David M. Stern

44.4k total citations · 15 hit papers
201 papers, 32.3k citations indexed

About

David M. Stern is a scholar working on Clinical Biochemistry, Molecular Biology and Physiology. According to data from OpenAlex, David M. Stern has authored 201 papers receiving a total of 32.3k indexed citations (citations by other indexed papers that have themselves been cited), including 69 papers in Clinical Biochemistry, 66 papers in Molecular Biology and 44 papers in Physiology. Recurrent topics in David M. Stern's work include Advanced Glycation End Products research (68 papers), Alzheimer's disease research and treatments (31 papers) and Neuroinflammation and Neurodegeneration Mechanisms (26 papers). David M. Stern is often cited by papers focused on Advanced Glycation End Products research (68 papers), Alzheimer's disease research and treatments (31 papers) and Neuroinflammation and Neurodegeneration Mechanisms (26 papers). David M. Stern collaborates with scholars based in United States, Japan and Germany. David M. Stern's co-authors include Ann Marie Schmidt, Shi Du Yan, Shi Fang Yan, Peter P. Nawroth, Yan Lü, Thomas Kislinger, Angelika Bierhaus, Akihiko Taguchi, Wu Qu and Jean‐Luc Wautier and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

David M. Stern

198 papers receiving 31.7k citations

Hit Papers

Endothelial cells in phys... 1995 2026 2005 2015 1998 1996 2000 2005 1995 500 1000 1.5k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Endothelial cells in physiology and in the pathophysiology of vascular disorders.
    1998 1.8k citations Ann Marie Schmidt, David M. Stern et al. profile →
  2. RAGE and amyloid-β peptide neurotoxicity in Alzheimer's disease
    1996 1.7k citations Shi Du Yan, Alex E. Roher et al. profile →
  3. Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases
    2000 1.1k citations Akihiko Taguchi, Wu Qu et al. profile →
  4. Understanding RAGE, the receptor for advanced glycation end products
    2005 1.0k citations Angelika Bierhaus, Michael Morcos et al. profile →
  5. The Receptor for Advanced Glycation End Products (RAGE) Is a Cellular Binding Site for Amphoterin
    1995 988 citations Osamu Hori, David M. Stern et al. profile →
  6. The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses
    2001 970 citations Ann Marie Schmidt, Shi Du Yan et al. Journal of Clinical Investigation profile →
  7. Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts
    1998 930 citations Yan Lü, David M. Stern et al. profile →
  8. The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses
    2001 895 citations Ann Marie Schmidt, Shi Du Yan et al. Journal of Clinical Investigation profile →
  9. Activation of NADPH oxidase by AGE links oxidant stress to altered gene expression via RAGE
    2001 867 citations David M. Stern, Ann Marie Schmidt et al. profile →
  10. N ε-(Carboxymethyl)Lysine Adducts of Proteins Are Ligands for Receptor for Advanced Glycation End Products That Activate Cell Signaling Pathways and Modulate Gene Expression
    1999 763 citations Thomas Kislinger, Wu Qu et al. profile →
  11. Activation of Receptor for Advanced Glycation End Products
    1999 644 citations Ann Marie Schmidt, Shi Du Yan et al. profile →
  12. Mitochondrial Aβ: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease
    2005 626 citations Alexander A. Sosunov, David M. Stern et al. The FASEB Journal profile →
  13. The biology of the receptor for advanced glycation end products and its ligands
    2000 587 citations Ann Marie Schmidt, Shi Du Yan et al. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research profile →
  14. Administration of CD34+ cells after stroke enhances neurogenesis via angiogenesisin a mouse model
    2004 566 citations Akihiko Taguchi, Toshihiro Soma et al. Journal of Clinical Investigation profile →
  15. Administration of CD34+ cells after stroke enhances neurogenesis via angiogenesisin a mouse model
    2004 516 citations Akihiko Taguchi, Toshihiro Soma et al. Journal of Clinical Investigation profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
David M. Stern 13.2k 10.6k 7.0k 5.3k 4.9k 201 32.3k
Peter P. Nawroth 11.1k 0.8× 9.8k 0.9× 6.4k 0.9× 5.1k 1.0× 6.5k 1.3× 494 36.3k
Angelika Bierhaus 10.1k 0.8× 6.2k 0.6× 3.3k 0.5× 4.1k 0.8× 4.5k 0.9× 247 22.7k
Ann Marie Schmidt 27.6k 2.1× 13.3k 1.3× 9.7k 1.4× 11.7k 2.2× 7.4k 1.5× 395 49.7k
Michael Brownlee 10.6k 0.8× 9.7k 0.9× 8.5k 1.2× 10.5k 2.0× 2.1k 0.4× 106 35.1k
Sho‐ichi Yamagishi 10.3k 0.8× 6.0k 0.6× 3.8k 0.5× 7.8k 1.5× 1.8k 0.4× 449 23.2k
Helen Vlassara 20.2k 1.5× 5.4k 0.5× 7.3k 1.0× 13.0k 2.5× 2.4k 0.5× 217 33.2k
George L. King 3.9k 0.3× 12.6k 1.2× 6.3k 0.9× 7.3k 1.4× 1.9k 0.4× 276 32.5k
Marco E. Bianchi 11.2k 0.9× 13.2k 1.2× 1.6k 0.2× 1.4k 0.3× 11.6k 2.3× 225 31.0k
D Stern 4.7k 0.4× 4.9k 0.5× 2.6k 0.4× 2.3k 0.4× 3.2k 0.6× 92 16.6k
J Brett 6.0k 0.5× 3.2k 0.3× 2.4k 0.3× 2.6k 0.5× 2.0k 0.4× 45 11.8k

