Hila Zigdon

791 citations
8 papers · 604 · h-index 8

Impact in

Papers in

    • Sphingolipid Metabolism and Signaling 3
    • Glycosylation and Glycoproteins Research 2
    • Congenital heart defects research 1
    • Lipid Membrane Structure and Behavior 1
    • Lysosomal Storage Disorders Research 5

Hila Zigdon

8 papers receiving 596 citations

Peers

Hila Zigdon
Comparison fields: 5 of 73
  • Physiology 44
  • Physiology 215
  • Aging 14
  • Cell Biology 125
  • Molecular Biology 411
Replace Sara Sepe with:
Sara Sepe Italy
Mylene Huebecker United Kingdom
Annabelle Chaussenot France
Michiel Krols Belgium
Stefania Assereto Italy
Qini Gan United States
Pascale Baden Germany
Annelies Rotthier Belgium
Nancy Sue Australia
Daniel H. Paushter United States
Hila Zigdon relative to Sara Sepe Italy Sara Sepe's profile →
Citations per field
00.5×1.5×2.2×
Sara Sepe · 1×
Citations per year

Countries citing papers authored by Hila Zigdon

Since Specialization
Citations

This map shows the geographic impact of Hila Zigdon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hila Zigdon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hila Zigdon more than expected).

Fields of papers citing papers by Hila Zigdon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hila Zigdon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hila Zigdon. The network helps show where Hila Zigdon may publish in the future.

Co-authors

The 25 scholars most cited alongside Hila Zigdon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Hila Zigdon Line = papers co-authored together Hila Zigdon links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 2009190
2 2013175
3 201068
4 201557
5 201651
6 201333
7 201718
8 201412

About Hila Zigdon

Hila Zigdon is a scholar working on Molecular Biology, Physiology, Cell Biology, Organic Chemistry and Epidemiology, having authored 8 papers that have together received 604 indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (5 papers), Cellular transport and secretion (4 papers), Sphingolipid Metabolism and Signaling (3 papers), Glycosylation and Glycoproteins Research (2 papers), Carbohydrate Chemistry and Synthesis (2 papers), Congenital heart defects research (1 paper), Phagocytosis and Immune Regulation (1 paper) and Lipid Membrane Structure and Behavior (1 paper). The work is most often cited by research in Physiology (44 citations), Physiology (215 citations), Aging (14 citations), Cell Biology (125 citations) and Molecular Biology (411 citations). Hila Zigdon has collaborated with scholars based in Israel, United States and Germany. Frequent co-authors include Anthony H. Futerman, Elisha Nathan, Eldad Tzahor, Sylvia Μ. Evans, Nuno Guimarães‐Camboa, Itamar Harel, Yael Pewzner‐Jung, Alfred H. Merrill, Aviram Kogot‐Levin and Ann Saada. Their work appears in journals such as Journal of Biological Chemistry, Developmental Cell, Cell Death and Disease, The Journal of Pathology and Human Molecular Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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