Péter Sántha

1.7k total citations
55 papers, 1.3k citations indexed

About

Péter Sántha is a scholar working on Physiology, Sensory Systems and Cellular and Molecular Neuroscience. According to data from OpenAlex, Péter Sántha has authored 55 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Physiology, 26 papers in Sensory Systems and 23 papers in Cellular and Molecular Neuroscience. Recurrent topics in Péter Sántha's work include Pain Mechanisms and Treatments (30 papers), Ion Channels and Receptors (25 papers) and Neuropeptides and Animal Physiology (19 papers). Péter Sántha is often cited by papers focused on Pain Mechanisms and Treatments (30 papers), Ion Channels and Receptors (25 papers) and Neuropeptides and Animal Physiology (19 papers). Péter Sántha collaborates with scholars based in Hungary, United Kingdom and Germany. Péter Sántha's co-authors include Gábor Jancsó, I. Nagy, Mária Dux, László Urbán, Arpan Tahim, Ildikó Dobos, António Avelino, Klára Matesz, Ana Charrua and Dorte X. Gram and has published in prestigious journals such as Journal of Neuroscience, The Journal of Physiology and The Journal of Comparative Neurology.

In The Last Decade

Péter Sántha

53 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Péter Sántha Hungary 19 599 527 343 280 232 55 1.3k
Krisztián Elekes Hungary 22 607 1.0× 551 1.0× 488 1.4× 311 1.1× 108 0.5× 27 1.3k
Kata Bölcskei Hungary 28 915 1.5× 694 1.3× 567 1.7× 430 1.5× 220 0.9× 64 2.0k
Donna M. Gauvin United States 17 677 1.1× 406 0.8× 424 1.2× 418 1.5× 165 0.7× 27 1.7k
António Avelino Portugal 27 598 1.0× 631 1.2× 280 0.8× 170 0.6× 222 1.0× 48 1.6k
Tomoko Moriyama Japan 13 664 1.1× 593 1.1× 367 1.1× 313 1.1× 134 0.6× 14 1.3k
Susanne K. Sauer Germany 26 823 1.4× 543 1.0× 445 1.3× 504 1.8× 119 0.5× 46 1.8k
Selena Harrison Italy 16 636 1.1× 586 1.1× 288 0.8× 229 0.8× 197 0.8× 19 1.3k
Chengmin Zhong United States 17 977 1.6× 407 0.8× 455 1.3× 384 1.4× 208 0.9× 17 1.6k
Gábor Pozsgai Hungary 20 377 0.6× 476 0.9× 407 1.2× 233 0.8× 59 0.3× 37 1.2k
Vittorio Vellani Italy 15 659 1.1× 732 1.4× 652 1.9× 399 1.4× 474 2.0× 28 1.7k

Countries citing papers authored by Péter Sántha

Since Specialization
Citations

This map shows the geographic impact of Péter Sántha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Péter Sántha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Péter Sántha more than expected).

Fields of papers citing papers by Péter Sántha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Péter Sántha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Péter Sántha. The network helps show where Péter Sántha may publish in the future.

