Asha Naidu

970 total citations · 1 hit paper
8 papers, 794 citations indexed

About

Asha Naidu is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Asha Naidu has authored 8 papers receiving a total of 794 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 4 papers in Cellular and Molecular Neuroscience and 2 papers in Molecular Biology. Recurrent topics in Asha Naidu's work include Alzheimer's disease research and treatments (6 papers), Nuclear Receptors and Signaling (4 papers) and Neonatal Respiratory Health Research (2 papers). Asha Naidu is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Nuclear Receptors and Signaling (4 papers) and Neonatal Respiratory Health Research (2 papers). Asha Naidu collaborates with scholars based in United States, Belgium and France. Asha Naidu's co-authors include Barbara Cordell, Ilse Dewachter, Fred Van Leuven, Frédéric Checler, Kurt Spittaels, Dieder Moechars, Kristin Lorent, Delphine Reversé, Chris Van den Haute and Veerle Baekelandt and has published in prestigious journals such as Journal of Biological Chemistry, Brain Research and Neuroreport.

In The Last Decade

Asha Naidu

8 papers receiving 773 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain

1999 553 citations Dieder Moechars, Ilse Dewachter et al. Journal of Biological Chemistry profile →

Peers

Countries citing papers authored by Asha Naidu

Since Specialization

Citations

This map shows the geographic impact of Asha Naidu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Asha Naidu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Asha Naidu more than expected).

Fields of papers citing papers by Asha Naidu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Asha Naidu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Asha Naidu. The network helps show where Asha Naidu may publish in the future.

Co-authorship network of co-authors of Asha Naidu

This figure shows the co-authorship network connecting the top 25 collaborators of Asha Naidu. A scholar is included among the top collaborators of Asha Naidu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Asha Naidu. Asha Naidu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Secretion of apolipoprotein E by brain glia requires protein prenylation and is suppressed by statins 2002 ·Brain Research ·Asha Naidu, Qiang Xu, Rosanne Catalano, Barbara Cordell	35
2	Conversion of brain apolipoprotein E to an insoluble form in a mouse model of Alzheimer disease 2001 ·Neuroreport ·Asha Naidu, Rosanne Catalano, Kelly R. Bales, Su Wu, Steven M. Paul, Barbara Cordell	9
3	Behavioral Disturbances without Amyloid Deposits in Mice Overexpressing Human Amyloid Precursor Protein with Flemish (A692G) or Dutch (E693Q) Mutation 2000 ·Neurobiology of Disease ·Samir Kumar‐Singh, Ilse Dewachter, (unknown), Ursula Lübke, (unknown), C. Ceuterick, Frédéric Checler, Asha Naidu, Barbara Cordell, Patrick Cras, Christine Van Broeckhoven, Fred Van Leuven	84
4	Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain breakdown → 1999 ·Journal of Biological Chemistry ·Dieder Moechars, Ilse Dewachter, Kristin Lorent, Delphine Reversé, Veerle Baekelandt, Asha Naidu, Ina Tesseur, Kurt Spittaels, Chris Van den Haute, Frédéric Checler, Emile Godaux, Barbara Cordell, Fred Van Leuven	553
5	β-Amyloid Peptide Secretion by a Microglial Cell Line Is Induced by β-Amyloid-(25–35) and Lipopolysaccharide 1996 ·Journal of Biological Chemistry ·(unknown), Asha Naidu, Barbara Cordell, Greer M. Murphy	65
6	Association of surfactant protein C with isolated alveolar type II cells 1995 ·Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism ·Ricardo A. Pinto, Samuel Hawgood, John A. Clements, Bradley Benson, Asha Naidu, Robert L. Hamilton, Jo Rae Wright	5
7	β-Amyloid Peptide Produced in Vitro Is Degraded by Proteinases Released by Cultured Cells 1995 ·Journal of Biological Chemistry ·Asha Naidu, Diana Quon, Barbara Cordell	37
8	Lung surfactant protein SP-C from human, bovine, and canine sources contains palmityl cysteine thioester linkages 1992 ·American Journal of Physiology-Lung Cellular and Molecular Physiology ·John T. Stults, Patrick R. Griffin, (unknown), Asha Naidu, Barbara Moffat, Bradley Benson	6

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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