indexed citations
17.
Katagiri, Hideki, Tomoichiro Asano, Hisamitsu Ishihara, et al.. (1996). Overexpression of Catalytic Subunit p110α of Phosphatidylinositol 3-Kinase Increases Glucose Transport Activity with Translocation of Glucose Transporters in 3T3-L1 Adipocytes. Journal of Biological Chemistry. 271(29). 16987–16990. 170 indexed citations
18.
Tashiro, Fumi, Koichi Ikuta, Hideki Katagiri, et al.. (1995). Inhibition of pancreatic β‐cell glucokinase by antisense RNA expression in transgenic mice: mouse strain‐dependent alteration of glucose tolerance. FEBS Letters. 371(3). 329–332. 18 indexed citations
19.
Katagiri, Hideki, Tomoichiro Asano, Hisamitsu Ishihara, et al.. (1992). Replacement of intracellular C-terminal domain of GLUT1 glucose transporter with that of GLUT2 increases Vmax and Km of transport activity.. Journal of Biological Chemistry. 267(31). 22550–22555. 56 indexed citations
20.
Katagiri, Hideki, Katsunori Tsukuda, Hisamitsu Ishihara, et al.. (1992). Deletion of C-terminal 12 amino acids of GLUT1 protein does not abolish the transport activity. Biochemical and Biophysical Research Communications. 184(2). 865–870. 11 indexed citations

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