Countries citing papers authored by David M. Stern

Since Specialization
Citations

This map shows the geographic impact of David M. Stern's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Stern with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Stern more than expected).

Fields of papers citing papers by David M. Stern

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Stern. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Stern. The network helps show where David M. Stern may publish in the future.

Co-authorship network of co-authors of David M. Stern

This figure shows the co-authorship network connecting the top 25 collaborators of David M. Stern. A scholar is included among the top collaborators of David M. Stern based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David M. Stern. David M. Stern is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Taguchi, Akihiko, Chiaki Sakai, Toshihiro Soma, et al.. (2015). Intravenous Autologous Bone Marrow Mononuclear Cell Transplantation for Stroke: Phase1/2a Clinical Trial in a Homogeneous Group of Stroke Patients. Stem Cells and Development. 24(19). 2207–2218. 84 indexed citations
2.
Kasahara, Yukiko, Masafumi Ihara, Takayuki Nakagomi, et al.. (2013). A highly reproducible model of cerebral ischemia/reperfusion with extended survival in CB-17 mice. Neuroscience Research. 76(3). 163–168. 14 indexed citations
3.
Schlotterer, Andreas, Andreas Hamann, G Kukudov, et al.. (2010). Apurinic/apyrimidinic endonuclease 1, p53, and thioredoxin are linked in control of aging inC. elegans. Aging Cell. 9(3). 420–432. 22 indexed citations
4.
Takuma, Kazuhiro, Fang Fang, Wensheng Zhang, et al.. (2009). RAGE-mediated signaling contributes to intraneuronal transport of amyloid-β and neuronal dysfunction. Proceedings of the National Academy of Sciences. 106(47). 20021–20026. 250 indexed citations
5.
Fang, Fang, Lih‐Fen Lue, Shiqiang Yan, et al.. (2009). RAGE‐dependent signaling in microglia contributes to neuroinflammation, Aβ accumulation, and impaired learning/memory in a mouse model of Alzheimer's disease. The FASEB Journal. 24(4). 1043–1055. 274 indexed citations
6.
Eubank, Timothy D., Sainath R. Kotha, M. Lakshmi Kuppusamy, et al.. (2009). Hyperglycemic oxoaldehyde, glyoxal, causes barrier dysfunction, cytoskeletal alterations, and inhibition of angiogenesis in vascular endothelial cells: aminoguanidine protection. Molecular and Cellular Biochemistry. 333(1-2). 9–26. 33 indexed citations
7.
Abeyama, Kazuhiro, David M. Stern, Yuji Ito, et al.. (2005). The N-terminal domain of thrombomodulin sequesters high-mobility group-B1 protein, a novel antiinflammatory mechanism. Journal of Clinical Investigation. 115(5). 1267–1274. 437 indexed citations
8.
Abeyama, Kazuhiro, David M. Stern, Yuji Ito, et al.. (2005). The N-terminal domain of thrombomodulin sequesters high-mobility group-B1 protein, a novel antiinflammatory mechanism. Journal of Clinical Investigation. 115(5). 1267–1274. 46 indexed citations
