Peng Deng

1.7k total citations
20 papers, 1.2k citations indexed

About

Peng Deng is a scholar working on Molecular Biology, Genetics and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Peng Deng has authored 20 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 4 papers in Genetics and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Peng Deng's work include Epigenetics and DNA Methylation (9 papers), Cancer-related gene regulation (6 papers) and Mesenchymal stem cell research (4 papers). Peng Deng is often cited by papers focused on Epigenetics and DNA Methylation (9 papers), Cancer-related gene regulation (6 papers) and Mesenchymal stem cell research (4 papers). Peng Deng collaborates with scholars based in United States, China and India. Peng Deng's co-authors include Cun‐Yu Wang, Quan Yuan, Jiong Li, John M. Szymanski, Christine Hong, Sivakumar Ramadoss, Bo Yu, Insoon Chang, Yingduan Cheng and Dong-Qiu Dai and has published in prestigious journals such as Nature Communications, Cell stem cell and Arteriosclerosis Thrombosis and Vascular Biology.

In The Last Decade

Peng Deng

20 papers receiving 1.2k citations

Peers

Peng Deng

ONCOL

GENET

PHYSI

Ariane Galaup France

D. Feng United States

Beatriz Velasco Spain

Gal Akiri United States

Annalisa Antonini Italy

Ekaterina Klyachko United States

Sumithra Urs United States

Chad L. Barber United States

Mattia Capulli Italy

N. Skuli United States

Ariane Galaup France

Peng Deng

667 ×0.8

372 ×1.2

148 ×0.8

ONCOL

127 ×0.8

GENET

105 ×1.0

PHYSI

Citations per year, relative to Peng Deng Peng Deng (= 1×) peers Ariane Galaup

Countries citing papers authored by Peng Deng

Since Specialization

Citations

This map shows the geographic impact of Peng Deng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peng Deng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peng Deng more than expected).

Fields of papers citing papers by Peng Deng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peng Deng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peng Deng. The network helps show where Peng Deng may publish in the future.

Co-authorship network of co-authors of Peng Deng

This figure shows the co-authorship network connecting the top 25 collaborators of Peng Deng. A scholar is included among the top collaborators of Peng Deng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peng Deng. Peng Deng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhang, Danting, Weimin Lin, Shuang Jiang, et al.. (2023). Lepr‐Expressing PDLSCs Contribute to Periodontal Homeostasis and Respond to Mechanical Force by Piezo1. Advanced Science. 10(29). e2303291–e2303291. 35 indexed citations

Deng, Peng, Insoon Chang, Jiongke Wang, et al.. (2022). Loss of KDM4B impairs osteogenic differentiation of OMSCs and promotes oral bone aging. International Journal of Oral Science. 14(1). 24–24. 15 indexed citations

Liu, Zhenqing, Jin Sook Suh, Peng Deng, et al.. (2022). Epigenetic Regulation of NGF-Mediated Osteogenic Differentiation in Human Dental Mesenchymal Stem Cells. Stem Cells. 40(9). 818–830. 16 indexed citations

Liu, Zhenqing, Hye Lim Lee, Jin Sook Suh, et al.. (2022). The ERα/KDM6B regulatory axis modulates osteogenic differentiation in human mesenchymal stem cells. Bone Research. 10(1). 29 indexed citations

Deng, Peng, Quan Yuan, Yingduan Cheng, et al.. (2021). Loss of KDM4B exacerbates bone-fat imbalance and mesenchymal stromal cell exhaustion in skeletal aging. Cell stem cell. 28(6). 1057–1073.e7. 116 indexed citations

Gautam, Sanjeev K., et al.. (2021). Urine Cells-derived iPSCs: An Upcoming Frontier in Regenerative Medicine. Current Medicinal Chemistry. 28(31). 6484–6505. 3 indexed citations

