Emel Esen

1.3k total citations
10 papers, 965 citations indexed

About

Emel Esen is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Emel Esen has authored 10 papers receiving a total of 965 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Immunology and 3 papers in Oncology. Recurrent topics in Emel Esen's work include Wnt/β-catenin signaling in development and cancer (3 papers), Cancer, Hypoxia, and Metabolism (2 papers) and CAR-T cell therapy research (2 papers). Emel Esen is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (3 papers), Cancer, Hypoxia, and Metabolism (2 papers) and CAR-T cell therapy research (2 papers). Emel Esen collaborates with scholars based in United States, Switzerland and United Kingdom. Emel Esen's co-authors include Fanxin Long, Courtney M. Karner, Adewole L. Okunade, Bruce W. Patterson, Jianquan Chen, Seung-Yon Lee, Burton M. Wice, Xiaolin Tu, Jeffrey M. Arbeit and Markus A. Rüegg and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Gastroenterology.

In The Last Decade

Emel Esen

10 papers receiving 961 citations

Peers

Emel Esen

MB

CR

ONCOL

PHYSI

GENET

Hwanhee Oh South Korea

Peng Deng United States

Pinglin Lai China

Shiqiao Ye United States

Erik R. Sampson United States

Shigehiro Ono Japan

Cassandra R. Diegel United States

Helmut Glantschnig Austria

Jolene Bohensky United States

Riccardo Chiusaroli United States

Hwanhee Oh South Korea

Emel Esen

653 ×1.0

MB

339 ×1.6

CR

156 ×0.8

ONCOL

79 ×0.6

PHYSI

131 ×1.1

GENET

Citations per year, relative to Emel Esen Emel Esen (= 1×) peers Hwanhee Oh

Countries citing papers authored by Emel Esen

Since Specialization

Citations

This map shows the geographic impact of Emel Esen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emel Esen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emel Esen more than expected).

Fields of papers citing papers by Emel Esen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emel Esen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emel Esen. The network helps show where Emel Esen may publish in the future.

Co-authorship network of co-authors of Emel Esen

This figure shows the co-authorship network connecting the top 25 collaborators of Emel Esen. A scholar is included among the top collaborators of Emel Esen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emel Esen. Emel Esen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Gampa, Gautham, Jennifer A. Getz, Yu Zhong, et al.. (2024). Preclinical and translational pharmacology of afucosylated anti‐CCR8 antibody for depletion of tumour‐infiltrating regulatory T cells. British Journal of Pharmacology. 181(13). 2033–2052. 3 indexed citations

2.

Esen, Emel, Ismail Sergin, Rajiv Jesudason, et al.. (2020). MAP4K4 negatively regulates CD8 T cell–mediated antitumor and antiviral immunity. Science Immunology. 5(45). 22 indexed citations

3.

Karner, Courtney M., Emel Esen, Jiakun Chen, et al.. (2016). Wnt Protein Signaling Reduces Nuclear Acetyl-CoA Levels to Suppress Gene Expression during Osteoblast Differentiation. Journal of Biological Chemistry. 291(25). 13028–13039. 43 indexed citations

4.

Sergin, Ismail, Somashubhra Bhattacharya, Roy Emanuel, et al.. (2016). Inclusion bodies enriched for p62 and polyubiquitinated proteins in macrophages protect against atherosclerosis. Science Signaling. 9(409). ra2–ra2. 85 indexed citations

5.

Esen, Emel, Seung-Yon Lee, Burton M. Wice, & Fanxin Long. (2015). PTH Promotes Bone Anabolism by Stimulating Aerobic Glycolysis via IGF Signaling. Journal of Bone and Mineral Research. 30(11). 1959–1968. 117 indexed citations

6.

Esen, Emel & Fanxin Long. (2014). Aerobic Glycolysis in Osteoblasts. Current Osteoporosis Reports. 12(4). 433–438. 71 indexed citations

7.

Chen, Jianquan, Xiaolin Tu, Emel Esen, et al.. (2014). WNT7B Promotes Bone Formation in part through mTORC1. PLoS Genetics. 10(1). e1004145–e1004145. 119 indexed citations

8.

Karner, Courtney M., Emel Esen, Adewole L. Okunade, Bruce W. Patterson, & Fanxin Long. (2014). Increased glutamine catabolism mediates bone anabolism in response to WNT signaling. Journal of Clinical Investigation. 125(2). 551–562. 123 indexed citations

9.

Esen, Emel, Jianquan Chen, Courtney M. Karner, et al.. (2013). WNT-LRP5 Signaling Induces Warburg Effect through mTORC2 Activation during Osteoblast Differentiation. Cell Metabolism. 17(5). 745–755. 286 indexed citations

10.

Huh, Won Jae, Emel Esen, Jessica H. Geahlen, et al.. (2010). XBP1 Controls Maturation of Gastric Zymogenic Cells by Induction of MIST1 and Expansion of the Rough Endoplasmic Reticulum. Gastroenterology. 139(6). 2038–2049. 96 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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