Bernd O. Evert
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 1%
- Physiology top 5%
- Neurology top 2%
- Epidemiology top 10%
- Co-authors
- Ullrich WüllnerThomas KlockgetherIna SchmittMichel EichelbaumPéter BreuerDavid C. RubinszteinZdenek BergerBrinda Ravikumar
- Topics
- Genetic Neurodegenerative Diseases (24 papers)Mitochondrial Function and Pathology (20 papers)Ubiquitin and proteasome pathways (9 papers)
- Partner nations
- GermanyNetherlandsUnited States
In The Last Decade
Bernd O. Evert
48 papers receiving 3.2k citations
Hit Papers
Peers
Comparison fields: 5 of 106
- Molecular Biology 2.0k
- Cellular and Molecular Neuroscience 1.3k
- Physiology 588
- Neurology 533
- Epidemiology 448
Countries citing papers authored by Bernd O. Evert
This map shows the geographic impact of Bernd O. Evert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bernd O. Evert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bernd O. Evert more than expected).
Fields of papers citing papers by Bernd O. Evert
This network shows the impact of papers produced by Bernd O. Evert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bernd O. Evert. The network helps show where Bernd O. Evert may publish in the future.
Co-authorship network of co-authors of Bernd O. Evert
This figure shows the co-authorship network connecting the top 25 collaborators of Bernd O. Evert. A scholar is included among the top collaborators of Bernd O. Evert based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bernd O. Evert. Bernd O. Evert is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 1 | |
| 3 | 2 | |
| 4 | 10 | |
| 5 | 17 | |
| 6 | 10 | |
| 7 | 32 | |
| 8 | 85 | |
| 9 | 78 | |
| 10 | 92 | |
| 11 | 103 | |
| 12 | 321 | |
| 13 | 125 | |
| 14 | 18 | |
| 15 | 124 | |
| 16 | 91 | |
| 17 | Rapamycin alleviates toxicity of different aggregate-prone proteinsbreakdown → | 532 |
| 18 | 14 | |
| 19 | 60 | |
| 20 | 41 |
About Bernd O. Evert
Bernd O. Evert is a scholar working on Cellular and Molecular Neuroscience, Pharmacology and Molecular Biology, having authored 49 papers that have together received 3.3k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (24 papers), Mitochondrial Function and Pathology (20 papers) and Ubiquitin and proteasome pathways (9 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.3k citations), Pharmacology (322 citations) and Neurology (533 citations). Bernd O. Evert has collaborated with scholars based in Germany, Netherlands and United States. Frequent co-authors include Ullrich Wüllner, Thomas Klockgether, Ina Schmitt, Michel Eichelbaum, Péter Breuer, David C. Rubinsztein, Zdenek Berger, Brinda Ravikumar, Cahir J. O’Kane and Benjamin R. Underwood. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.