9.
Tawfik, Amany, Liming Jin, Amy Banes‐Berceli, et al.. (2005). Hyperglycemia and reactive oxygen species mediate apoptosis in aortic endothelial cells through Janus kinase 2. Vascular Pharmacology. 43(5). 320–326. 33 indexed citations
10.
Chaney, Michael O., W. Blaine Stine, Tyler A. Kokjohn, et al.. (2005). RAGE and amyloid beta interactions: Atomic force microscopy and molecular modeling. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1741(1-2). 199–205. 81 indexed citations
11.
Xiong, Wen‐Cheng & David M. Stern. (2005). The marriage of glucose and blood vessels: It isn’t all that sweet. Cell Metabolism. 2(4). 212–215. 1 indexed citations
12.
Nawroth, Peter P., Angelika Bierhaus, Mario B. Marrero, Hiroshi Yamamoto, & David M. Stern. (2005). Atherosclerosis and restenosis: Is there a role for rage?. Current Diabetes Reports. 5(1). 11–16. 26 indexed citations
13.
Taguchi, Akihiko, Toshihiro Soma, Hidekazu Tanaka, et al.. (2004). Administration of CD34+ cells after stroke enhances neurogenesis via angiogenesisin a mouse model. Journal of Clinical Investigation. 114(3). 330–338. 566 indexed citations breakdown →
14.
Chen, Yali, Shirley ShiDu Yan, John Colgan, et al.. (2004). Blockade of Late Stages of Autoimmune Diabetes by Inhibition of the Receptor for Advanced Glycation End Products. The Journal of Immunology. 173(2). 1399–1405. 109 indexed citations
15.
Wendt, Thoralf, Nozomu Tanji, Jiancheng Guo, et al.. (2003). RAGE Drives the Development of Glomerulosclerosis and Implicates Podocyte Activation in the Pathogenesis of Diabetic Nephropathy. American Journal Of Pathology. 162(4). 1123–1137. 484 indexed citations
16.
Hudson, Barry I., M. Hofmann, Loredana Bucciarelli, et al.. (2002). Glycation and diabetes: The RAGE connection. Current Science. 83(12). 1515–1521. 41 indexed citations
17.
Krieger, Monty & David M. Stern. (2001). Series Introduction: Multiligand receptors and human disease. Journal of Clinical Investigation. 108(5). 645–647. 43 indexed citations
18.
Yan, Shi Du, Alex E. Roher, Michael P. Chaney, et al.. (2000). Cellular cofactors potentiating induction of stress and cytotoxicity by amyloid β-peptide. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1502(1). 145–157. 50 indexed citations
19.
Schmidt, Ann Marie, Shi Du Yan, Shi Fang Yan, & David M. Stern. (2000). The biology of the receptor for advanced glycation end products and its ligands. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1498(2-3). 99–111. 587 indexed citations breakdown →
20.
Sheikh, Sajila, Ranjit S. Parhar, Aaron Kwaasi, et al.. (2000). ALPHA-GAL-INDEPENDENT DUAL RECOGNITION AND ACTIVATION OF XENOGENEIC ENDOTHELIAL CELLS AND HUMAN NA??VE NATURAL KILLER CELLS1. Transplantation. 70(6). 917–928. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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