Tina Schwabe
- Neurology top 1%
- Cellular and Molecular Neuroscience top 5%
- Molecular Biology
- Immunology top 10%
- Physiology top 10%
- Co-authors
- Ulrike GaulRoland J. BaintonUlrike HeberleinHervé RhinnArnon RosenthalRichard D. FetterIlaria TassiAdiljan Ibrahim
- Topics
- Neurobiology and Insect Physiology Research (8 papers)Neuroinflammation and Neurodegeneration Mechanisms (7 papers)Inflammation biomarkers and pathways (6 papers)
- Cited by
- NeurologyBiological PsychiatryAging
- Partner nations
- GermanyUnited StatesChina
In The Last Decade
Tina Schwabe
29 papers receiving 1.4k citations
Hit Papers
Peers
Comparison fields: 5 of 89
- Neurology 629
- Cellular and Molecular Neuroscience 498
- Molecular Biology 445
- Immunology 394
- Physiology 301
Countries citing papers authored by Tina Schwabe
This map shows the geographic impact of Tina Schwabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tina Schwabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tina Schwabe more than expected).
Fields of papers citing papers by Tina Schwabe
This network shows the impact of papers produced by Tina Schwabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tina Schwabe. The network helps show where Tina Schwabe may publish in the future.
Co-authorship network of co-authors of Tina Schwabe
This figure shows the co-authorship network connecting the top 25 collaborators of Tina Schwabe. A scholar is included among the top collaborators of Tina Schwabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tina Schwabe. Tina Schwabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 38 | |
| 3 | 3 | |
| 4 | 4 | |
| 5 | 1 | |
| 6 | 15 | |
| 7 | 10 | |
| 8 | 1 | |
| 9 | 257 | |
| 10 | Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease modelbreakdown → | 345 |
| 11 | 58 | |
| 12 | 2 | |
| 13 | 2 | |
| 14 | 39 | |
| 15 | 38 | |
| 16 | 52 | |
| 17 | 7 | |
| 18 | 183 | |
| 19 | 186 | |
| 20 | 90 |
About Tina Schwabe
Tina Schwabe is a scholar working on Neurology, Cellular and Molecular Neuroscience and Immunology, having authored 30 papers that have together received 1.4k indexed citations. Recurring topics across this work include Neurobiology and Insect Physiology Research (8 papers), Neuroinflammation and Neurodegeneration Mechanisms (7 papers) and Inflammation biomarkers and pathways (6 papers). The work is most often cited by research in Neurology (629 citations), Biological Psychiatry (103 citations) and Aging (66 citations). Tina Schwabe has collaborated with scholars based in Germany, United States and China. Frequent co-authors include Ulrike Gaul, Roland J. Bainton, Ulrike Heberlein, Hervé Rhinn, Arnon Rosenthal, Richard D. Fetter, Ilaria Tassi, Adiljan Ibrahim, Thomas R. Clandinin and Linus Tsai. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.