Bipasha Guptaroy
- Cellular and Molecular Neuroscience top 5%
- Molecular Biology
- Cell Biology
- Cognitive Neuroscience
- Psychiatry and Mental health
- Co-authors
- Margaret E. GnegyMinjia ZhangL’Aurelle A. JohnsonAurelio GalliJonathan A. JavitchCheryse A. FurmanHabibeh KhoshboueiNamita Sen
- Topics
- Neurotransmitter Receptor Influence on Behavior (11 papers)Neuroscience and Neuropharmacology Research (10 papers)Receptor Mechanisms and Signaling (9 papers)
- Partner nations
- United StatesPakistanCanada
In The Last Decade
Bipasha Guptaroy
21 papers receiving 777 citations
Peers
Comparison fields: 5 of 72
- Cellular and Molecular Neuroscience 575
- Molecular Biology 507
- Cell Biology 73
- Cognitive Neuroscience 58
- Psychiatry and Mental health 55
Countries citing papers authored by Bipasha Guptaroy
This map shows the geographic impact of Bipasha Guptaroy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bipasha Guptaroy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bipasha Guptaroy more than expected).
Fields of papers citing papers by Bipasha Guptaroy
This network shows the impact of papers produced by Bipasha Guptaroy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bipasha Guptaroy. The network helps show where Bipasha Guptaroy may publish in the future.
Co-authorship network of co-authors of Bipasha Guptaroy
This figure shows the co-authorship network connecting the top 25 collaborators of Bipasha Guptaroy. A scholar is included among the top collaborators of Bipasha Guptaroy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bipasha Guptaroy. Bipasha Guptaroy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 10 | |
| 2 | 23 | |
| 3 | 8 | |
| 4 | 2 | |
| 5 | 34 | |
| 6 | 9 | |
| 7 | 19 | |
| 8 | 78 | |
| 9 | 1 | |
| 10 | 53 | |
| 11 | 7 | |
| 12 | 82 | |
| 13 | 112 | |
| 14 | 204 | |
| 15 | 20 | |
| 16 | 25 | |
| 17 | 21 | |
| 18 | 23 | |
| 19 | 21 | |
| 20 | 11 |
About Bipasha Guptaroy
Bipasha Guptaroy is a scholar working on Cellular and Molecular Neuroscience, Toxicology and Molecular Biology, having authored 21 papers that have together received 782 indexed citations. Recurring topics across this work include Neurotransmitter Receptor Influence on Behavior (11 papers), Neuroscience and Neuropharmacology Research (10 papers) and Receptor Mechanisms and Signaling (9 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (575 citations), Molecular Biology (507 citations) and Toxicology (20 citations). Bipasha Guptaroy has collaborated with scholars based in United States, Pakistan and Canada. Frequent co-authors include Margaret E. Gnegy, Minjia Zhang, L’Aurelle A. Johnson, Aurelio Galli, Jonathan A. Javitch, Cheryse A. Furman, Habibeh Khoshbouei, Namita Sen, Colin P. Johnson and Leslie C. Griffith. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.