Matthew J. Walter

28.1k total citations · 2 hit papers
107 papers, 7.6k citations indexed

About

Matthew J. Walter is a scholar working on Hematology, Molecular Biology and Cancer Research. According to data from OpenAlex, Matthew J. Walter has authored 107 papers receiving a total of 7.6k indexed citations (citations by other indexed papers that have themselves been cited), including 72 papers in Hematology, 60 papers in Molecular Biology and 24 papers in Cancer Research. Recurrent topics in Matthew J. Walter's work include Acute Myeloid Leukemia Research (68 papers), RNA Research and Splicing (20 papers) and Cancer Genomics and Diagnostics (17 papers). Matthew J. Walter is often cited by papers focused on Acute Myeloid Leukemia Research (68 papers), RNA Research and Splicing (20 papers) and Cancer Genomics and Diagnostics (17 papers). Matthew J. Walter collaborates with scholars based in United States, United Kingdom and Germany. Matthew J. Walter's co-authors include Timothy J. Ley, Richard K. Wilson, Li Ding, John S. Welch, Christopher A. Miller, Michael D. McLellan, Michael C. Wendl, Joshua F. McMichael, Charles Lu and Mingchao Xie and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Matthew J. Walter

101 papers receiving 7.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mutational landscape and significance across 12 major cancer types

2013 3.1k citations Christopher A. Miller, John S. Welch et al. profile →
Age-related mutations associated with clonal hematopoietic expansion and malignancies

2014 1.2k citations Christopher A. Miller, John S. Welch et al. profile →

Peers

Matthew J. Walter

HEMAT

ONCOL

GENET

Alexander Kohlmann Germany

Joshua F. McMichael United States

Alexei Protopopov United States

Charles Lu United States

Cyrus V. Hedvat United States

Stefan Fröhling Germany

Mingchao Xie United States

Edward A. Fox United States

Sakari Knuutila Finland

Peter J.M. Valk Netherlands

Alexander Kohlmann Germany

Matthew J. Walter

3.8k ×0.8

3.6k ×1.4

HEMAT

1.4k ×0.6

1.7k ×1.0

ONCOL

1.8k ×1.6

GENET

Citations per year, relative to Matthew J. Walter Matthew J. Walter (= 1×) peers Alexander Kohlmann

Countries citing papers authored by Matthew J. Walter

Since Specialization

Citations

This map shows the geographic impact of Matthew J. Walter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Walter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Walter more than expected).

Fields of papers citing papers by Matthew J. Walter

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Walter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Walter. The network helps show where Matthew J. Walter may publish in the future.

Co-authorship network of co-authors of Matthew J. Walter

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Walter. A scholar is included among the top collaborators of Matthew J. Walter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Walter. Matthew J. Walter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

DeZern, Amy E., Michael Otterstatter, Nancy Gillis, et al.. (2024). Progression of Myelodysplastic Syndromes in the MDS Natural History Study (MDS NHS). Blood. 144(Supplement 1). 1831–1831.

Chung, Yang Jo, Michael J. Difilippantonio, Yuelin J. Zhu, et al.. (2024). The DNA Methyltransferase Inhibitor 5-Aza-4′-thio-2′-Deoxycytidine Induces C>G Transversions and Acute Lymphoid Leukemia Development. Cancer Research. 84(15). 2518–2532. 3 indexed citations

Tran, Duc, J. Scott Beeler, Jie Liu, et al.. (2024). Plasma Proteomic Signature Predicts Myeloid Neoplasm Risk. Clinical Cancer Research. 30(15). 3220–3228. 3 indexed citations

Butler, Christopher, Jaebok Choi, Gábor Szalai, et al.. (2024). HSPA9/mortalin inhibition disrupts erythroid maturation through a TP53-dependent mechanism in human CD34+ hematopoietic progenitor cells. Cell Stress and Chaperones. 29(2). 300–311. 3 indexed citations

Abel, Haley, Karolyn A. Oetjen, Christopher A. Miller, et al.. (2023). Genomic landscape of TP53-mutated myeloid malignancies. Blood Advances. 7(16). 4586–4598. 15 indexed citations

