Felix L. Yeh
- Neurology top 1%
- Neurology top 2%
- Physiology top 5%
- Cellular and Molecular Neuroscience top 5%
- Immunology top 5%
- Co-authors
- Morgan ShengIrene TomYuanyuan WangLino C. GonzalezEdwin R. ChapmanMatthew H. BaileyEric A. JohnsonWilliam H. Tepp
- Topics
- Neuroinflammation and Neurodegeneration Mechanisms (8 papers)Amyotrophic Lateral Sclerosis Research (5 papers)Inflammation biomarkers and pathways (5 papers)
- Cited by
- NeurologyBiological Psychiatry
- Partner nations
- United StatesFranceGermany
In The Last Decade
Felix L. Yeh
22 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 94
- Neurology 760
- Neurology 667
- Physiology 626
- Cellular and Molecular Neuroscience 507
- Immunology 461
Countries citing papers authored by Felix L. Yeh
This map shows the geographic impact of Felix L. Yeh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Felix L. Yeh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Felix L. Yeh more than expected).
Fields of papers citing papers by Felix L. Yeh
This network shows the impact of papers produced by Felix L. Yeh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Felix L. Yeh. The network helps show where Felix L. Yeh may publish in the future.
Co-authorship network of co-authors of Felix L. Yeh
This figure shows the co-authorship network connecting the top 25 collaborators of Felix L. Yeh. A scholar is included among the top collaborators of Felix L. Yeh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Felix L. Yeh. Felix L. Yeh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 7 | |
| 3 | 38 | |
| 4 | 6 | |
| 5 | 3 | |
| 6 | 23 | |
| 7 | 2 | |
| 8 | 0 | |
| 9 | 4 | |
| 10 | 3 | |
| 11 | 1 | |
| 12 | 10 | |
| 13 | 8 | |
| 14 | 322 | |
| 15 | TREM2 Binds to Apolipoproteins, Including APOE and CLU/APOJ, and Thereby Facilitates Uptake of Amyloid-Beta by Microgliabreakdown → | 650 |
| 16 | 53 | |
| 17 | 7 | |
| 18 | 102 | |
| 19 | 64 | |
| 20 | SV2 Is the Protein Receptor for Botulinum Neurotoxin Abreakdown → | 584 |
About Felix L. Yeh
Felix L. Yeh is a scholar working on Neurology, Biological Psychiatry and Neurology, having authored 24 papers that have together received 1.9k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (8 papers), Amyotrophic Lateral Sclerosis Research (5 papers) and Inflammation biomarkers and pathways (5 papers). The work is most often cited by research in Neurology (760 citations), Biological Psychiatry (117 citations) and Neurology (667 citations). Felix L. Yeh has collaborated with scholars based in United States, France and Germany. Frequent co-authors include Morgan Sheng, Irene Tom, Yuanyuan Wang, Lino C. Gonzalez, Edwin R. Chapman, Matthew H. Bailey, Eric A. Johnson, William H. Tepp, Min Dong and Camin Dean. Their work appears in journals such as Science, Neuron and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.