David J. Easty

1.4k total citations
34 papers, 1.1k citations indexed

About

David J. Easty is a scholar working on Molecular Biology, Cell Biology and Immunology and Allergy. According to data from OpenAlex, David J. Easty has authored 34 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 13 papers in Cell Biology and 6 papers in Immunology and Allergy. Recurrent topics in David J. Easty's work include Melanoma and MAPK Pathways (8 papers), melanin and skin pigmentation (7 papers) and Cell Adhesion Molecules Research (6 papers). David J. Easty is often cited by papers focused on Melanoma and MAPK Pathways (8 papers), melanin and skin pigmentation (7 papers) and Cell Adhesion Molecules Research (6 papers). David J. Easty collaborates with scholars based in United Kingdom, Ireland and United States. David J. Easty's co-authors include Dorothy C. Bennett, Dorothy C. Bennett, Christine J. Farr, Viví Ann Flørenes, Colin R. Goding, Ugur Yavuzer, Suzanne Carreira, Simon P. Hill, Meenhard Herlyn and Philip J. Mitchell and has published in prestigious journals such as Molecular and Cellular Biology, Oncogene and The FASEB Journal.

In The Last Decade

David J. Easty

33 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David J. Easty United Kingdom 16 767 321 241 223 109 34 1.1k
Stéphane Ory France 20 1.2k 1.6× 641 2.0× 291 1.2× 122 0.5× 187 1.7× 45 1.8k
Pamela J. Woodring United States 10 744 1.0× 242 0.8× 114 0.5× 99 0.4× 78 0.7× 11 1.1k
Marı́a J. Caloca Spain 19 910 1.2× 316 1.0× 195 0.8× 97 0.4× 142 1.3× 33 1.2k
Kristin Roovers United States 6 966 1.3× 284 0.9× 310 1.3× 96 0.4× 126 1.2× 7 1.3k
Michael D. Waterfield United Kingdom 9 1.3k 1.7× 370 1.2× 303 1.3× 86 0.4× 220 2.0× 9 1.6k
Shane Minogue United Kingdom 24 1.1k 1.4× 660 2.1× 166 0.7× 74 0.3× 144 1.3× 36 1.6k
Emma Villa-Moruzzi Italy 23 1.2k 1.6× 510 1.6× 253 1.0× 69 0.3× 122 1.1× 55 1.5k
Jean Kloss United States 14 645 0.8× 201 0.6× 178 0.7× 57 0.3× 161 1.5× 21 1.1k
Ha Kun Kim United States 7 808 1.1× 290 0.9× 140 0.6× 91 0.4× 153 1.4× 7 1.1k
Mehran Amoui United States 18 839 1.1× 123 0.4× 195 0.8× 137 0.6× 144 1.3× 26 1.1k

Countries citing papers authored by David J. Easty

Since Specialization
Citations

This map shows the geographic impact of David J. Easty's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David J. Easty with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David J. Easty more than expected).

Fields of papers citing papers by David J. Easty

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David J. Easty. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David J. Easty. The network helps show where David J. Easty may publish in the future.

Co-authorship network of co-authors of David J. Easty

This figure shows the co-authorship network connecting the top 25 collaborators of David J. Easty. A scholar is included among the top collaborators of David J. Easty based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David J. Easty. David J. Easty is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rashed, Ahmed Nabih Zaki, David J. Easty, Dearbhaile Catherine Collins, et al.. (2023). An observational study of dose dense chemotherapy with lipegfilgrastim support in early breast cancer. BMC Cancer. 23(1). 171–171. 1 indexed citations
2.
Baird, Anne‐Marie, Kenneth J. O’Byrne, David J. Easty, et al.. (2014). 79 The RON (MST1R)/MSP pathway is a potential therapeutic target in malignant pleural mesothelioma. Lung Cancer. 83. S29–S30. 3 indexed citations
3.
Easty, David J., Steven G. Gray, Kenneth J. O’Byrne, D. O’Donnell, & Dorothy C. Bennett. (2011). Receptor tyrosine kinases and their activation in melanoma. Pigment Cell & Melanoma Research. 24(3). 446–461. 61 indexed citations
4.
Easty, David J., et al.. (2006). Protein Tyrosine Phosphatases, New Targets for Cancer Therapy. Current Cancer Drug Targets. 6(6). 519–532. 53 indexed citations
5.
Patel, Ketan J., David J. Easty, & Michael J. Dünn. (2003). Detection of Proteins in Polyacrylamide Gels Using an Ultrasensitive Silver Staining Technique. Humana Press eBooks. 3. 159–168. 9 indexed citations
6.
Bergin, Orla, et al.. (2003). Tyrosine phosphate in melanoma progression. British Journal of Dermatology. 149(2). 289–295. 9 indexed citations
7.
Easty, David J. & Dorothy C. Bennett. (2000). Protein tyrosine kinases in malignant melanoma. Melanoma Research. 10(5). 401–411. 95 indexed citations
8.
Easty, David J., Simon P. Hill, Mary E. Fallowfield, et al.. (1999). Up-regulation of ephrin-a1 during melanoma progression. International Journal of Cancer. 84(5). 494–501. 113 indexed citations
9.
Easty, David J., et al.. (1999). Requirement for focal adhesion kinase in tumor cell adhesion. Oncogene. 18(48). 6824–6828. 47 indexed citations
10.
Mills, Walter, et al.. (1999). Cloning and characterisation of the chicken gene encoding the telomeric protein TRF2. Gene. 239(1). 81–90. 19 indexed citations
11.
12.
Sviderskaya, Elena V., David J. Easty, Amanda J. Wilson, et al.. (1998). Melanosomal Defects in Melanocytes from Mice Lacking Expression of thePink-Eyed DilutionGene: Correction by Culture in the Presence of Excess Tyrosine. Experimental Cell Research. 239(2). 344–352. 39 indexed citations
13.
Bennett, Dorothy C., Ian D. Trayner, Xianhua Piao, et al.. (1998). recessive spotting: a linked locus that interacts with W/Kit but is not allelic. Genes to Cells. 3(4). 235–244. 5 indexed citations
14.
Sviderskaya, Elena V., David J. Easty, & Dorothy C. Bennett. (1998). Impaired growth and differentiation of diploid but not immortal melanoblasts from endothelin receptor B mutant (piebald) mice. Developmental Dynamics. 213(4). 452–463. 13 indexed citations
15.
Easty, David J., Philip J. Mitchell, Ketan Patel, et al.. (1997). Loss of expression of receptor tyrosine kinase family genesPTK7 andSEK in metastatic melanoma. International Journal of Cancer. 71(6). 1061–1065. 114 indexed citations
16.
Easty, David J., et al.. (1993). Novel and Known Protein Tyrosine Kinases and Their Abnormal Expression in Human Melanoma. Journal of Investigative Dermatology. 101(5). 679–684. 37 indexed citations
17.
Farr, Christine J., David J. Easty, Jiannis Ragoussis, et al.. (1993). Characterization and mapping of the human SOX4 gene. Mammalian Genome. 4(10). 577–584. 50 indexed citations
18.
Easty, David J., Ian R. Hart, Colin Seymour, et al.. (1991). Changes in protein expression during melanoma differentiation determined by computer analysis of 2-D gels. Clinical & Experimental Metastasis. 9(3). 221–230. 5 indexed citations
19.
20.
Farr, Christine J., Chris Marshall, David J. Easty, et al.. (1988). A study of ras gene mutations in colonic adenomas from familial polyposis coli patients.. PubMed. 3(6). 673–8. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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