William H. Baldridge

1.9k citations
51 papers · 1.6k indexed · h-index 26
Topics
Retinal Development and Disorders (33 papers)Neuroscience and Neuropharmacology Research (23 papers)Photoreceptor and optogenetics research (19 papers)

In The Last Decade

William H. Baldridge

51 papers receiving 1.6k citations

Peers

William H. Baldridge
Comparison fields: 5 of 83
  • Molecular Biology 1.1k
  • Cellular and Molecular Neuroscience 920
  • Endocrine and Autonomic Systems 252
  • Ophthalmology 248
  • Cognitive Neuroscience 107
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Citations per field
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Citations per year

Countries citing papers authored by William H. Baldridge

Since Specialization
Citations

This map shows the geographic impact of William H. Baldridge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William H. Baldridge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William H. Baldridge more than expected).

Fields of papers citing papers by William H. Baldridge

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William H. Baldridge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William H. Baldridge. The network helps show where William H. Baldridge may publish in the future.

Co-authorship network of co-authors of William H. Baldridge

This figure shows the co-authorship network connecting the top 25 collaborators of William H. Baldridge. A scholar is included among the top collaborators of William H. Baldridge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William H. Baldridge. William H. Baldridge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 25
2
Retinal characterization of the Thy1-GCaMP3 mouse after optic nerve transection
2
3 12
4
NMDA-induced Calcium Dynamics Are Altered In Retinas Of Adult Mice Deficient In The Neural Cell Adhesion Molecule (NCAM)
2
5 6
6 16
7 62
8 39
9 12
10 25
11 110
12 32
13 89
14 36
15 41
16 50
17 42
18 17
19 7
20 15

About William H. Baldridge

William H. Baldridge is a scholar working on Cellular and Molecular Neuroscience, Ophthalmology and Physiology, having authored 51 papers that have together received 1.6k indexed citations. Recurring topics across this work include Retinal Development and Disorders (33 papers), Neuroscience and Neuropharmacology Research (23 papers) and Photoreceptor and optogenetics research (19 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (920 citations), Endocrine and Autonomic Systems (252 citations) and Ophthalmology (248 citations). William H. Baldridge has collaborated with scholars based in Canada, United States and United Kingdom. Frequent co-authors include Alexander K. Ball, Andrew T. E. Hartwick, Steven Barnes, Reto Weiler, Michael E. Kelly, Bryan A. Daniels, Rodman G. Miller, Balwantray C. Chauhan, Melanie E. M. Kelly and David I. Vaney. Their work appears in journals such as Journal of Neuroscience, The Journal of Immunology and The Journal of Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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