Heidi Anthoni

658 total citations
7 papers, 441 citations indexed

About

Heidi Anthoni is a scholar working on Genetics, Developmental and Educational Psychology and Statistics and Probability. According to data from OpenAlex, Heidi Anthoni has authored 7 papers receiving a total of 441 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Genetics, 6 papers in Developmental and Educational Psychology and 3 papers in Statistics and Probability. Recurrent topics in Heidi Anthoni's work include Reading and Literacy Development (6 papers), Genetics and Neurodevelopmental Disorders (6 papers) and Cognitive and developmental aspects of mathematical skills (3 papers). Heidi Anthoni is often cited by papers focused on Reading and Literacy Development (6 papers), Genetics and Neurodevelopmental Disorders (6 papers) and Cognitive and developmental aspects of mathematical skills (3 papers). Heidi Anthoni collaborates with scholars based in Sweden, Finland and Germany. Heidi Anthoni's co-authors include Juha Kere, Myriam Peyrard‐Janvid, Markus M. Nöthen, Johannes Schumacher, Marco Zucchelli, Andreas Warnke, Gerd Schulte‐Körne, Helmut Remschmidt, Andreas Ziegler and Bertram Müller‐Myhsok and has published in prestigious journals such as Journal of Neuroscience, The American Journal of Human Genetics and Human Molecular Genetics.

In The Last Decade

Heidi Anthoni

7 papers receiving 432 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Heidi Anthoni Sweden	7	320	247	118	104	89	7	441
Tom S. Scerri United Kingdom	4	350 1.1×	280 1.1×	107 0.9×	92 0.9×	147 1.7×	4	492
Haiying Meng United States	9	485 1.5×	331 1.3×	144 1.2×	186 1.8×	163 1.8×	18	672
Ellen Plume Germany	9	331 1.0×	208 0.8×	71 0.6×	142 1.4×	122 1.4×	11	420
Darina Roeske Germany	9	133 0.4×	138 0.6×	112 0.9×	50 0.5×	72 0.8×	11	478
Jaana Nopola‐Hemmi Finland	13	710 2.2×	564 2.3×	257 2.2×	223 2.1×	204 2.3×	14	1.0k
Dongnhu T. Truong United States	10	114 0.4×	77 0.3×	62 0.5×	38 0.4×	102 1.1×	18	256
Natalie R. Powers United States	11	173 0.5×	154 0.6×	263 2.2×	50 0.5×	73 0.8×	18	492
William M. Brandler United Kingdom	8	138 0.4×	232 0.9×	102 0.9×	22 0.2×	303 3.4×	8	494
Katherine M. Mackenzie United States	5	31 0.1×	85 0.3×	47 0.4×	41 0.4×	128 1.4×	6	233

Countries citing papers authored by Heidi Anthoni

Since Specialization

Citations

This map shows the geographic impact of Heidi Anthoni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heidi Anthoni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heidi Anthoni more than expected).

Fields of papers citing papers by Heidi Anthoni

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Heidi Anthoni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heidi Anthoni. The network helps show where Heidi Anthoni may publish in the future.

Co-authorship network of co-authors of Heidi Anthoni

This figure shows the co-authorship network connecting the top 25 collaborators of Heidi Anthoni. A scholar is included among the top collaborators of Heidi Anthoni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Heidi Anthoni. Heidi Anthoni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

Matsson, Hans, Kristiina Tammimies, Marco Zucchelli, et al.. (2011). SNP Variations in the 7q33 Region Containing DGKI are Associated with Dyslexia in the Finnish and German Populations. Behavior Genetics. 41(1). 134–140. 17 indexed citations

Rantamäki, Tomi, Aino Vesikansa, Heidi Anthoni, et al.. (2009). Brain-Derived Neurotrophic Factor Controls Activity-Dependent Maturation of CA1 Synapses by Downregulating Tonic Activationof Presynaptic Kainate Receptors. Journal of Neuroscience. 29(36). 11294–11303. 32 indexed citations

Anthoni, Heidi, Isabel Tapia‐Páez, Myriam Peyrard‐Janvid, et al.. (2009). Further evidence for DYX1C1 as a susceptibility factor for dyslexia. Psychiatric Genetics. 19(2). 59–63. 53 indexed citations

Ludwig, Kerstin U., Johannes Schumacher, Gerd Schulte‐Körne, et al.. (2008). Investigation of the DCDC2 intron 2 deletion/compound short tandem repeat polymorphism in a large German dyslexia sample. Psychiatric Genetics. 18(6). 310–312. 46 indexed citations

Anthoni, Heidi, Marco Zucchelli, Hans Matsson, et al.. (2007). A locus on 2p12 containing the co-regulated MRPL19 and C2ORF3 genes is associated to dyslexia. Human Molecular Genetics. 16(6). 667–677. 91 indexed citations

Schumacher, Johannes, Heidi Anthoni, Inke R. König, et al.. (2005). Strong Genetic Evidence of DCDC2 as a Susceptibility Gene for Dyslexia. The American Journal of Human Genetics. 78(1). 52–62. 180 indexed citations

Peyrard‐Janvid, Myriam, Heidi Anthoni, Marco Zucchelli, et al.. (2004). Fine mapping of the 2p11 dyslexia locus and exclusion of TACR1 as a candidate gene. Human Genetics. 114(5). 510–516. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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