Satoshi Ogawa
- Molecular Biology top 2%
- Plant Science top 1%
- Cell Biology top 0.5%
- Clinical Biochemistry top 0.1%
- Epidemiology top 2%
- Co-authors
- Osamu HoriDavid Julian McClementsEric A. DeckerDavid M. SternKentaro OzawaYasuko KitaoMasaya TohyamaD Stern
- Topics
- Endoplasmic Reticulum Stress and Disease (31 papers)Autophagy in Disease and Therapy (17 papers)Plant Parasitism and Resistance (10 papers)
- Partner nations
- JapanUnited StatesColombia
In The Last Decade
Satoshi Ogawa
121 papers receiving 8.5k citations
Hit Papers
Peers
Comparison fields: 5 of 160
- Molecular Biology 3.3k
- Plant Science 2.1k
- Cell Biology 1.9k
- Clinical Biochemistry 1.3k
- Epidemiology 1.1k
Countries citing papers authored by Satoshi Ogawa
This map shows the geographic impact of Satoshi Ogawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satoshi Ogawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satoshi Ogawa more than expected).
Fields of papers citing papers by Satoshi Ogawa
This network shows the impact of papers produced by Satoshi Ogawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satoshi Ogawa. The network helps show where Satoshi Ogawa may publish in the future.
Co-authorship network of co-authors of Satoshi Ogawa
This figure shows the co-authorship network connecting the top 25 collaborators of Satoshi Ogawa. A scholar is included among the top collaborators of Satoshi Ogawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Satoshi Ogawa. Satoshi Ogawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 16 | |
| 4 | 9 | |
| 5 | 26 | |
| 6 | 1 | |
| 7 | 35 | |
| 8 | 27 | |
| 9 | 1 | |
| 10 | 17 | |
| 11 | 2 | |
| 12 | 7 | |
| 13 | 22 | |
| 14 | 171 | |
| 15 | 47 | |
| 16 | 7 | |
| 17 | 26 | |
| 18 | Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastasesbreakdown → | 1058 |
| 19 | 86 | |
| 20 | Nonlinear Modeling by Radial Basis Function Networks (Special Section on Nonlinear Theory and its Applications) | 5 |
About Satoshi Ogawa
Satoshi Ogawa is a scholar working on Cell Biology, Clinical Biochemistry and Physiology, having authored 125 papers that have together received 8.7k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (31 papers), Autophagy in Disease and Therapy (17 papers) and Plant Parasitism and Resistance (10 papers). The work is most often cited by research in Clinical Biochemistry (1.3k citations), Cell Biology (1.9k citations) and Plant Science (2.1k citations). Satoshi Ogawa has collaborated with scholars based in Japan, United States and Colombia. Frequent co-authors include Osamu Hori, David Julian McClements, Eric A. Decker, David M. Stern, Kentaro Ozawa, Yasuko Kitao, Masaya Tohyama, D Stern, J Brett and Manabu Ishitani. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.