Fei Liu

12.2k total citations · 3 hit papers
127 papers, 9.6k citations indexed

About

Fei Liu is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Fei Liu has authored 127 papers receiving a total of 9.6k indexed citations (citations by other indexed papers that have themselves been cited), including 92 papers in Physiology, 64 papers in Molecular Biology and 27 papers in Cellular and Molecular Neuroscience. Recurrent topics in Fei Liu's work include Alzheimer's disease research and treatments (89 papers), Neuroscience and Neuropharmacology Research (21 papers) and Prion Diseases and Protein Misfolding (15 papers). Fei Liu is often cited by papers focused on Alzheimer's disease research and treatments (89 papers), Neuroscience and Neuropharmacology Research (21 papers) and Prion Diseases and Protein Misfolding (15 papers). Fei Liu collaborates with scholars based in United States, China and Japan. Fei Liu's co-authors include Cheng‐Xin Gong, Khalid Iqbal, Inge Grundke‐Iqbal, Ying Liu, Chun‐Ling Dai, Jian‐Zhi Wang, Alejandra del C. Alonso, Zhihou Liang, Jianhua Shi and Bin Li and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and The Journal of Cell Biology.

In The Last Decade

Fei Liu

126 papers receiving 9.5k citations

Hit Papers

Tau pathology in Alzheimer disease and other tauopathies 2004 2026 2011 2018 2004 2005 2015 250 500 750

Peers

Fei Liu
Antonella Caccamo United States
Hyoung‐gon Lee United States
Inhee Mook‐Jung South Korea
Wendy Noble United Kingdom
Daniel C. Lee United States
Milene L. Brownlow United States
Fei Liu
Citations per year, relative to Fei Liu Fei Liu (= 1×) peers Jian‐Zhi Wang

Countries citing papers authored by Fei Liu

Since Specialization
Citations

This map shows the geographic impact of Fei Liu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fei Liu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fei Liu more than expected).

Fields of papers citing papers by Fei Liu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fei Liu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fei Liu. The network helps show where Fei Liu may publish in the future.

