Caroline M. Gorvin

1.9k total citations
63 papers, 1.1k citations indexed

About

Caroline M. Gorvin is a scholar working on Molecular Biology, Nephrology and Oncology. According to data from OpenAlex, Caroline M. Gorvin has authored 63 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 20 papers in Nephrology and 14 papers in Oncology. Recurrent topics in Caroline M. Gorvin's work include Parathyroid Disorders and Treatments (18 papers), Erythrocyte Function and Pathophysiology (11 papers) and Receptor Mechanisms and Signaling (11 papers). Caroline M. Gorvin is often cited by papers focused on Parathyroid Disorders and Treatments (18 papers), Erythrocyte Function and Pathophysiology (11 papers) and Receptor Mechanisms and Signaling (11 papers). Caroline M. Gorvin collaborates with scholars based in United Kingdom, United States and Denmark. Caroline M. Gorvin's co-authors include Rajesh V. Thakker, Fadil Hannan, M. Andrew Nesbit, Paul Newey, Treena Cranston, Siân E. Piret, Asuka Inoue, Gerda E. Breitwieser, Morten Frost and Angela Rogers and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Caroline M. Gorvin

59 papers receiving 1.1k citations

Peers

Caroline M. Gorvin
Marcello Filopanti Italy
Yah‐Huei Wu Chou Taiwan
Alda Tufró United States
Edna E. Mancilla United States
Lucia Anna Muscarella Italy
Masakatsu Fukuda Japan
Ci-Di Chen United States
Xinli Shao United States
Bei Guo China
Belén Bornstein Spain
Marcello Filopanti Italy
Caroline M. Gorvin
Citations per year, relative to Caroline M. Gorvin Caroline M. Gorvin (= 1×) peers Marcello Filopanti

Countries citing papers authored by Caroline M. Gorvin

Since Specialization
Citations

This map shows the geographic impact of Caroline M. Gorvin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Caroline M. Gorvin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Caroline M. Gorvin more than expected).

Fields of papers citing papers by Caroline M. Gorvin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Caroline M. Gorvin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Caroline M. Gorvin. The network helps show where Caroline M. Gorvin may publish in the future.

