B Hemmer

771 total citations
18 papers, 371 citations indexed

About

B Hemmer is a scholar working on Neurology, Neurology and Immunology. According to data from OpenAlex, B Hemmer has authored 18 papers receiving a total of 371 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Neurology, 6 papers in Neurology and 5 papers in Immunology. Recurrent topics in B Hemmer's work include Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Stress Responses and Cortisol (2 papers) and Peripheral Neuropathies and Disorders (2 papers). B Hemmer is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Stress Responses and Cortisol (2 papers) and Peripheral Neuropathies and Disorders (2 papers). B Hemmer collaborates with scholars based in United States, Germany and Denmark. B Hemmer's co-authors include F.X. Glocker, Günther Deuschl, C. H. Lücking, M. Schumacher, Martin Schumacher, Vasileios K. Katsaros, Joseph M. Castellano, Dieter Riemann, Andreas Kupsch and Stefanie Klaffke and has published in prestigious journals such as SHILAP Revista de lepidopterología, Neurology and Journal of Neurology Neurosurgery & Psychiatry.

In The Last Decade

B Hemmer

16 papers receiving 356 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
B Hemmer United States	9	149	143	63	50	49	18	371
Gabriella Passerini Italy	11	71 0.5×	77 0.5×	39 0.6×	42 0.8×	122 2.5×	17	338
Waleed Altwaijri Saudi Arabia	11	132 0.9×	32 0.2×	49 0.8×	52 1.0×	46 0.9×	25	478
Anne Bolette Tjensvoll Norway	15	168 1.1×	236 1.7×	37 0.6×	36 0.7×	305 6.2×	23	642
Christopher Kneebone Australia	8	227 1.5×	29 0.2×	24 0.4×	74 1.5×	44 0.9×	12	420
Adrian Wills United Kingdom	13	316 2.1×	49 0.3×	62 1.0×	233 4.7×	25 0.5×	27	534
Gül Yalçın Çakmaklı Türkiye	10	169 1.1×	35 0.2×	41 0.7×	46 0.9×	41 0.8×	29	298
Raffaello Canapicchi Italy	12	73 0.5×	33 0.2×	23 0.4×	47 0.9×	30 0.6×	20	628
Thomas M. Walshe United States	12	165 1.1×	32 0.2×	27 0.4×	68 1.4×	32 0.7×	16	410
E Fernández-Álvarez Spain	12	282 1.9×	50 0.3×	38 0.6×	146 2.9×	47 1.0×	41	514
T Mannen Japan	11	179 1.2×	57 0.4×	35 0.6×	102 2.0×	18 0.4×	32	339

Countries citing papers authored by B Hemmer

Since Specialization

Citations

This map shows the geographic impact of B Hemmer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B Hemmer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B Hemmer more than expected).

Fields of papers citing papers by B Hemmer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by B Hemmer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B Hemmer. The network helps show where B Hemmer may publish in the future.

Co-authorship network of co-authors of B Hemmer

This figure shows the co-authorship network connecting the top 25 collaborators of B Hemmer. A scholar is included among the top collaborators of B Hemmer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with B Hemmer. B Hemmer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Friese, Matthew B., Taranjit S. Gujral, Arvind Palanisamy, et al.. (2023). Anesthetics inhibit phosphorylation of the ribosomal protein S6 in mouse cultured cortical cells and developing brain. Frontiers in Aging Neuroscience. 15. 1060186–1060186. 1 indexed citations

Hemmer, B, et al.. (2023). Youth-associated platelet-derived chemokine reverses brain aging through neuroimmune mechanisms. Trends in Molecular Medicine. 30(1). 10–12. 3 indexed citations

Ferreira, Ana Catarina, B Hemmer, Hanxiao Liu, et al.. (2023). Neuronal TIMP2 regulates hippocampus-dependent plasticity and extracellular matrix complexity. Molecular Psychiatry. 28(9). 3943–3954. 13 indexed citations

Neuner, Sarah M., B Hemmer, Yiyuan Liu, et al.. (2022). APOE4 confers transcriptomic and functional alterations to primary mouse microglia. Neurobiology of Disease. 164. 105615–105615. 41 indexed citations

Hemmer, B, et al.. (2020). Social housing enhances acquisition of task set independently of environmental enrichment: A longitudinal study in the Barnes maze. Learning & Behavior. 48(3). 322–334. 12 indexed citations

Chandratre, Saleel, Olga Ciccarelli, J. de Sèze, et al.. (2020). Treatment of MOG antibody associated disorders: results of an international survey. Journal of Neurology. 267(12). 3565–3577. 9 indexed citations

Hemmer, B, et al.. (2019). Social vs. Nonsocial Housing Differentially Affects Perseverative Behavior in Rats (Ratus norvegicus). SHILAP Revista de lepidopterología. 6(3). 168–178. 3 indexed citations

Kovács, Katalin, Sudhakar Reddy Kalluri, Tomas Deierborg, et al.. (2015). Longitudinal analysis of autoantibodies, IFN-gamma, and IL-17 in the serum of patients with concomitant NMO and SLE. University of Southern Denmark Research Portal (University of Southern Denmark). 21. 81–82. 1 indexed citations

Cepok, Sabine, et al.. (2014). A Novel Bio-Assay To Determine and Characterize the Antibodies to Aquaporin-4.

10.

Krogias, Christos, Georgios Tsivgoulis, Martin Grond, et al.. (2013). Review and Analysis of International Case-Control Studies on CCSVI and Multiple Sclerosis. mediaTUM (Technical University of Munich). 2 indexed citations

11.

Valet, Michael, Harald Gündel, Till Sprenger, et al.. (2007). Gray-matter alterations in pain processing brain structures in patients with somatoform pain disorder – a voxel-based morphometric study. Aktuelle Neurologie. 34(S 2). 1 indexed citations

12.

Nessler, Sylvie, et al.. (2007). The antidepressant venlafaxine ameliorates murine experimental autoimmune encephalomyelitis. Aktuelle Neurologie. 34(S 2). 1 indexed citations

13.

Hague, Stephen, Stefanie Klaffke, Jordi Clarimón, et al.. (2006). Lack of association with TorsinA haplotype in German patients with sporadic dystonia. Neurology. 66(6). 951–952. 37 indexed citations

14.

Cepok, Sabine, et al.. (2006). Accumulation of class switched IgD-IgM- memory B cells in the cerebrospinal fluid during neuroinflammation. Aktuelle Neurologie. 33(S 1). 1 indexed citations

15.

Cepok, Sabine, Frederick Vogel, Verena Grummel, et al.. (2005). Short-lived plasma blasts are the main B cell effector subset during the course of multiple sclerosis. Aktuelle Neurologie. 32(S 4). 8 indexed citations

16.

Hemmer, B, F.X. Glocker, M. Schumacher, Günther Deuschl, & C. H. Lücking. (1998). Subacute combined degeneration: clinical, electrophysiological, and magnetic resonance imaging findings. Journal of Neurology Neurosurgery & Psychiatry. 65(6). 822–827. 159 indexed citations

17.

Katsaros, Vasileios K., F.X. Glocker, B Hemmer, & Martin Schumacher. (1998). MRI of spinal cord and brain lesions in subacute combined degeneration. Neuroradiology. 40(11). 716–719. 53 indexed citations

18.

Hemmer, B, Dieter Riemann, F.X. Glocker, C. H. Lücking, & Günther Deuschl. (1995). Restless legs syndrome after a borrelia‐induced myelitis. Movement Disorders. 10(4). 521–522. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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