Edwin A. Azen

2.2k total citations
69 papers, 1.9k citations indexed

About

Edwin A. Azen is a scholar working on Physiology, Molecular Biology and Nutrition and Dietetics. According to data from OpenAlex, Edwin A. Azen has authored 69 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Physiology, 31 papers in Molecular Biology and 11 papers in Nutrition and Dietetics. Recurrent topics in Edwin A. Azen's work include Salivary Gland Disorders and Functions (31 papers), RNA and protein synthesis mechanisms (9 papers) and Biochemical Analysis and Sensing Techniques (9 papers). Edwin A. Azen is often cited by papers focused on Salivary Gland Disorders and Functions (31 papers), RNA and protein synthesis mechanisms (9 papers) and Biochemical Analysis and Sensing Techniques (9 papers). Edwin A. Azen collaborates with scholars based in United States, Canada and Japan. Edwin A. Azen's co-authors include Oliver Smithies, Linda M. Sabatini, Carter Denniston, Frank G. Oppenheim, T. Warner, Nobuo Maeda, Pao‐Lo Yu, Patricia A. Goodman, Eiichi Saitoh and Karen M. Lyons and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Edwin A. Azen

69 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Edwin A. Azen United States 28 950 927 298 284 210 69 1.9k
Ursula Bretz Japan 15 612 0.6× 209 0.2× 90 0.3× 119 0.4× 86 0.4× 24 1.8k
Takayuki Tsukuba Japan 31 1.3k 1.4× 286 0.3× 373 1.3× 87 0.3× 101 0.5× 107 2.5k
Eiko Sakai Japan 27 1.8k 1.9× 204 0.2× 364 1.2× 44 0.2× 89 0.4× 76 2.7k
Kazuo Sanada Japan 14 305 0.3× 450 0.5× 294 1.0× 69 0.2× 42 0.2× 29 920
Márta Széll Hungary 27 972 1.0× 182 0.2× 65 0.2× 130 0.5× 48 0.2× 125 2.4k
H S Shin United States 20 734 0.8× 187 0.2× 32 0.1× 69 0.2× 90 0.4× 30 2.0k
Alvin S. Zelickson United States 25 462 0.5× 161 0.2× 50 0.2× 25 0.1× 102 0.5× 52 1.8k
Vincenzo Mitolo Italy 25 594 0.6× 298 0.3× 28 0.1× 74 0.3× 29 0.1× 75 1.7k
J I Gallin United States 22 636 0.7× 264 0.3× 23 0.1× 64 0.2× 73 0.3× 25 2.0k
Kazufumi Katayama Japan 27 1.4k 1.5× 219 0.2× 82 0.3× 24 0.1× 79 0.4× 64 2.3k

Countries citing papers authored by Edwin A. Azen

Since Specialization
Citations

This map shows the geographic impact of Edwin A. Azen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edwin A. Azen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edwin A. Azen more than expected).

Fields of papers citing papers by Edwin A. Azen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edwin A. Azen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edwin A. Azen. The network helps show where Edwin A. Azen may publish in the future.

Co-authorship network of co-authors of Edwin A. Azen

This figure shows the co-authorship network connecting the top 25 collaborators of Edwin A. Azen. A scholar is included among the top collaborators of Edwin A. Azen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edwin A. Azen. Edwin A. Azen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
3.
Sabatini, Linda M. & Edwin A. Azen. (1994). Two coding change mutations in theHIS22 allele characterize the salivary histatin 3-2 protein variant. Human Mutation. 4(1). 12–19. 11 indexed citations
4.
Sabatini, Linda M., Tatsuya Ota, & Edwin A. Azen. (1993). Nucleotide sequence analysis of the human salivary protein genes HIS1 and HIS2, and evolution of the STATH/HIS gene family.. Molecular Biology and Evolution. 10(3). 497–511. 38 indexed citations
5.
Azen, Edwin A.. (1993). Genetics of Salivary Protein Polymorphisms. Critical Reviews in Oral Biology & Medicine. 4(3). 479–485. 44 indexed citations
6.
Maggio, John C., et al.. (1992). Intermediate sucrose octa-acetate sensitivity suggests a third allele at mouse bitter taste locus Soa and Soa-Rua identity. Chemical Senses. 17(4). 391–401. 23 indexed citations
7.
Sabatini, Linda M., et al.. (1990). Structure and sequence determination of the gene encoding human salivary statherin. Gene. 89(2). 245–251. 13 indexed citations
8.
Sabatini, Linda M., T. Warner, Eiichi Saitoh, & Edwin A. Azen. (1989). Tissue Distribution of RNAs for Cystatins, Histatins, Statherin, and Proline-rich Salivary Proteins in Humans and Macaques. Journal of Dental Research. 68(7). 1138–1145. 63 indexed citations
9.
Saitoh, Eiichi, Linda M. Sabatini, Roger L. Eddy, et al.. (1989). The human cystatin C gene (CST3) is a member of the cystatin gene family which is localized on chromosome 20. Biochemical and Biophysical Research Communications. 162(3). 1324–1331. 41 indexed citations
10.
O’Hanlon, Karen, Karen Weissbecker, Victoria K. Cortessis, M. Anne Spence, & Edwin A. Azen. (1988). Genes for salivary proline-rich proteins and taste for phenylthiourea are not closely linked in humans. Cytogenetic and Genome Research. 49(4). 315–317. 4 indexed citations
11.
Azen, Edwin A., Hyungsuk Kim, Patricia Goodman, Shannon Flynn, & Nobuo Maeda. (1987). Alleles at the PRH1 locus coding for the human salivary-acidic proline-rich proteins Pa, Db, and PIF.. PubMed. 41(6). 1035–47. 24 indexed citations
12.
Sabatini, Linda M., et al.. (1987). cDNA cloning and chromosomal localization (4q11-13) of a gene for statherin, a regulator of calcium in saliva.. PubMed. 41(6). 1048–60. 25 indexed citations
13.
Yu, Pao‐Lo, David Bixler, Patricia A. Goodman, et al.. (1986). Human parotid proline‐rich proteins: Correlation of genetic polymorphisms to dental caries. Genetic Epidemiology. 3(3). 147–152. 24 indexed citations
14.
Azen, Edwin A., Robert C. Karn, & Richard M. Pauli. (1984). A human lymphoblastoid cell line from the offspring of a brother-sister mating. Cytogenetic and Genome Research. 38(3). 238–239. 11 indexed citations
15.
Friedman, Richard D., et al.. (1980). Heritable Salivary Proteins and Dental Disease. Human Heredity. 30(6). 372–375. 13 indexed citations
16.
Azen, Edwin A., Carolyn Katovich Hurley, & Carter Denniston. (1979). Genetic polymorphism of the major parotid salivary glycoprotein (Gl) with linkage to the genes for Pr, Db, and Pa. Biochemical Genetics. 17(3-4). 257–279. 39 indexed citations
17.
Azen, Edwin A.. (1978). Genetic protein polymrophisms in human saliva: An interpretive review. Biochemical Genetics. 16(1-2). 79–99. 48 indexed citations
18.
Azen, Edwin A.. (1978). Salivary peroxidase activity and thiocyanate concentration in human subjects with genetic variants of salivary peroxidase. Archives of Oral Biology. 23(9). 801–805. 17 indexed citations
19.
Goodfriend, Theodore L., et al.. (1976). Peptide-binding macromolecules in the blood of seriously ill or mentally retarded patients.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 87(2). 299–319. 2 indexed citations
20.
Azen, Edwin A.. (1964). Prolonged Survival in Goodpasture's Syndrome. Archives of Internal Medicine. 114(3). 453–453. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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