Co-authorship network of co-authors of Fei Liu

This figure shows the co-authorship network connecting the top 25 collaborators of Fei Liu. A scholar is included among the top collaborators of Fei Liu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fei Liu. Fei Liu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gu, Jianlan, et al.. (2025). Rhythmic TDP-43 affects RNA splicing of USP13, resulting in alteration of BMAL1 ubiquitination. The Journal of Cell Biology. 224(5). 1 indexed citations
2.
Wang, Ruci, Yuji Murayama, Fei Liu, et al.. (2025). Impact of urban morphology on land surface temperature: A case study of the central Tokyo, Japan. City and Environment Interactions. 28. 100227–100227. 1 indexed citations
3.
Liu, Fei, Nana Jin, Dandan Chu, et al.. (2023). Two simple assays for assessing the seeding activity of proteopathic tau. Frontiers in Aging Neuroscience. 15. 1 indexed citations
5.
Wei, Wei, Yinghua Liu, Chun‐Ling Dai, et al.. (2021). Neurotrophic Treatment Initiated During Early Postnatal Development Prevents the Alzheimer-Like Behavior and Synaptic Dysfunction. Journal of Alzheimer s Disease. 82(2). 631–646. 15 indexed citations
6.
Li, Longfei, Ruirui Shi, Jianlan Gu, et al.. (2021). Alzheimer’s disease brain contains tau fractions with differential prion-like activities. Acta Neuropathologica Communications. 9(1). 28–28. 44 indexed citations
7.
Dai, Chun‐Ling, Xin Hu, Jin Zhang, et al.. (2020). Neonatal Exposure to Anesthesia Leads to Cognitive Deficits in Old Age: Prevention with Intranasal Administration of Insulin in Mice. Neurotoxicity Research. 38(2). 299–311. 20 indexed citations
8.
Dai, Chun‐Ling, Jin‐Hua Gu, Fei Liu, Khalid Iqbal, & Cheng‐Xin Gong. (2018). Neuronal O-GlcNAc transferase regulates appetite, body weight, and peripheral insulin resistance. Neurobiology of Aging. 70. 40–50. 21 indexed citations
9.
Gu, Jin‐Hua, Jianhua Shi, Chun‐Ling Dai, et al.. (2017). O-GlcNAcylation Reduces Ischemia-Reperfusion–Induced Brain Injury. Scientific Reports. 7(1). 10686–10686. 34 indexed citations
10.
Gu, Jianlan, Feng Chen, Khalid Iqbal, et al.. (2017). Transactive response DNA-binding protein 43 (TDP-43) regulates alternative splicing of tau exon 10: Implications for the pathogenesis of tauopathies. Journal of Biological Chemistry. 292(25). 10600–10612. 65 indexed citations
11.
Shi, Jianhua, Jin‐Hua Gu, Chun‐Ling Dai, et al.. (2015). O-GlcNAcylation regulates ischemia-induced neuronal apoptosis through AKT signaling. Scientific Reports. 5(1). 14500–14500. 64 indexed citations
12.
Jin, Nana, Xiaomin Yin, Jianlan Gu, et al.. (2015). Truncation and Activation of Dual Specificity Tyrosine Phosphorylation-regulated Kinase 1A by Calpain I. Journal of Biological Chemistry. 290(24). 15219–15237. 49 indexed citations
13.
Yu, Yang, Xiaojing Li, Julie Blanchard, et al.. (2014). Insulin sensitizers improve learning and attenuate tau hyperphosphorylation and neuroinflammation in 3xTg-AD mice. Journal of Neural Transmission. 122(4). 593–606. 55 indexed citations
14.
Arif, Mohammad Ali, Jianshe Wei, Qi Zhang, et al.. (2014). Cytoplasmic Retention of Protein Phosphatase 2A Inhibitor 2 (I2PP2A) Induces Alzheimer-like Abnormal Hyperphosphorylation of Tau. Journal of Biological Chemistry. 289(40). 27677–27691. 46 indexed citations
15.
Yin, Xiaomin, Nana Jin, Jianlan Gu, et al.. (2012). Dual-specificity Tyrosine Phosphorylation-regulated Kinase 1A (Dyrk1A) Modulates Serine/Arginine-rich Protein 55 (SRp55)-promoted Tau Exon 10 Inclusion. Journal of Biological Chemistry. 287(36). 30497–30506. 79 indexed citations
16.
Liu, Ying, Fei Liu, Inge Grundke‐Iqbal, Khalid Iqbal, & Cheng‐Xin Gong. (2009). Brain glucose transporters, O‐GlcNAcylation and phosphorylation of tau in diabetes and Alzheimer’s disease. Journal of Neurochemistry. 111(1). 242–249. 160 indexed citations
17.
Liu, Ying, Fei Liu, Khalid Iqbal, Inge Grundke‐Iqbal, & Cheng‐Xin Gong. (2008). Decreased glucose transporters correlate to abnormal hyperphosphorylation of tau in Alzheimer disease. FEBS Letters. 582(2). 359–364. 302 indexed citations
18.
Liu, Fei, et al.. (2007). Site‐specific effects of tau phosphorylation on its microtubule assembly activity and self‐aggregation. European Journal of Neuroscience. 26(12). 3429–3436. 161 indexed citations
19.
Liang, Zhihou, Fei Liu, Inge Grundke‐Iqbal, Khalid Iqbal, & Cheng‐Xin Gong. (2007). Down‐regulation of cAMP‐dependent protein kinase by over‐activated calpain in Alzheimer disease brain. Journal of Neurochemistry. 103(6). 2462–2470. 113 indexed citations
20.
Gong, Cheng‐Xin, Fei Liu, Inge Grundke‐Iqbal, & Khalid Iqbal. (2006). Dysregulation of Protein Phosphorylation/Dephosphorylation in Alzheimer′s Disease: A Therapeutic Target. BioMed Research International. 2006(1). 31825–31825. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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