Isabel Tapia‐Páez

1.4k total citations
28 papers, 816 citations indexed

About

Isabel Tapia‐Páez is a scholar working on Molecular Biology, Genetics and Dermatology. According to data from OpenAlex, Isabel Tapia‐Páez has authored 28 papers receiving a total of 816 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 18 papers in Genetics and 4 papers in Dermatology. Recurrent topics in Isabel Tapia‐Páez's work include Genetics and Neurodevelopmental Disorders (10 papers), Congenital heart defects research (5 papers) and Genetic and Kidney Cyst Diseases (5 papers). Isabel Tapia‐Páez is often cited by papers focused on Genetics and Neurodevelopmental Disorders (10 papers), Congenital heart defects research (5 papers) and Genetic and Kidney Cyst Diseases (5 papers). Isabel Tapia‐Páez collaborates with scholars based in Sweden, Finland and United States. Isabel Tapia‐Páez's co-authors include Juha Kere, Kristiina Tammimies, Satu Massinen, Jan P. Dumanski, Hans Matsson, Elisabeth Blennow, Eero Ċastrén, Ananda L. Roy, Britt‐Marie Anderlid and Roel Quintens and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Scientific Reports.

In The Last Decade

Isabel Tapia‐Páez

28 papers receiving 800 citations

Peers

Isabel Tapia‐Páez
R. Curtis Rogers United States
Theresa A. Grebe United States
Yoonha Lee South Korea
J.-M. Lalouel United States
Markus Zweier Switzerland
Isabel Tapia‐Páez
Citations per year, relative to Isabel Tapia‐Páez Isabel Tapia‐Páez (= 1×) peers L. Leigh Field

Countries citing papers authored by Isabel Tapia‐Páez

Since Specialization
Citations

This map shows the geographic impact of Isabel Tapia‐Páez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Isabel Tapia‐Páez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Isabel Tapia‐Páez more than expected).

Fields of papers citing papers by Isabel Tapia‐Páez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Isabel Tapia‐Páez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Isabel Tapia‐Páez. The network helps show where Isabel Tapia‐Páez may publish in the future.

