Fadil Hannan

4.6k total citations · 3 hit papers
67 papers, 2.6k citations indexed

About

Fadil Hannan is a scholar working on Nephrology, Molecular Biology and Physiology. According to data from OpenAlex, Fadil Hannan has authored 67 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Nephrology, 31 papers in Molecular Biology and 15 papers in Physiology. Recurrent topics in Fadil Hannan's work include Parathyroid Disorders and Treatments (39 papers), Erythrocyte Function and Pathophysiology (13 papers) and Bone health and treatments (11 papers). Fadil Hannan is often cited by papers focused on Parathyroid Disorders and Treatments (39 papers), Erythrocyte Function and Pathophysiology (13 papers) and Bone health and treatments (11 papers). Fadil Hannan collaborates with scholars based in United Kingdom, United States and Australia. Fadil Hannan's co-authors include Rajesh V. Thakker, Valerie Babinsky, M. Andrew Nesbit, Enikö Kállay, Maria Luisa Brandi, Treena Cranston, Wenhan Chang, Sarah Howles, Nigel Rust and Caroline M. Gorvin and has published in prestigious journals such as Nature, New England Journal of Medicine and Journal of Biological Chemistry.

In The Last Decade

Fadil Hannan

62 papers receiving 2.5k citations

Hit Papers

The calcium-sensing receptor in physiology and in calcitr... 2018 2026 2020 2023 2018 2021 2022 50 100 150 200

Peers

Fadil Hannan
Elena Ambrogini United States
Fadil Hannan
Citations per year, relative to Fadil Hannan Fadil Hannan (= 1×) peers Elena Ambrogini

Countries citing papers authored by Fadil Hannan

Since Specialization
Citations

This map shows the geographic impact of Fadil Hannan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fadil Hannan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fadil Hannan more than expected).

Fields of papers citing papers by Fadil Hannan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fadil Hannan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fadil Hannan. The network helps show where Fadil Hannan may publish in the future.

