David H. Spencer

12.2k total citations · 1 hit paper
57 papers, 3.8k citations indexed

About

David H. Spencer is a scholar working on Molecular Biology, Hematology and Cancer Research. According to data from OpenAlex, David H. Spencer has authored 57 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 20 papers in Hematology and 19 papers in Cancer Research. Recurrent topics in David H. Spencer's work include Acute Myeloid Leukemia Research (19 papers), Cancer Genomics and Diagnostics (15 papers) and Epigenetics and DNA Methylation (11 papers). David H. Spencer is often cited by papers focused on Acute Myeloid Leukemia Research (19 papers), Cancer Genomics and Diagnostics (15 papers) and Epigenetics and DNA Methylation (11 papers). David H. Spencer collaborates with scholars based in United States, Canada and United Kingdom. David H. Spencer's co-authors include Maynard V. Olson, Rajinder Kaul, Christopher K. Raymond, Eric Haugen, Chunrong Liu, Ruth Levy, Michael A. Jacobs, Stephen A. Ernst, Iyarit Thaipisuttikul and Colin Manoil and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and JAMA.

In The Last Decade

David H. Spencer

56 papers receiving 3.7k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Comprehensive transposon mutant library of Pseudomonas aeruginosa

2003 932 citations David H. Spencer, Maynard V. Olson et al. profile →

Peers

Countries citing papers authored by David H. Spencer

Since Specialization

Citations

This map shows the geographic impact of David H. Spencer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David H. Spencer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David H. Spencer more than expected).

Fields of papers citing papers by David H. Spencer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David H. Spencer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David H. Spencer. The network helps show where David H. Spencer may publish in the future.

Co-authorship network of co-authors of David H. Spencer

This figure shows the co-authorship network connecting the top 25 collaborators of David H. Spencer. A scholar is included among the top collaborators of David H. Spencer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David H. Spencer. David H. Spencer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Innovations in Short-Read Sequencing Technologies and Their Applications to Clinical Genomics	Clinical Chemistry	Katarzyna Polonis, Joseph H. Blommel et al.	5
2	A fully next-generation sequencing-based method of classifying molecular sub-types of endometrial cancer retains prognostic value and expands biomarker targets	International Journal of Gynecological Cancer	Mark C. Valentine, Amanda Wong et al.	0
3	Missense Mutations in Myc Box I Influence Nucleocytoplasmic Transport to Promote Leukemogenesis	Clinical Cancer Research	Marianna B. Ruzinova, Ashutosh Kumar et al.	2
4	Genomic landscape of TP53-mutated myeloid malignancies	Blood Advances	Haley Abel, Karolyn A. Oetjen et al.	15
5	KDM6B protects T-ALL cells from NOTCH1-induced oncogenic stress	Leukemia	Christine Zhang, Wentao Han et al.	9
6	Clinical whole‐genome sequencing in cancer diagnosis	Human Mutation	Ying‐Chen Claire Hou, Julie Neidich et al.	13
7	Failure to Detect Mutations in U2AF1 due to Changes in the GRCh38 Reference Sequence	Journal of Molecular Diagnostics	Christopher A. Miller, Jason Walker et al.	14
8	Heterozygous variants of CLPB are a cause of severe congenital neutropenia	Blood	Julia T. Warren, Ryan R. Cupo et al.	24
9	Focal disruption of DNA methylation dynamics at enhancers in IDH-mutant AML cells	Leukemia	Nichole Helton, Sharon E. Heath et al.	19
10	Tumor suppressor function of <I>WT1</I> in acute promyelocytic leukemia	Haematologica	Matthew Christopher, Margery Gang et al.	6
11	Contribution of CTCF binding to transcriptional activity at the HOXA locus in NPM1-mutant AML cells	Leukemia	Reza Ghasemi, David H. Spencer et al.	18
12	CpG Island Hypermethylation Mediated by DNMT3A Is a Consequence of AML Progression	Cell	David H. Spencer, David A. Russler‐Germain et al.	149
13	mlo ‐based powdery mildew resistance in hexaploid bread wheat generated by a non‐transgenic TILLING approach	Plant Biotechnology Journal	Johanna Acevedo‐Garcia, David H. Spencer et al.	134
14	The R882H DNMT3A Mutation Associated with AML Dominantly Inhibits Wild-Type DNMT3A by Blocking Its Ability to Form Active Tetramers	Cancer Cell	David A. Russler‐Germain, David H. Spencer et al.	316
15	Functional Heterogeneity of Genetically Defined Subclones in Acute Myeloid Leukemia	Cancer Cell	Jeffery M. Klco, David H. Spencer et al.	264
16	Comparison of Clinical Targeted Next-Generation Sequence Data from Formalin-Fixed and Fresh-Frozen Tissue Specimens	Journal of Molecular Diagnostics	David H. Spencer, Jennifer K. Sehn et al.	123
17	Performance of Common Analysis Methods for Detecting Low-Frequency Single Nucleotide Variants in Targeted Next-Generation Sequence Data	Journal of Molecular Diagnostics	David H. Spencer, Manoj Tyagi et al.	84
18	Growth phenotypes of Pseudomonas aeruginosa lasR mutants adapted to the airways of cystic fibrosis patients	Molecular Microbiology	David A. D’Argenio, Manhong Wu et al.	278
19	Detecting Disease-Causing Mutations in the Human Genome by Haplotype Matching	The American Journal of Human Genetics	David H. Spencer, Kerry L. Bubb et al.	3
20	A field study of use of synchronous computer-mediated communication in asynchronous learning networks		David H. Spencer, Starr Roxanne Hiltz	6

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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