Dawn Zhu

1.9k total citations
10 papers, 1.1k citations indexed

About

Dawn Zhu is a scholar working on Cellular and Molecular Neuroscience, Sensory Systems and Physiology. According to data from OpenAlex, Dawn Zhu has authored 10 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cellular and Molecular Neuroscience, 6 papers in Sensory Systems and 5 papers in Physiology. Recurrent topics in Dawn Zhu's work include Ion Channels and Receptors (6 papers), Neuropeptides and Animal Physiology (5 papers) and Pain Mechanisms and Treatments (4 papers). Dawn Zhu is often cited by papers focused on Ion Channels and Receptors (6 papers), Neuropeptides and Animal Physiology (5 papers) and Pain Mechanisms and Treatments (4 papers). Dawn Zhu collaborates with scholars based in United States, United Kingdom and New Zealand. Dawn Zhu's co-authors include Sonya G. Lehto, Narender R. Gavva, Kenneth D. Wild, Anthony W. Bannon, David Immke, Lana Klionsky, Mark H. Norman, Rami Tamir, James Treanor and Jean‐Claude Louis and has published in prestigious journals such as Journal of Neuroscience, Pain and Journal of Medicinal Chemistry.

In The Last Decade

Dawn Zhu

10 papers receiving 1.1k citations

Peers

Dawn Zhu
Dawn Zhu
Citations per year, relative to Dawn Zhu Dawn Zhu (= 1×) peers Péter Sántha

Countries citing papers authored by Dawn Zhu

Since Specialization
Citations

This map shows the geographic impact of Dawn Zhu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dawn Zhu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dawn Zhu more than expected).

Fields of papers citing papers by Dawn Zhu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dawn Zhu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dawn Zhu. The network helps show where Dawn Zhu may publish in the future.

Co-authorship network of co-authors of Dawn Zhu

This figure shows the co-authorship network connecting the top 25 collaborators of Dawn Zhu. A scholar is included among the top collaborators of Dawn Zhu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dawn Zhu. Dawn Zhu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Hu, Essa, Fang‐Tsao Hong, Derek E. Piper, et al.. (2021). Discovery of Selective Pituitary Adenylate Cyclase 1 Receptor (PAC1R) Antagonist Peptides Potent in a Maxadilan/PACAP38-Induced Increase in Blood Flow Pharmacodynamic Model. Journal of Medicinal Chemistry. 64(6). 3427–3438. 6 indexed citations
2.
Hoffmann, Jan, Silke Miller, Margarida Martins-Oliveira, et al.. (2020). PAC1 receptor blockade reduces central nociceptive activity: new approach for primary headache?. Pain. 161(7). 1670–1681. 51 indexed citations
3.
Garami, András, Eszter Pákai, Heath A. McDonald, et al.. (2018). TRPV1 antagonists that cause hypothermia, instead of hyperthermia, in rodents: Compounds’ pharmacological profiles, in vivo targets, thermoeffectors recruited and implications for drug development. Acta Physiologica. 223(3). e13038–e13038. 68 indexed citations
4.
Shi, Lei, Sonya G. Lehto, Dawn Zhu, et al.. (2015). Pharmacologic Characterization of AMG 334, a Potent and Selective Human Monoclonal Antibody against the Calcitonin Gene-Related Peptide Receptor. Journal of Pharmacology and Experimental Therapeutics. 356(1). 223–231. 180 indexed citations
5.
Rao, Sara, Hong Sun, Dawn Zhu, et al.. (2014). EHMTI-0315. AMG 334, the first potent and selective human monoclonal antibody antagonist against the CGRP receptor. The Journal of Headache and Pain. 15(S1). 8 indexed citations
6.
Gavva, Narender R., Carl D. Davis, Sonya G. Lehto, et al.. (2012). Transient Receptor Potential Melastatin 8 (TRPM8) Channels are Involved in Body Temperature Regulation. Molecular Pain. 8. 36–36. 82 indexed citations
7.
Wild, Kenneth D., Di Bian, Dawn Zhu, et al.. (2007). Antibodies to Nerve Growth Factor Reverse Established Tactile Allodynia in Rodent Models of Neuropathic Pain without Tolerance. Journal of Pharmacology and Experimental Therapeutics. 322(1). 282–287. 90 indexed citations
8.
Gavva, Narender R., Anthony W. Bannon, Sekhar Surapaneni, et al.. (2007). The Vanilloid Receptor TRPV1 Is Tonically Activated In Vivo and Involved in Body Temperature Regulation. Journal of Neuroscience. 27(13). 3366–3374. 258 indexed citations
9.
Ognyanov, Vassil I., Balan Chenera, Anthony W. Bannon, et al.. (2006). Design of Potent, Orally Available Antagonists of the Transient Receptor Potential Vanilloid 1. Structure−Activity Relationships of 2-Piperazin-1-yl-1H-benzimidazoles. Journal of Medicinal Chemistry. 49(12). 3719–3742. 70 indexed citations
10.
Gavva, Narender R., Rami Tamir, Yusheng Qu, et al.. (2005). AMG 9810 [(E)-3-(4-t-Butylphenyl)-N-(2,3-dihydrobenzo[b][1,4] dioxin-6-yl)acrylamide], a Novel Vanilloid Receptor 1 (TRPV1) Antagonist with Antihyperalgesic Properties. Journal of Pharmacology and Experimental Therapeutics. 313(1). 474–484. 330 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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