Co-authorship network of co-authors of Péter Sántha

This figure shows the co-authorship network connecting the top 25 collaborators of Péter Sántha. A scholar is included among the top collaborators of Péter Sántha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Péter Sántha. Péter Sántha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tóth, Máté, et al.. (2022). Insulin sensitizes neural and vascular TRPV1 receptors in the trigeminovascular system. The Journal of Headache and Pain. 23(1). 7–7. 11 indexed citations
2.
Jancsó, Gábor, et al.. (2018). The insulin receptor is differentially expressed in somatic and visceral primary sensory neurons. Cell and Tissue Research. 374(2). 243–249. 3 indexed citations
3.
Jancsó, Gábor & Péter Sántha. (2015). The foundation of sensory pharmacology: Nicholas (Miklós) Jancsó and the Szeged contribution. Temperature. 2(2). 152–157. 12 indexed citations
4.
5.
Horvath, Andrea R., Péter Sántha, Nóra Török, et al.. (2013). Rapid genotyping of genetically modified laboratory animals from whole blood samples without DNA preparation. Acta Biologica Hungarica. 64(2). 262–265.
6.
White, John P., Péter Sántha, John B. Davis, et al.. (2010). Xenon reduces activation of transient receptor potential vanilloid type 1 (TRPV1) in rat dorsal root ganglion cells and in human TRPV1-expressing HEK293 cells. Life Sciences. 88(3-4). 141–149. 5 indexed citations
7.
8.
Lever, Isobel J., Michelle Robinson, Mario Cibelli, et al.. (2009). Localization of the Endocannabinoid-Degrading Enzyme Fatty Acid Amide Hydrolase in Rat Dorsal Root Ganglion Cells and Its Regulation after Peripheral Nerve Injury. Journal of Neuroscience. 29(12). 3766–3780. 50 indexed citations
9.
10.
Gram, Dorte X., Bo Åhrén, I. Nagy, et al.. (2007). Capsaicin‐sensitive sensory fibers in the islets of Langerhans contribute to defective insulin secretion in Zucker diabetic rat, an animal model for some aspects of human type 2 diabetes. European Journal of Neuroscience. 25(1). 213–223. 116 indexed citations
11.
Dux, Mária, et al.. (2007). Loss of capsaicin-induced meningeal neurogenic sensory vasodilatation in diabetic rats. Neuroscience. 150(1). 194–201. 17 indexed citations
12.
Sántha, Péter, António Avelino, Ana Charrua, et al.. (2007). Neurochemical characterization of insulin receptor‐expressing primary sensory neurons in wild‐type and vanilloid type 1 transient receptor potential receptor knockout mice. The Journal of Comparative Neurology. 503(2). 334–347. 35 indexed citations
13.
Sántha, Péter, et al.. (2003). Effects of tacrolimus on ischemia-reperfusion injury. Liver Transplantation. 9(2). 105–116. 31 indexed citations
14.
Dux, Mária, Péter Sántha, & Gábor Jancsó. (2003). Capsaicin‐sensitive neurogenic sensory vasodilatation in the dura mater of the rat. The Journal of Physiology. 552(3). 859–867. 105 indexed citations
15.
Avelino, António, Ana Charrua, Péter Sántha, et al.. (2003). Insulin induces cobalt uptake in a subpopulation of rat cultured primary sensory neurons. European Journal of Neuroscience. 18(9). 2477–2486. 38 indexed citations
16.
Domoki, Ferenc, Péter Sántha, Ferenc Bari, & Gábor Jancsó. (2003). Perineural capsaicin treatment attenuates reactive hyperaemia in the rat skin. Neuroscience Letters. 341(2). 127–130. 9 indexed citations
17.
Klukovits, Anna, Róbert Gáspár, Péter Sántha, Gábor Jancsó, & George Falkay. (2002). Functional and Histochemical Characterization of a Uterine Adrenergic Denervation Process in Pregnant Rats1. Biology of Reproduction. 67(3). 1013–1017. 33 indexed citations
18.
Sántha, Péter, et al.. (2000). Inhibitory neurogenic modulation of histamine-induced cutaneous plasma extravasation in the pigeon. Regulatory Peptides. 95(1-3). 75–80. 23 indexed citations
19.
Sántha, Péter, et al.. (1999). Inhibitory modulation of cutaneous vascular responses by endogenous galanin in the pigeon. Neuroscience Letters. 273(1). 64–66. 11 indexed citations
20.
Jancsó, Gábor, Ferenc Domoki, Péter Sántha, et al.. (1998). β-Amyloid (1–42) peptide impairs blood-brain barrier function after intracarotid infusion in rats. Neuroscience Letters. 253(2). 139–141. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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