G. Forster

2.7k total citations
24 papers, 1.6k citations indexed

About

G. Forster is a scholar working on Pathology and Forensic Medicine, Oncology and Otorhinolaryngology. According to data from OpenAlex, G. Forster has authored 24 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Pathology and Forensic Medicine, 6 papers in Oncology and 5 papers in Otorhinolaryngology. Recurrent topics in G. Forster's work include Alzheimer's disease research and treatments (5 papers), Head and Neck Cancer Studies (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). G. Forster is often cited by papers focused on Alzheimer's disease research and treatments (5 papers), Head and Neck Cancer Studies (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). G. Forster collaborates with scholars based in United Kingdom, United States and Norway. G. Forster's co-authors include Paul G. Ince, Stephen B. Wharton, Pamela J. Shaw, Fiona E. Matthews, Carol Brayne, George M. Savva, Julie E. Simpson, G. Lace, Michael V. Knopp and John Evans and has published in prestigious journals such as Brain, Neurology and British Journal of Cancer.

In The Last Decade

G. Forster

24 papers receiving 1.6k citations

Peers

G. Forster

PHYSI

NEURO

CMN

Jan Sällström Sweden

André Huss Germany

Ellen Bennett United States

J. Bohl Germany

Brian E. Mace United States

Mario Merlini United States

Ichiro Ikegaki Japan

Saeid Taheri United States

Yumiko Motoi Japan

Jaime Ramos‐Cejudo United States

Jan Sällström Sweden

G. Forster

616 ×1.1

PHYSI

590 ×1.0

NEURO

511 ×1.2

212 ×0.6

NEURO

145 ×0.6

CMN

Citations per year, relative to G. Forster G. Forster (= 1×) peers Jan Sällström

Countries citing papers authored by G. Forster

Since Specialization

Citations

This map shows the geographic impact of G. Forster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Forster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Forster more than expected).

Fields of papers citing papers by G. Forster

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Forster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Forster. The network helps show where G. Forster may publish in the future.

Co-authorship network of co-authors of G. Forster

This figure shows the co-authorship network connecting the top 25 collaborators of G. Forster. A scholar is included among the top collaborators of G. Forster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Forster. G. Forster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Forster, G., et al.. (2023). Built Environment Accessibility and Disability as Predictors of Well-Being among Older Adults: A Norwegian Cross-Sectional Study. International Journal of Environmental Research and Public Health. 20(10). 5898–5898. 3 indexed citations

Ince, Paul G., Thaı́s Minett, G. Forster, Carol Brayne, & Stephen B. Wharton. (2016). Microinfarcts in an older population‐representative brain donor cohort (MRC CFAS): Prevalence, relation to dementia and mobility, and implications for the evaluation of cerebral Small Vessel Disease. Neuropathology and Applied Neurobiology. 43(5). 409–418. 39 indexed citations

Garwood, Claire J., Stephen B. Wharton, Paul G. Ince, et al.. (2013). Calcium dysregulation in relation to Alzheimer‐type pathology in the ageing brain. Neuropathology and Applied Neurobiology. 39(7). 788–799. 37 indexed citations

Wharton, Stephen B., James P. O’Callaghan, George M. Savva, et al.. (2009). Population Variation in Glial Fibrillary Acidic Protein Levels in Brain Ageing: Relationship to Alzheimer-Type Pathology and Dementia. Dementia and Geriatric Cognitive Disorders. 27(5). 465–473. 45 indexed citations

Simpson, Julie E., Paul G. Ince, Catherine Gelsthorpe, et al.. (2009). Population variation in oxidative stress and astrocyte DNA damage in relation to Alzheimer-type pathology in the ageing brain. Neuropathology and Applied Neurobiology. 36(1). 25–40. 84 indexed citations

Lace, G., George M. Savva, G. Forster, et al.. (2009). Hippocampal tau pathology is related to neuroanatomical connections: an ageing population-based study. Brain. 132(5). 1324–1334. 164 indexed citations

Simpson, Julie E., Paul G. Ince, G. Lace, et al.. (2008). Astrocyte phenotype in relation to Alzheimer-type pathology in the ageing brain. Neurobiology of Aging. 31(4). 578–590. 304 indexed citations

Simpson, Julie E., Paul G. Ince, Catherine Gelsthorpe, et al.. (2007). Microglial activation in white matter lesions and nonlesional white matter of ageing brains. Neuropathology and Applied Neurobiology. 33(6). 670–683. 113 indexed citations

Fernando, M. Shanika, Luke Clark, Paul G. Ince, et al.. (2007). White matter lesions in an unselected cohort of the elderly: astrocytic, microglial and oligodendrocyte precursor cell responses. Neuropathology and Applied Neurobiology. 33(4). 410–419. 171 indexed citations

10.

Fernando, Malee, John T. O’Brien, R. H. Perry, et al.. (2004). Comparison of the pathology of cerebral white matter with post‐mortem magnetic resonance imaging (MRI) in the elderly brain. Neuropathology and Applied Neurobiology. 30(4). 385–395. 111 indexed citations

11.

Ince, Paul G., John Evans, Michael V. Knopp, et al.. (2003). Corticospinal tract degeneration in the progressive muscular atrophy variant of ALS. Neurology. 60(8). 1252–1258. 220 indexed citations

12.

Clemons, Mark, M C Bibby, G. Forster, et al.. (2002). Heterogeneity of O6-alkylguanine DNA-alkyltransferase expression in human breast tumours. British Journal of Cancer. 86(11). 1797–1802. 12 indexed citations

13.

West, Catharine, S.E. Davidson, RD Hunter, et al.. (1998). Levels of the DNA repair enzyme human apurinic/apyrimidinic endonuclease (APE1, APEX, Ref-1) are associated with the intrinsic radiosensitivity of cervical cancers. British Journal of Cancer. 78(9). 1128–1133. 80 indexed citations

14.

Cooke, T G, et al.. (1994). Flow cytometric analysis of DNA content in squamous carcinoma of the tongue: the relationship to host and tumour factors and survival. Clinical Otolaryngology. 19(2). 131–134. 12 indexed citations

15.

Cooke, Timothy G., et al.. (1994). Prognostic value of ploidy of primary tumour and nodal secondaries in colorectal cancers. Surgical Oncology. 3(6). 345–349. 3 indexed citations

16.

Jones, Andrew, et al.. (1994). A comparison of cellular proliferation markers in squamous cell carcinoma of the head and neck. The Journal of Laryngology & Otology. 108(10). 859–864. 34 indexed citations

17.

Cooke, TG, et al.. (1994). Prospective evaluation of cell kinetics in head and neck squamous carcinoma: the relationship to tumour factors and survival. British Journal of Cancer. 69(4). 717–720. 27 indexed citations

18.

Forster, G., et al.. (1992). Tumour growth rates in squamous carcinoma of the head and neck measured by in vivo bromodeoxyuridine incorporation and flow cytometry. British Journal of Cancer. 65(5). 698–702. 30 indexed citations

19.

Cooke, TG, et al.. (1991). Cellular DNA content and prognosis in surgically treated squamous carcinoma of the larynx. British Journal of Cancer. 63(6). 1018–1020. 7 indexed citations

20.

Cooke, T G, Friedrich Bootz, G. Forster, et al.. (1990). Ploidy as a prognostic indicator in end stage squamous cell carcinoma of the head and neck region treated with cisplatinum. British Journal of Cancer. 61(5). 759–762. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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