Félix Hernández
- Molecular Biology top 1%
- Physiology top 0.2%
- Cellular and Molecular Neuroscience top 0.5%
- Neurology top 0.5%
- Pharmacology top 0.5%
- Co-authors
- Jesús ÁvilaJosé J. LucasMar PérezMaría Llorens‐MartínAlmudena Fuster‐MatanzoTobías EngelMiguel Díaz‐HernándezJerónimo Jurado‐Arjona
- Topics
- Alzheimer's disease research and treatments (109 papers)Neuroscience and Neuropharmacology Research (60 papers)Wnt/β-catenin signaling in development and cancer (26 papers)
- Partner nations
- SpainUnited StatesIreland
In The Last Decade
Félix Hernández
193 papers receiving 9.4k citations
Hit Papers
Peers
Comparison fields: 5 of 133
- Molecular Biology 4.9k
- Physiology 4.7k
- Cellular and Molecular Neuroscience 3.1k
- Neurology 1.5k
- Pharmacology 1.3k
Countries citing papers authored by Félix Hernández
This map shows the geographic impact of Félix Hernández's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Félix Hernández with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Félix Hernández more than expected).
Fields of papers citing papers by Félix Hernández
This network shows the impact of papers produced by Félix Hernández. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Félix Hernández. The network helps show where Félix Hernández may publish in the future.
Co-authorship network of co-authors of Félix Hernández
This figure shows the co-authorship network connecting the top 25 collaborators of Félix Hernández. A scholar is included among the top collaborators of Félix Hernández based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Félix Hernández. Félix Hernández is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 2 | |
| 4 | 4 | |
| 5 | 10 | |
| 6 | 58 | |
| 7 | 14 | |
| 8 | 21 | |
| 9 | 19 | |
| 10 | 28 | |
| 11 | 28 | |
| 12 | 11 | |
| 13 | 81 | |
| 14 | 1 | |
| 15 | 15 | |
| 16 | 20 | |
| 17 | 28 | |
| 18 | 33 | |
| 19 | 33 | |
| 20 | 2 |
About Félix Hernández
Félix Hernández is a scholar working on Physiology, Cellular and Molecular Neuroscience and Developmental Neuroscience, having authored 199 papers that have together received 9.5k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (109 papers), Neuroscience and Neuropharmacology Research (60 papers) and Wnt/β-catenin signaling in development and cancer (26 papers). The work is most often cited by research in Neurology (1.5k citations), Physiology (4.7k citations) and Developmental Neuroscience (722 citations). Félix Hernández has collaborated with scholars based in Spain, United States and Ireland. Frequent co-authors include Jesús Ávila, José J. Lucas, Mar Pérez, María Llorens‐Martín, Almudena Fuster‐Matanzo, Tobías Engel, Miguel Díaz‐Hernández, Jerónimo Jurado‐Arjona, Raquel Cuadros and Elena Gómez de Barreda. Their work appears in journals such as Journal of Biological Chemistry, Nature Medicine and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.