Ahmadov, Ulvi, Thomas B. Hansen, Zongliang Gao, et al.. (2023). Impact of U2AF1 mutations on circular RNA expression in myelodysplastic neoplasms. Leukemia. 37(5). 1113–1125. 7 indexed citations

Choi, Jaebok, Matthew Cooper, Mark A. Fiala, et al.. (2023). Phase I-II Trial of Early Azacitidine after Matched Unrelated Donor Hematopoietic Cell Transplantation. Transplantation and Cellular Therapy. 29(11). 699.e1–699.e9. 2 indexed citations

Miller, Christopher A., Jason Walker, Robert S. Fulton, et al.. (2022). Failure to Detect Mutations in U2AF1 due to Changes in the GRCh38 Reference Sequence. Journal of Molecular Diagnostics. 24(3). 219–223. 14 indexed citations

Li, Shan, Sridhar Nonavinkere Srivatsan, Yuhao Chen, et al.. (2021). Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations. Cancer Research. 81(17). 4499–4513. 37 indexed citations

10.

Jacoby, Meagan A., David A. Sallman, Bart L. Scott, et al.. (2021). A Pilot Study of CPX-351 (Vyxeos ©) for Transplant Eligible, Higher Risk Patients with Myelodysplastic Syndrome. Blood. 138(Supplement 1). 540–540. 6 indexed citations

11.

Brunner, Andrew M., Yiwen Liu, Lourdes Mendez, et al.. (2021). Inhibition of ATR with AZD6738 (Ceralasertib) for the Treatment of Progressive or Relapsed Myelodysplastic Syndromes and Chronic Myelomonocytic Leukemia: Safety and Preliminary Activity from a Phase Ib/II Study. Blood. 138(Supplement 1). 1521–1521. 6 indexed citations

12.

Helton, Nichole, Sharon E. Heath, Robert S. Fulton, et al.. (2021). Focal disruption of DNA methylation dynamics at enhancers in IDH-mutant AML cells. Leukemia. 36(4). 935–945. 19 indexed citations

13.

Zhang, Ling, Donald M. Stablein, Pearlie K. Epling‐Burnette, et al.. (2018). Diagnosis of Myelodysplastic Syndromes and Related Conditions: Rates of Discordance between Local and Central Review in the NHLBI MDS Natural History Study. Blood. 132(Supplement 1). 4370–4370. 3 indexed citations

14.

Nguyen, Hai Dang, Wan Yee Leong, Weiling Li, et al.. (2018). Spliceosome Mutations Induce R Loop-Associated Sensitivity to ATR Inhibition in Myelodysplastic Syndromes. Cancer Research. 78(18). 5363–5374. 115 indexed citations

15.

Jacoby, Meagan A., Eric J. Duncavage, Gue Su Chang, et al.. (2017). Subclonal Evolution Characterizes MDS Disease Progression Following Allogeneic Stem Cell Transplant. Blood. 130. 4232. 1 indexed citations

16.

Leong, Wan Yee, Hai Dang Nguyen, Weiling Li, et al.. (2017). Spliceosomal Mutations Induce R Loop-Associated ATR Signaling. Blood. 130. 116–116. 1 indexed citations

17.

Miller, Christopher A., Brian S. White, Nathan D. Dees, et al.. (2014). SciClone: Inferring Clonal Architecture and Tracking the Spatial and Temporal Patterns of Tumor Evolution. PLoS Computational Biology. 10(8). e1003665–e1003665. 281 indexed citations

18.

Payton, Jacqueline E., et al.. (2010). POU4F1 is associated with t(8;21) acute myeloid leukemia and contributes directly to its unique transcriptional signature. Leukemia. 24(5). 950–957. 15 indexed citations

19.

Martin, Mike G., John S. Welch, Jingqin Luo, et al.. (2009). Therapy related acute myeloid leukemia in breast cancer survivors, a population-based study. Breast Cancer Research and Treatment. 118(3). 593–598. 36 indexed citations

20.

Keller, U, et al.. (1978). Phaeochromocytoma with lactic acidosis.. BMJ. 2(6137). 606–607. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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