Co-authorship network of co-authors of Isabel Tapia‐Páez

This figure shows the co-authorship network connecting the top 25 collaborators of Isabel Tapia‐Páez. A scholar is included among the top collaborators of Isabel Tapia‐Páez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Isabel Tapia‐Páez. Isabel Tapia‐Páez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Eisfeldt, Jesper, Kassahun Desalegn Bilcha, Carl‐Fredrik Wahlgren, et al.. (2024). Uncommon Variants in FLG2 and NOD2 Are Associated with Atopic Dermatitis in the Ethiopian Population. SHILAP Revista de lepidopterología. 4(4). 100284–100284. 2 indexed citations
2.
Nikamo, Pernilla, Leena Laasonen, Björn Guðbjörnsson, et al.. (2024). Rare coding variants in NOX4 link high ROS levels to psoriatic arthritis mutilans. EMBO Molecular Medicine. 16(3). 596–615. 5 indexed citations
3.
Padhi, Avinash, Rokeya Sultana Rekha, Magda Lourda, et al.. (2023). Baricitinib Blocks Cytokine-Mediated Downregulation of PAD1 in Human Keratinocytes: A Possible Molecular Link to the Effects of JAK Inhibitors in Atopic Dermatitis. Journal of Investigative Dermatology. 143(7). 1306–1309.e4. 5 indexed citations
4.
Chandrasekar, Gayathri, et al.. (2023). Genetic and protein interaction studies between the ciliary dyslexia candidate genes DYX1C1 and DCDC2. BMC Molecular and Cell Biology. 24(1). 20–20. 2 indexed citations
5.
Yoshihara, Masahito, Shintaro Katayama, Kaarel Krjutškov, et al.. (2020). Dyslexia Candidate Gene and Ciliary Gene Expression Dynamics During Human Neuronal Differentiation. Molecular Neurobiology. 57(7). 2944–2958. 9 indexed citations
6.
Sahlén, Pelin, Jesper Eisfeldt, Ankit Srivastava, et al.. (2020). Chromatin interactions in differentiating keratinocytes reveal novel atopic dermatitis– and psoriasis-associated genes. Journal of Allergy and Clinical Immunology. 147(5). 1742–1752. 21 indexed citations
7.
Einarsdóttir, Elísabet, Hans Matsson, Harriet E. Nilsson, et al.. (2020). Rare variants in dynein heavy chain genes in two individuals with situs inversus and developmental dyslexia: a case report. BMC Medical Genetics. 21(1). 87–87. 4 indexed citations
8.
Tapia‐Páez, Isabel, et al.. (2018). Evaluation of Single Nucleotide Variants in Ethiopian Patients with Atopic Dermatitis. Acta Dermato Venereologica. 99(1). 0–0. 3 indexed citations
9.
Yu, Nancy, Shintaro Katayama, Elísabet Einarsdóttir, et al.. (2017). Acute doses of caffeine shift nervous system cell expression profiles toward promotion of neuronal projection growth. Scientific Reports. 7(1). 11458–11458. 14 indexed citations
10.
Massinen, Satu, et al.. (2016). Genomic sequencing of a dyslexia susceptibility haplotype encompassing ROBO1. Journal of Neurodevelopmental Disorders. 8(1). 4–4. 8 indexed citations
11.
Chandrasekar, Gayathri, Liselotte Vesterlund, Kjell Hultenby, Isabel Tapia‐Páez, & Juha Kere. (2013). The Zebrafish Orthologue of the Dyslexia Candidate Gene DYX1C1 Is Essential for Cilia Growth and Function. PLoS ONE. 8(5). e63123–e63123. 44 indexed citations
12.
Tammimies, Kristiina, Morana Vitezic, Hans Matsson, et al.. (2012). Molecular Networks of DYX1C1 Gene Show Connection to Neuronal Migration Genes and Cytoskeletal Proteins. Biological Psychiatry. 73(6). 583–590. 31 indexed citations
13.
Tammimies, Kristiina, Isabel Tapia‐Páez, Joëlle Rüegg, et al.. (2012). The rs3743205 SNP Is Important for the Regulation of the Dyslexia Candidate GeneDYX1C1by Estrogen Receptor β and DNA Methylation. Molecular Endocrinology. 26(4). 619–629. 23 indexed citations
14.
Tammimies, Kristiina, Morana Vitezic, Hans Matsson, et al.. (2012). ISDN2012_0190: The molecular network of the dyslexia candidate gene DYX1C1 shows connection to neuronal migration genes and cytoskeletal proteins. International Journal of Developmental Neuroscience. 30(8). 675–676. 2 indexed citations
15.
Massinen, Satu, Hans Matsson, Kristiina Tammimies, et al.. (2011). Increased Expression of the Dyslexia Candidate Gene DCDC2 Affects Length and Signaling of Primary Cilia in Neurons. PLoS ONE. 6(6). e20580–e20580. 94 indexed citations
16.
Massinen, Satu, Kristiina Tammimies, Isabel Tapia‐Páez, et al.. (2009). Functional interaction of DYX1C1 with estrogen receptors suggests involvement of hormonal pathways in dyslexia. Human Molecular Genetics. 18(15). 2802–2812. 48 indexed citations
17.
Otonkoski, Timo, Nina Kaminen‐Ahola, Isabel Tapia‐Páez, et al.. (2007). Physical Exercise–Induced Hypoglycemia Caused by Failed Silencing of Monocarboxylate Transporter 1 in Pancreatic β Cells. The American Journal of Human Genetics. 81(3). 467–474. 158 indexed citations
18.
Anderlid, Britt‐Marie, Jacqueline Schoumans, Göran Annerén, et al.. (2002). FISH-mapping of a 100-kb terminal 22q13 deletion. Human Genetics. 110(5). 439–443. 55 indexed citations
19.
Grigelioniené, Giedré, Jacqueline Schoumans, Sten A. Ivarsson, et al.. (2001). Analysis of short stature homeobox-containing gene ( SHOX ) and auxological phenotype in dyschondrosteosis and isolated Madelung deformity. Human Genetics. 109(5). 551–558. 38 indexed citations
20.
Tapia‐Páez, Isabel, Kevin P. O'Brien, Maria Kost‐Alimova, et al.. (2000). Fine mapping of the constitutional translocation t(11;22)(q23;q11). Human Genetics. 106(5). 506–516. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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