Chris Marshall
- Molecular Biology top 5%
- Oncology top 2%
- Cancer Research top 2%
- Cell Biology top 2%
- Immunology top 5%
- Co-authors
- Harvey W. SmithRichard MaraisAmine SadokPhilip CohenHugh F. PatersonHugh PatersonKlaus SeedorfAxel Ullrich
- Topics
- Protein Kinase Regulation and GTPase Signaling (12 papers)Melanoma and MAPK Pathways (7 papers)Endometrial and Cervical Cancer Treatments (3 papers)
- Partner nations
- United KingdomUnited StatesAustralia
In The Last Decade
Chris Marshall
33 papers receiving 3.5k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Molecular Biology 2.2k
- Oncology 999
- Cancer Research 676
- Cell Biology 608
- Immunology 445
Countries citing papers authored by Chris Marshall
This map shows the geographic impact of Chris Marshall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chris Marshall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chris Marshall more than expected).
Fields of papers citing papers by Chris Marshall
This network shows the impact of papers produced by Chris Marshall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chris Marshall. The network helps show where Chris Marshall may publish in the future.
Co-authorship network of co-authors of Chris Marshall
This figure shows the co-authorship network connecting the top 25 collaborators of Chris Marshall. A scholar is included among the top collaborators of Chris Marshall based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chris Marshall. Chris Marshall is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 6 | |
| 3 | 4 | |
| 4 | 1 | |
| 5 | Regulation of cell signalling by uPARbreakdown → | 743 |
| 6 | 59 | |
| 7 | 246 | |
| 8 | 2 | |
| 9 | New technologies in gynecologic cytology. | 17 |
| 10 | 131 | |
| 11 | 186 | |
| 12 | 13 | |
| 13 | 36 | |
| 14 | 411 | |
| 15 | 173 | |
| 16 | 29 | |
| 17 | 152 | |
| 18 | 138 | |
| 19 | 17 | |
| 20 | A study of ras gene mutations in colonic adenomas from familial polyposis coli patients. | 48 |
About Chris Marshall
Chris Marshall is a scholar working on Ecological Modeling, Obstetrics and Gynecology and Immunology and Allergy, having authored 34 papers that have together received 3.6k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (12 papers), Melanoma and MAPK Pathways (7 papers) and Endometrial and Cervical Cancer Treatments (3 papers). The work is most often cited by research in Immunology and Allergy (347 citations), Cancer Research (676 citations) and Cell Biology (608 citations). Chris Marshall has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include Harvey W. Smith, Richard Marais, Amine Sadok, Philip Cohen, Hugh F. Paterson, Hugh Paterson, Klaus Seedorf, Axel Ullrich, Sarah Traverse and Tony Magee. Their work appears in journals such as Nature, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.