Co-authorship network of co-authors of Fadil Hannan

This figure shows the co-authorship network connecting the top 25 collaborators of Fadil Hannan. A scholar is included among the top collaborators of Fadil Hannan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fadil Hannan. Fadil Hannan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hannan, Fadil, Kreepa Kooblall, Mark Stevenson, et al.. (2025). Characterization of quinazolinone calcilytic therapy for autosomal dominant hypocalcemia type 1 (ADH1). Journal of Biological Chemistry. 301(4). 108404–108404.
2.
Hannan, Fadil, Melvin Khee‐Shing Leow, Jason Lee, et al.. (2024). Endocrine effects of heat exposure and relevance to climate change. Nature Reviews Endocrinology. 20(11). 673–684. 16 indexed citations
3.
Zecchin, Davide, Nicole Knöpfel, Mark Stevenson, et al.. (2023). GNAQ/GNA11 Mosaicism Causes Aberrant Calcium Signaling Susceptible to Targeted Therapeutics. Journal of Investigative Dermatology. 144(4). 811–819.e4. 7 indexed citations
4.
Lovegrove, Catherine, Jelena Bešević, Akira Wiberg, et al.. (2023). Central Adiposity Increases Risk of Kidney Stone Disease through Effects on Serum Calcium Concentrations. Journal of the American Society of Nephrology. 34(12). 1991–2011. 22 indexed citations
5.
Elston, Marianne S., Taha Elajnaf, Fadil Hannan, & Rajesh V. Thakker. (2022). Autosomal Dominant Hypocalcemia Type 1 (ADH1) Associated With Myoclonus and Intracerebral Calcifications. Journal of the Endocrine Society. 6(5). bvac042–bvac042. 7 indexed citations
6.
Price, Helen P., Taha Elajnaf, R. R. Humphrey, et al.. (2022). Protocol for an observational study investigating hormones triggering the onset of sustained lactation: the INSIGHT study. BMJ Open. 12(8). e062478–e062478.
7.
Gao, Yang, Michael J. Robertson, Alpay B. Seven, et al.. (2021). Asymmetric activation of the calcium-sensing receptor homodimer. Nature. 595(7867). 455–459. 86 indexed citations
8.
Lanzer, Peter, Fadil Hannan, Jan D. Lanzer, et al.. (2021). Medial Arterial Calcification. Journal of the American College of Cardiology. 78(11). 1145–1165. 187 indexed citations breakdown →
9.
Gorvin, Caroline M., Raghu Metpally, Diane T. Smelser, et al.. (2020). Familial Hypocalciuric Hypercalcemia Type 1 and Autosomal-Dominant Hypocalcemia Type 1: Prevalence in a Large Healthcare Population. The American Journal of Human Genetics. 106(6). 734–747. 48 indexed citations
10.
Gorvin, Caroline M., Valerie Babinsky, Tomas Malinauskas, et al.. (2018). A calcium-sensing receptor mutation causing hypocalcemia disrupts a transmembrane salt bridge to activate β-arrestin–biased signaling. Science Signaling. 11(518). 31 indexed citations
11.
Stokes, Victoria, Morten Frost Nielsen, Fadil Hannan, & Rajesh V. Thakker. (2017). Hypercalcemic Disorders in Children. Journal of Bone and Mineral Research. 32(11). 2157–2170. 72 indexed citations
12.
Babinsky, Valerie, Fadil Hannan, Reshma Ramracheya, et al.. (2017). Mutant Mice With Calcium-Sensing Receptor Activation Have Hyperglycemia That Is Rectified by Calcilytic Therapy. Endocrinology. 158(8). 2486–2502. 24 indexed citations
13.
Esapa, Christopher T., Fadil Hannan, Valerie Babinsky, et al.. (2015). N-ethyl-N-Nitrosourea (ENU) Induced Mutations within the Klotho Gene Lead to Ectopic Calcification and Reduced Lifespan in Mouse Models. PLoS ONE. 10(4). e0122650–e0122650. 16 indexed citations
14.
Hannan, Fadil & Rajesh V. Thakker. (2013). Calcium-sensing receptor (CaSR) mutations and disorders of calcium, electrolyte and water metabolism. Best Practice & Research Clinical Endocrinology & Metabolism. 27(3). 359–371. 92 indexed citations
15.
Nesbit, M. Andrew, Fadil Hannan, Sarah Howles, et al.. (2013). Mutations Affecting G-Protein Subunit α11in Hypercalcemia and Hypocalcemia. New England Journal of Medicine. 368(26). 2476–2486. 240 indexed citations
16.
Hannan, Fadil, M. Andrew Nesbit, Treena Cranston, & Rajesh V. Thakker. (2012). A calcium-sensing receptor (CaSR) variant, Glu250Lys, present in familial hypocalciuric hypercalcaemia (FHH) and autosomal dominant hypocalcaemic hypercalciuria (ADHH) probands represents a functionally neutral polymorphism: lessons for CaSR mutational analysis. 28. 3 indexed citations
17.
18.
Hannan, Fadil, Gerard Walls, Enikö Kállay, et al.. (2008). The calcilytic agent NPS2143 rectifies hypocalcaemia in a mouse model, Nuf, that is due to an activating calcium-sensing-receptor (CaSR) mutation: relevance to autosomal dominant hypocalcaemia with hypercalciuria. 15. 1 indexed citations
19.
Hannan, Fadil, Nicholas A. Athanasou, James Teh, et al.. (2008). Oncogenic hypophosphataemic osteomalacia: biomarker roles of fibroblast growth factor 23, 1,25-dihydroxyvitamin D3 and lymphatic vessel endothelial hyaluronan receptor 1. European Journal of Endocrinology. 158(2). 265–271. 25 indexed citations
20.
Hannan, Fadil, Paul T. Christie, Willy Lissens, et al.. (2007). A novel homozygous inactivating mutation, Pro339Thr, of the calcium-sensing receptor is associated with isolated primary hyperparathyroidism. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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