Xu, Xiaoyang, Yan Xu, Peng Deng, et al.. (2020). SAMD14 promoter methylation is strongly associated with gene expression and poor prognosis in gastric cancer. International Journal of Clinical Oncology. 25(6). 1105–1114. 12 indexed citations

Wang, Yuan, Peng Deng, Yuting Liu, et al.. (2020). Alpha-ketoglutarate ameliorates age-related osteoporosis via regulating histone methylations. Nature Communications. 11(1). 5596–5596. 172 indexed citations

Yu, Bo, Lihong Huo, Yunsong Liu, et al.. (2018). PGC-1α Controls Skeletal Stem Cell Fate and Bone-Fat Balance in Osteoporosis and Skeletal Aging by Inducing TAZ. Cell stem cell. 23(2). 193–209.e5. 141 indexed citations

10.

Huang, Xiaofan, Yue Zhang, Jia Wu, et al.. (2018). MicroRNA-21 Knockout Exacerbates Angiotensin II–Induced Thoracic Aortic Aneurysm and Dissection in Mice With Abnormal Transforming Growth Factor-β–SMAD3 Signaling. Arteriosclerosis Thrombosis and Vascular Biology. 38(5). 1086–1101. 59 indexed citations

11.

Li, Jiong, Bo Yu, Peng Deng, et al.. (2017). KDM3 epigenetically controls tumorigenic potentials of human colorectal cancer stem cells through Wnt/β-catenin signalling. Nature Communications. 8(1). 15146–15146. 104 indexed citations

12.

Chen, Demeng, Man-Si Wu, Li Yang, et al.. (2017). Targeting BMI1 + Cancer Stem Cells Overcomes Chemoresistance and Inhibits Metastases in Squamous Cell Carcinoma. Cell stem cell. 20(5). 621–634.e6. 222 indexed citations

13.

Zhou, Chenchen, Qiuchan Xiong, Xinxing Zhu, et al.. (2017). AFF1 and AFF4 differentially regulate the osteogenic differentiation of human MSCs. Bone Research. 5(1). 17044–17044. 38 indexed citations

14.

Yu, Yongxin, Peng Deng, Bo Yu, et al.. (2017). Inhibition of EZH2 Promotes Human Embryonic Stem Cell Differentiation into Mesoderm by Reducing H3K27me3. Stem Cell Reports. 9(3). 752–761. 38 indexed citations

15.

Xu, Xiaoyang, Xiaoying Xue, Zhenhua Li, et al.. (2016). NDRG1 Controls Gastric Cancer Migration and Invasion through Regulating MMP-9. Pathology & Oncology Research. 22(4). 789–796. 24 indexed citations

16.

Deng, Peng, Chenchen Zhou, Ruth Álvarez, Christine Hong, & Cun‐Yu Wang. (2016). Inhibition of IKK/NF-κB Signaling Enhances Differentiation of Mesenchymal Stromal Cells from Human Embryonic Stem Cells. Stem Cell Reports. 6(4). 456–465. 46 indexed citations

17.

Xu, Xiaoyang, Zhenhua Li, Jiang Wang, et al.. (2015). Aberrant SOX11 promoter methylation is associated with poor prognosis in gastric cancer. Cellular Oncology. 38(3). 183–194. 27 indexed citations

18.

Xu, Xiaoyang, Xiaoying Xue, Zhenhua Li, et al.. (2014). NDRG1 expression is related to the progression and prognosis of gastric cancer patients through modulating proliferation, invasion and cell cycle of gastric cancer cells. Molecular Biology Reports. 41(9). 6215–6223. 29 indexed citations

19.

Li, Zhenhua, et al.. (2014). RASSF10 is an epigenetically silenced tumor suppressor in gastric cancer. Oncology Reports. 31(4). 1661–1668. 18 indexed citations

20.

Li, Zhenhua, et al.. (2012). DNA Methylation of NDRG2 in Gastric Cancer and Its Clinical Significance. Digestive Diseases and Sciences. 58(3). 715–723. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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