Co-authorship network of co-authors of Caroline M. Gorvin

This figure shows the co-authorship network connecting the top 25 collaborators of Caroline M. Gorvin. A scholar is included among the top collaborators of Caroline M. Gorvin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Caroline M. Gorvin. Caroline M. Gorvin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Broichhagen, Johannes, et al.. (2026). MRAP2 potentiates GPCR signaling by conserved mechanisms that are disrupted by obesity-associated genetic variants. Cell Reports. 45(3). 117018–117018.
2.
Thompson, Miles D., Peter Chidiac, Pedro A. José, Alexander S. Hauser, & Caroline M. Gorvin. (2025). Genetic variants of accessory proteins and G proteins in human genetic disease. Critical Reviews in Clinical Laboratory Sciences. 62(2). 113–134. 1 indexed citations
3.
Jamaluddin, Aqfan, Joon Lee, Georgina K.C. Dowsett, et al.. (2025). The accessory protein MRAP2 directly interacts with melanocortin-3 receptor to enhance signaling. Science Signaling. 18(917). eadu4315–eadu4315. 1 indexed citations
4.
Thompson, Miles D., Maire E. Percy, David E.C. Cole, et al.. (2024). G protein-coupled receptor (GPCR) gene variants and human genetic disease. Critical Reviews in Clinical Laboratory Sciences. 61(5). 317–346. 10 indexed citations
5.
Hansen, Morten Steen, Kent Søe, Yasunori Omata, et al.. (2024). Transcriptional reprogramming during human osteoclast differentiation identifies regulators of osteoclast activity. Bone Research. 12(1). 5–5. 26 indexed citations
6.
Gorvin, Caroline M.. (2024). Measuring IP3 Generation in Real-Time Using a NanoBiT Luminescence Biosensor. Methods in molecular biology. 2861. 33–42. 4 indexed citations
7.
Gorvin, Caroline M.. (2024). Measuring Calcium-Sensing Receptor Agonist-Driven Insertional Signaling (ADIS) and Trafficking by TIRF Microscopy. Methods in molecular biology. 2861. 111–126. 1 indexed citations
8.
Read, Martin L., Selvambigai Manivannan, Jana Kim, et al.. (2023). Combined Vorinostat and Chloroquine Inhibit Sodium–Iodide Symporter Endocytosis and Enhance Radionuclide Uptake In Vivo. Clinical Cancer Research. 30(7). 1352–1366. 8 indexed citations
9.
Gorvin, Caroline M.. (2022). Recent advances in calcium-sensing receptor structures and signaling pathways. Progress in molecular biology and translational science. 195. 121–135. 2 indexed citations
10.
Gorvin, Caroline M., Raghu Metpally, Diane T. Smelser, et al.. (2020). Familial Hypocalciuric Hypercalcemia Type 1 and Autosomal-Dominant Hypocalcemia Type 1: Prevalence in a Large Healthcare Population. The American Journal of Human Genetics. 106(6). 734–747. 48 indexed citations
11.
Gorvin, Caroline M., Valerie Babinsky, Tomas Malinauskas, et al.. (2018). A calcium-sensing receptor mutation causing hypocalcemia disrupts a transmembrane salt bridge to activate β-arrestin–biased signaling. Science Signaling. 11(518). 31 indexed citations
12.
Babinsky, Valerie, Fadil Hannan, Reshma Ramracheya, et al.. (2017). Mutant Mice With Calcium-Sensing Receptor Activation Have Hyperglycemia That Is Rectified by Calcilytic Therapy. Endocrinology. 158(8). 2486–2502. 24 indexed citations
13.
Howles, Sarah, Fadil Hannan, Caroline M. Gorvin, et al.. (2017). Cinacalcet corrects hypercalcemia in mice with an inactivating Gα11 mutation. JCI Insight. 2(20). 12 indexed citations
14.
Piret, Siân E., Caroline M. Gorvin, Anne Trinh, et al.. (2016). Autosomal dominant osteopetrosis associated with renal tubular acidosis is due to a CLCN7 mutation. American Journal of Medical Genetics Part A. 170(11). 2988–2992. 10 indexed citations
15.
Gorvin, Caroline M.. (2015). The prolactin receptor: Diverse and emerging roles in pathophysiology. Journal of Clinical & Translational Endocrinology. 2(3). 85–91. 44 indexed citations
16.
Gorvin, Caroline M., Martijn J. Wilmer, Siân E. Piret, et al.. (2013). Receptor-mediated endocytosis and endosomal acidification is impaired in proximal tubule epithelial cells of Dent disease patients. Proceedings of the National Academy of Sciences. 110(17). 7014–7019. 67 indexed citations
17.
Loh, Nellie Y., Liz Bentley, Henrik Dimke, et al.. (2013). Autosomal Dominant Hypercalciuria in a Mouse Model Due to a Mutation of the Epithelial Calcium Channel, TRPV5. PLoS ONE. 8(1). e55412–e55412. 27 indexed citations
18.
Newey, Paul, Caroline M. Gorvin, Stephen J. Cleland, et al.. (2013). Mutant Prolactin Receptor and Familial Hyperprolactinemia. New England Journal of Medicine. 369(21). 2012–2020. 77 indexed citations
19.
Piret, Siân E., Christopher T. Esapa, Caroline M. Gorvin, et al.. (2012). A Mouse Model of Early-Onset Renal Failure Due to a Xanthine Dehydrogenase Nonsense Mutation. PLoS ONE. 7(9). e45217–e45217. 8 indexed citations
20.
Newey, Paul, M. Andrew Nesbit, Andrew J. Rimmer, et al.. (2012). Whole-Exome Sequencing Studies of Nonhereditary (Sporadic) Parathyroid Adenomas. The Journal of Clinical Endocrinology & Metabolism. 97(10). E1995–E2005. 95 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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