Andrew E. Arrant

1.4k total citations
29 papers, 946 citations indexed

About

Andrew E. Arrant is a scholar working on Neurology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Andrew E. Arrant has authored 29 papers receiving a total of 946 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Neurology, 10 papers in Cellular and Molecular Neuroscience and 10 papers in Physiology. Recurrent topics in Andrew E. Arrant's work include Amyotrophic Lateral Sclerosis Research (16 papers), Parkinson's Disease Mechanisms and Treatments (11 papers) and Alzheimer's disease research and treatments (9 papers). Andrew E. Arrant is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (16 papers), Parkinson's Disease Mechanisms and Treatments (11 papers) and Alzheimer's disease research and treatments (9 papers). Andrew E. Arrant collaborates with scholars based in United States, Germany and Bulgaria. Andrew E. Arrant's co-authors include Cynthia M. Kuhn, Erik D. Roberson, Nicole L. Schramm‐Sapyta, Anthony J. Filiano, Amanda E.D. Van Swearingen, Florian Daniel Zepf, Cristina Matesanz López, Jeremy H. Herskowitz, Erik G. Gentry and Benjamin W. Henderson and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Experimental Medicine and Journal of Neuroscience.

In The Last Decade

Andrew E. Arrant

27 papers receiving 930 citations

Peers

Andrew E. Arrant
Andrew E. Arrant
Citations per year, relative to Andrew E. Arrant Andrew E. Arrant (= 1×) peers Andrea B. Cragnolini

Countries citing papers authored by Andrew E. Arrant

Since Specialization
Citations

This map shows the geographic impact of Andrew E. Arrant's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew E. Arrant with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew E. Arrant more than expected).

Fields of papers citing papers by Andrew E. Arrant

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew E. Arrant. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew E. Arrant. The network helps show where Andrew E. Arrant may publish in the future.

Co-authorship network of co-authors of Andrew E. Arrant

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew E. Arrant. A scholar is included among the top collaborators of Andrew E. Arrant based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew E. Arrant. Andrew E. Arrant is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Vollmer, G, et al.. (2025). TFEB overexpression alleviates autophagy-lysosomal deficits caused by progranulin insufficiency. Scientific Reports. 15(1). 26217–26217.
2.
Tadepalli, Anjaneyulu S., Nicholas T. Seyfried, Thomas Kukar, et al.. (2025). Reduction of sphingomyelinase activity associated with progranulin deficiency and frontotemporal dementia. Neurobiology of Disease. 213. 107024–107024.
3.
Murchison, Charles, et al.. (2024). Increased levels of extracellular matrix proteins associated with extracellular vesicles from brains of aged mice. Aging Cell. 24(1). e14359–e14359. 1 indexed citations
4.
Oliveira, José Fernando de, et al.. (2024). Adolescent stress accelerates postpartum novelty recognition impairment in 5xFAD mice. Frontiers in Neuroscience. 18. 1366199–1366199. 2 indexed citations
5.
Greathouse, Kelsey M., et al.. (2024). Dendritic spine head diameter is reduced in the prefrontal cortex of progranulin haploinsufficient mice. Molecular Brain. 17(1). 33–33. 1 indexed citations
6.
Jaunarajs, Karen L. Eskow, et al.. (2023). Regulation of extracellular progranulin in medial prefrontal cortex. Neurobiology of Disease. 188. 106326–106326. 2 indexed citations
7.
Stoyka, Lindsay E., et al.. (2019). Behavioral defects associated with amygdala and cortical dysfunction in mice with seeded α-synuclein inclusions. Neurobiology of Disease. 134. 104708–104708. 48 indexed citations
8.
Arrant, Andrew E., Charles Murchison, Eliana Marisa Ramos, et al.. (2019). Impaired β-glucocerebrosidase activity and processing in frontotemporal dementia due to progranulin mutations. Acta Neuropathologica Communications. 7(1). 218–218. 43 indexed citations
9.
Arrant, Andrew E., et al.. (2018). Reduction of microglial progranulin does not exacerbate pathology or behavioral deficits in neuronal progranulin-insufficient mice. Neurobiology of Disease. 124. 152–162. 18 indexed citations
10.
Arrant, Andrew E., Alexandra M. Nicholson, Xiaolai Zhou, Rosa Rademakers, & Erik D. Roberson. (2018). Partial Tmem106b reduction does not correct abnormalities due to progranulin haploinsufficiency. Molecular Neurodegeneration. 13(1). 32–32. 22 indexed citations
11.
Arrant, Andrew E., et al.. (2018). Progranulin Gene Therapy Improves Lysosomal Dysfunction and Microglial Pathology Associated with Frontotemporal Dementia and Neuronal Ceroid Lipofuscinosis. Journal of Neuroscience. 38(9). 2341–2358. 111 indexed citations
12.
Gentry, Erik G., Benjamin W. Henderson, Andrew E. Arrant, et al.. (2016). Rho Kinase Inhibition as a Therapeutic for Progressive Supranuclear Palsy and Corticobasal Degeneration. Journal of Neuroscience. 36(4). 1316–1323. 73 indexed citations
13.
Arrant, Andrew E., et al.. (2015). Effects of Exercise on Progranulin Levels and Gliosis in Progranulin-Insufficient Mice. eNeuro. 2(3). ENEURO.0061–14.2015. 6 indexed citations
14.
López, Cristina Matesanz, et al.. (2015). Simplified dietary acute tryptophan depletion: effects of a novel amino acid mixture on the neurochemistry of C57BL/6J mice. Food & Nutrition Research. 59(1). 27424–27424. 14 indexed citations
15.
González-Hunt, Claudia P., Maxwell C. K. Leung, Rakesh Bodhicharla, et al.. (2014). Exposure to Mitochondrial Genotoxins and Dopaminergic Neurodegeneration in Caenorhabditis elegans. PLoS ONE. 9(12). e114459–e114459. 68 indexed citations
17.
Arrant, Andrew E., Nicole L. Schramm‐Sapyta, & Cynthia M. Kuhn. (2013). Use of the light/dark test for anxiety in adult and adolescent male rats. Behavioural Brain Research. 256. 119–127. 140 indexed citations
18.
López, Cristina Matesanz, Amanda E.D. Van Swearingen, Andrew E. Arrant, Cynthia M. Kuhn, & Florian Daniel Zepf. (2013). Dietary manipulation of serotonergic and dopaminergic function in C57BL/6J mice with amino acid depletion mixtures. Journal of Neural Transmission. 121(2). 153–162. 12 indexed citations
19.
López, Cristina Matesanz, et al.. (2012). Effects of Acute Tryptophan Depletion on Brain Serotonin Function and Concentrations of Dopamine and Norepinephrine in C57BL/6J and BALB/cJ Mice. PLoS ONE. 7(5). e35916–e35916. 59 indexed citations
20.
Walker, Q. David, et al.. (2010). Dopamine Uptake Inhibitors but Not Dopamine Releasers Induce Greater Increases in Motor Behavior and Extracellular Dopamine in Adolescent Rats Than in Adult Male Rats. Journal of Pharmacology and Experimental Therapeutics. 335(1). 124–132. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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