Carl W. Cotman

4.0k total citations · 1 hit paper
32 papers, 3.3k citations indexed

About

Carl W. Cotman is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Carl W. Cotman has authored 32 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 17 papers in Physiology and 14 papers in Cellular and Molecular Neuroscience. Recurrent topics in Carl W. Cotman's work include Alzheimer's disease research and treatments (17 papers), Neuroscience and Neuropharmacology Research (10 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Carl W. Cotman is often cited by papers focused on Alzheimer's disease research and treatments (17 papers), Neuroscience and Neuropharmacology Research (10 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Carl W. Cotman collaborates with scholars based in United States, Canada and Netherlands. Carl W. Cotman's co-authors include Joseph H. Su, B. Cummings, Kevin Anderson, Sandra Lee, David H. Cribbs, Edward R. Whittemore, Elizabeth Head, Ben H. Choi, James W. Geddes and A.J. Anderson and has published in prestigious journals such as Nature, Brain Research and FEBS Letters.

In The Last Decade

Carl W. Cotman

32 papers receiving 3.3k citations

Hit Papers

Basic fibroblast growth factor prevents death of lesioned... 1988 2026 2000 2013 1988 100 200 300 400 500

Peers

Carl W. Cotman
R E Rydel United States
Richard Killick United Kingdom
MP Mattson United States
Joseph H. Su United States
Surojit Paul United States
R E Rydel United States
Carl W. Cotman
Citations per year, relative to Carl W. Cotman Carl W. Cotman (= 1×) peers R E Rydel

Countries citing papers authored by Carl W. Cotman

Since Specialization
Citations

This map shows the geographic impact of Carl W. Cotman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carl W. Cotman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carl W. Cotman more than expected).

Fields of papers citing papers by Carl W. Cotman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Carl W. Cotman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carl W. Cotman. The network helps show where Carl W. Cotman may publish in the future.

Co-authorship network of co-authors of Carl W. Cotman

This figure shows the co-authorship network connecting the top 25 collaborators of Carl W. Cotman. A scholar is included among the top collaborators of Carl W. Cotman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Carl W. Cotman. Carl W. Cotman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rohn, Troy T., et al.. (2001). Activation of Caspase-8 in the Alzheimer's Disease Brain. Neurobiology of Disease. 8(6). 1006–1016. 147 indexed citations
2.
Rohn, Troy T., Elizabeth Head, Joseph H. Su, et al.. (2001). Correlation between Caspase Activation and Neurofibrillary Tangle Formation in Alzheimer’s Disease. American Journal Of Pathology. 158(1). 189–198. 134 indexed citations
3.
Azizeh, Bassem Y., Elizabeth Head, Reidun Torp, et al.. (2000). Molecular Dating of Senile Plaques in the Brains of Individuals with Down Syndrome and in Aged Dogs. Experimental Neurology. 163(1). 111–122. 47 indexed citations
4.
Callahan, Heather, Candace J Ikeda-Douglas, Elizabeth Head, Carl W. Cotman, & Norton W. Milgram. (2000). Development of a protocol for studying object recognition memory in the dog. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 24(5). 693–707. 43 indexed citations
5.
Davis, Judianne, David H. Cribbs, Carl W. Cotman, & William E. Van Nostrand. (1999). Pathogenic amyloid β-protein induces apoptosis in cultured human cerebrovascular smooth muscle cells. Amyloid. 6(3). 157–164. 56 indexed citations
6.
Cribbs, David H., Liansheng Chen, Carl W. Cotman, & Frank M. LaFerla. (1996). Injury induces presenilin-1 gene expression in mouse brain. Neuroreport. 7(11). 1773–1776. 34 indexed citations
7.
Deng, Gangmin, Christian J. Pike, & Carl W. Cotman. (1996). Alzheimer‐associated presenilin‐2 confers increased sensitivity to poptosis in PC12 cells. FEBS Letters. 397(1). 50–54. 112 indexed citations
8.
Woods, Alisa G., David H. Cribbs, Edward R. Whittemore, & Carl W. Cotman. (1995). Heparan sulfate and chondroitin sulfate glycosaminoglycan attenuate β-amyloid(25–35) induced neurodegeneration in cultured hippocampal neurons. Brain Research. 697(1-2). 53–62. 61 indexed citations
9.
Pike, Christian J. & Carl W. Cotman. (1995). Calretinin-immunoreactive neurons are resistant to β-amyloid toxicity in vitro. Brain Research. 671(2). 293–298. 38 indexed citations
10.
Satou, Takao, B. Cummings, & Carl W. Cotman. (1995). Immunoreactivity for Bcl-2 protein within neurons in the Alzheimer's disease brain increases with disease severity. Brain Research. 697(1-2). 35–43. 110 indexed citations
11.
Vaughan, Patrick, et al.. (1994). Protease nexin-1, a potent thrombin inhibitor, is reduced around cerebral blood vessels in Alzheimer's disease. Brain Research. 668(1-2). 160–170. 58 indexed citations
12.
Anderson, A.J., B. Cummings, & Carl W. Cotman. (1994). Increased Immunoreactivity for Jun- and Fos-Related Proteins in Alzheimer's Disease: Association with Pathology. Experimental Neurology. 125(2). 286–295. 142 indexed citations
13.
Ułas, J., et al.. (1992). Elevated NMDA receptors in Parkinsonian striatum. Neuroreport. 3(11). 977–980. 52 indexed citations
14.
Whittemore, Edward R. & Carl W. Cotman. (1991). Agonists selective for phosphoinositide-coupled receptors sensitize neurons to depolarization by l-2-amino-4-phosphonobutanoic acid (l-AP4). Brain Research. 555(2). 215–219. 11 indexed citations
15.
Geddes, James W., John C.T. Wong, Ben H. Choi, et al.. (1990). Increased expression of the embryonic form of a developmentally regulated mRNA in Alzheimer's disease. Neuroscience Letters. 109(1-2). 54–61. 53 indexed citations
16.
Geddes, James W., et al.. (1990). Altered distribution of excitatory amino acid receptors in temporal lobe epilepsy. Experimental Neurology. 108(3). 214–220. 72 indexed citations
17.
Peterson, Christine, John H. Neal, & Carl W. Cotman. (1989). Development of excitotoxicity in cultured hippocampal neurons. Developmental Brain Research. 48(2). 187–195. 95 indexed citations
18.
Balázs, R., N. Hack, Ole Steen Jørgensen, & Carl W. Cotman. (1989). promotes the survival of cerebellar granule cells: Pharmacological characterization. Neuroscience Letters. 101(3). 241–246. 86 indexed citations
19.
Anderson, Kevin, et al.. (1988). Basic fibroblast growth factor prevents death of lesioned cholinergic neurons in vivo. Nature. 332(6162). 360–361. 524 indexed citations breakdown →
20.
Palmer, Elizabeth E., Daniel T. Monaghan, & Carl W. Cotman. (1988). Glutamate receptors and phosphoinositide metabolism: stimulation via quisqualate receptors is inhibited by receptor activation. Molecular Brain Research. 4(2). 161–165. 96 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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