Heekyong Bae

932 citations
10 papers · 709 indexed · 1 hit paper · h-index 9
Topics
Liver Disease Diagnosis and Treatment (4 papers)Liver Diseases and Immunity (3 papers)Autophagy in Disease and Therapy (2 papers)

In The Last Decade

Heekyong Bae

9 papers receiving 705 citations

Hit Papers

Early TCR Signaling Induces Rapid Aerobic Glycolysis Enab...20182026202020232018100200300

Peers

Heekyong Bae
Comparison fields: 5 of 77
  • Immunology 290
  • Molecular Biology 260
  • Epidemiology 191
  • Oncology 147
  • Hepatology 115
Replace Na Zhao with:
Na Zhao China
Dianyuan Zhao China
Nobumi Suzuki Japan
Murugabaskar Balan United States
Daniel Seidel Germany
Hairong Chen China
I‐Wei Chang Taiwan
Yasumasa Minoda Japan
Ivan Mattioli Switzerland
Keita Saeki Japan
Heekyong Bae relative to Na Zhao China Na Zhao's profile →
Citations per field
00.5×2.6×
Na Zhao · 1×
Citations per year

Countries citing papers authored by Heekyong Bae

Since Specialization
Citations

This map shows the geographic impact of Heekyong Bae's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heekyong Bae with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heekyong Bae more than expected).

Fields of papers citing papers by Heekyong Bae

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Heekyong Bae. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heekyong Bae. The network helps show where Heekyong Bae may publish in the future.

Co-authorship network of co-authors of Heekyong Bae

This figure shows the co-authorship network connecting the top 25 collaborators of Heekyong Bae. A scholar is included among the top collaborators of Heekyong Bae based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Heekyong Bae. Heekyong Bae is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
#WorkIndexed citations
1 0
2 18
3 24
4 16
5
Early TCR Signaling Induces Rapid Aerobic Glycolysis Enabling Distinct Acute T Cell Effector Functionsbreakdown →
343
6 46
7 88
8 46
9 77
10 51

About Heekyong Bae

Heekyong Bae is a scholar working on Hepatology, Epidemiology and Rheumatology, having authored 10 papers that have together received 709 indexed citations. Recurring topics across this work include Liver Disease Diagnosis and Treatment (4 papers), Liver Diseases and Immunity (3 papers) and Autophagy in Disease and Therapy (2 papers). The work is most often cited by research in Hepatology (115 citations), Immunology (290 citations) and Cancer Research (101 citations). Heekyong Bae has collaborated with scholars based in United States, South Korea and Japan. Frequent co-authors include Howard A. Young, Jun‐Lin Guan, Greg M. Delgoffe, Xue Zeng, Jianxin Xie, Sonia R. Salvatore, Nicole E. Scharping, Stacy G. Wendell, Ashley V. Menk and Rebecca S. Moreci. Their work appears in journals such as Hepatology, Clinical Cancer Research and International Journal of Molecular Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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